Pathophysiology of Immunity, d. Synthesize lysosomes.
Inflammation, Infection & Stress Response
3. Which secretion is a first line of defense
– Comprehensive Test Bank (Chapters 7–
against pathogen invasion that involves
11)
antibacterial and antifungal fatty acids, as
CHAPTER 7 well as lactic acid? a. Optic tears
b. Oral saliva
1. Which action is a purpose of the
c. Sweat gland perspiration
inflammatory process?
d. Sebaceous gland sebum
a. To provide specific responses toward
antigens 4. Which bacterium grows in the intestines
b. To lyse cell membranes of after prolonged antibiotic therapy?
microorganisms a. Lactobacillus
c. To prevent infection of the injured tissue b. Candida albicans
d. To create immunity against subsequent c. Clostridium difficile
tissue injury d. Helicobacter pylori
2. How do surfactant proteins A through D 5. What causes the edema that occurs
provide innate resistance? during the inflammatory process?
a. Initiate the complement cascade. a. Vasodilation of blood vessels
b. Promote phagocytosis. b. Increased capillary permeability
c. Secrete mucus. c. Endothelial cell expansion
,d. Emigration of neutrophils 9. Which component of the plasma
6. What process causes heat and redness to protein system tags pathogenic
occur during the inflammatory process? microorganisms for destruction by
a. Vasodilation of blood vessels neutrophils and macrophages? a.
b. Platelet aggregation Complement cascade
c. Decreased capillary permeability b. Coagulation system
d. Endothelial cell contraction c. Kinin system
d. Immune system
7. What does activation of the classical
pathway begin with? 10. What is the vascular effect of histamine
a. Viruses released from mast cells?
b. Antigen-antibody complexes a. Platelet adhesion
c. Mast cells b. Initiation of the clotting cascade
d. Macrophages c. Vasodilation
d. Increased endothelial adhesiveness
8. What plasma protein system forms a
11. What is an outcome of the complement
fibrinous meshwork at an inflamed
cascade?
site?
a. Activation of the clotting cascade
a. Complement
b. Prevention of the spread of infection to
b. Coagulation
adjacent tissues
c. Kinin
d. Fibrinolysis
,c. Inactivation of chemical mediators such b. VII
as histamine c. X
d. Lysis of bacterial cell membranes d. V
12. What is the function of opsonization 14. Which chemical interacts among all
related to the complement cascade? plasma protein systems by degrading blood
a. To tag pathogenic microorganisms for clots, activating complement, and
destruction by neutrophils and activating the Hageman factor?
a. Kallikrein
macrophages
b. Histamine
b. To process pathogenic microorganisms so c. Bradykinin
that activated lymphocytes can be d. Plasmin
created for acquired immunity
c. To destroy glycoprotein cell membranes 15. How does the chemotactic factor affect
of pathogenic microorganisms the inflammatory process?
d. To promote anaphylatoxic activity, a. By causing vasodilation around the
resulting in mast cell degranulation inflamed area
b. By stimulating smooth muscle contraction
13. In the coagulation (clotting) cascade,
in the inflamed area
the intrinsic and the extrinsic pathways
c. By directing leukocytes to the inflamed
converge at which factor?
area
a. XII
, d. By producing edema around the inflamed b. Complement and chemotaxis are
area deficient.
16. What effect does the process of c. Underlying chronic illness(es) exists.
histamine binding to the histamine-2 (H2) d. Number of mast cells is insufficient.
receptor have on inflammation? a.
19. Which chemical mediator derived from
Inhibition
mast cells retracts endothelial cells to
b. Activation
increase vascular permeability and to
c. Acceleration
cause leukocyte adhesion to endothelial
d. Termination
cells? a. Chemokines
17. Frequently when H1 and H2 receptors b. Prostaglandin E
are located on the same cells, they act in c. Platelet-activating factor
what fashion? a. Synergistically d. Bradykinin
b. Additively
c. Antagonistically
20. What is the inflammatory effect of
nitric oxide (NO)?
