ARDS - nursing from Elizabeth Questions and
Answers (100% Correct Answers) Already
Graded A+
ARDS [ Ans: ] - acute respiratory distress syndrome
- sudden progressive form of acute respiratorty failure
- alveolar capillary interface becomes damaged and more
permeable to intravascular fluid
- alveoli fill with fluid
- hypoxia that persists even when oxygen is administered
at 100%
- decreased pulmonary compliance
- dyspnea
- non-cardiac associated bilateral pulmonary edema
- dense pulmonary infiltrates seen on xray
causes of lung injury in ARDS [ Ans: ] - systemic
inflammatory response is the common pathway
- intrinsically the alveolar-capillary membrane is injured
from conditions such as sepsis and shock
,- extrinsically the alveolar-capillary membrane is injured
from conditions such as aspiration or inhalation injury
diagnostic assessment of ARDS [ Ans: ] - lower PaO2 value
on ABG
- poor response to refractory hypoxemia
- ground-glass appearance to chest xray
- no cardiac involvement on ECG
- low to normal PCWP
Interventions for ARDS [ Ans: ] - endotracheal intubation
and mechanical ventilation with positive end-expiratory
pressure or continuous positive airway pressure
- drug therapy
- nutrition therapy
- fluid therapy
- case management
Chest trauma [ Ans: ] includes
- pulmonary contusion
- rib fracture
, - flail chest
- pneumothorax
- tension pneumothorax
- hemothorax
- tracheobronchial trauma
S+S of ARDS [ Ans: ] - severe dyspnea
- hypoxia
- decreased lung compliance
- diffuse pulmonary infiltrates
Etiology of ARDS [ Ans: ] - develops from a variety of
direct/indirect lung injuries
- most common cause of ARDS is sepsis
- neutrophils are attracted and release mediators which
produce changes in the lungs
- causes increased pulmonary capillary membrane
permeability and pulmonary artery vasoconstriction
exudative phase of ARDS [ Ans: ] - 1-7 days after direct
lung injury or host insult
Answers (100% Correct Answers) Already
Graded A+
ARDS [ Ans: ] - acute respiratory distress syndrome
- sudden progressive form of acute respiratorty failure
- alveolar capillary interface becomes damaged and more
permeable to intravascular fluid
- alveoli fill with fluid
- hypoxia that persists even when oxygen is administered
at 100%
- decreased pulmonary compliance
- dyspnea
- non-cardiac associated bilateral pulmonary edema
- dense pulmonary infiltrates seen on xray
causes of lung injury in ARDS [ Ans: ] - systemic
inflammatory response is the common pathway
- intrinsically the alveolar-capillary membrane is injured
from conditions such as sepsis and shock
,- extrinsically the alveolar-capillary membrane is injured
from conditions such as aspiration or inhalation injury
diagnostic assessment of ARDS [ Ans: ] - lower PaO2 value
on ABG
- poor response to refractory hypoxemia
- ground-glass appearance to chest xray
- no cardiac involvement on ECG
- low to normal PCWP
Interventions for ARDS [ Ans: ] - endotracheal intubation
and mechanical ventilation with positive end-expiratory
pressure or continuous positive airway pressure
- drug therapy
- nutrition therapy
- fluid therapy
- case management
Chest trauma [ Ans: ] includes
- pulmonary contusion
- rib fracture
, - flail chest
- pneumothorax