d. Agonistically
a. Increases capillary permeability, and
18. Some older adults have impaired causes pain
inflammation and wound healing because b. Increases neutrophil chemotaxis and
of which problem? platelet aggregation
a. Circulatory system cannot adequately c. Causes smooth muscle contraction and
perfuse tissues. fever
Inflammation, Infection & Stress Response
3. Which secretion is a first line of defense
– Comprehensive Test Bank (Chapters 7–
against pathogen invasion that involves
11)
antibacterial and antifungal fatty acids, as
CHAPTER 7 well as lactic acid? a. Optic tears
b. Oral saliva
1. Which action is a purpose of the
c. Sweat gland perspiration
inflammatory process?
d. Sebaceous gland sebum
a. To provide specific responses toward
antigens 4. Which bacterium grows in the intestines
b. To lyse cell membranes of after prolonged antibiotic therapy?
microorganisms a. Lactobacillus
c. To prevent infection of the injured tissue b. Candida albicans
d. To create immunity against subsequent c. Clostridium difficile
tissue injury d. Helicobacter pylori
2. How do surfactant proteins A through D 5. What causes the edema that occurs
provide innate resistance? during the inflammatory process?
a. Initiate the complement cascade. a. Vasodilation of blood vessels
b. Promote phagocytosis. b. Increased capillary permeability
c. Secrete mucus. c. Endothelial cell expansion
,d. Emigration of neutrophils 9. Which component of the plasma
6. What process causes heat and redness to protein system tags pathogenic
occur during the inflammatory process? microorganisms for destruction by
a. Vasodilation of blood vessels neutrophils and macrophages? a.
b. Platelet aggregation Complement cascade
c. Decreased capillary permeability b. Coagulation system
d. Endothelial cell contraction c. Kinin system
d. Immune system
7. What does activation of the classical
pathway begin with? 10. What is the vascular effect of histamine
a. Viruses released from mast cells?
b. Antigen-antibody complexes a. Platelet adhesion
c. Mast cells b. Initiation of the clotting cascade
d. Macrophages c. Vasodilation
d. Increased endothelial adhesiveness
8. What plasma protein system forms a
11. What is an outcome of the complement
fibrinous meshwork at an inflamed
cascade?
site?
a. Activation of the clotting cascade
a. Complement
b. Prevention of the spread of infection to
b. Coagulation
adjacent tissues
c. Kinin
d. Fibrinolysis
,c. Inactivation of chemical mediators such b. VII
as histamine c. X
d. Lysis of bacterial cell membranes d. V
12. What is the function of opsonization 14. Which chemical interacts among all
related to the complement cascade? plasma protein systems by degrading blood
a. To tag pathogenic microorganisms for clots, activating complement, and
destruction by neutrophils and activating the Hageman factor?
a. Kallikrein
macrophages
b. Histamine
b. To process pathogenic microorganisms so c. Bradykinin
that activated lymphocytes can be d. Plasmin
created for acquired immunity
c. To destroy glycoprotein cell membranes 15. How does the chemotactic factor affect
of pathogenic microorganisms the inflammatory process?
d. To promote anaphylatoxic activity, a. By causing vasodilation around the
resulting in mast cell degranulation inflamed area
b. By stimulating smooth muscle contraction
13. In the coagulation (clotting) cascade,
in the inflamed area
the intrinsic and the extrinsic pathways
c. By directing leukocytes to the inflamed
converge at which factor?
area
a. XII
, d. By producing edema around the inflamed b. Complement and chemotaxis are
area deficient.
16. What effect does the process of c. Underlying chronic illness(es) exists.
histamine binding to the histamine-2 (H2) d. Number of mast cells is insufficient.
receptor have on inflammation? a.
19. Which chemical mediator derived from
Inhibition
mast cells retracts endothelial cells to
b. Activation
increase vascular permeability and to
c. Acceleration
cause leukocyte adhesion to endothelial
d. Termination
cells? a. Chemokines
17. Frequently when H1 and H2 receptors b. Prostaglandin E
are located on the same cells, they act in c. Platelet-activating factor
what fashion? a. Synergistically d. Bradykinin
b. Additively
c. Antagonistically
20. What is the inflammatory effect of
nitric oxide (NO)?
d. Agonistically
a. Increases capillary permeability, and
18. Some older adults have impaired causes pain
inflammation and wound healing because b. Increases neutrophil chemotaxis and
of which problem? platelet aggregation
a. Circulatory system cannot adequately c. Causes smooth muscle contraction and
perfuse tissues. fever
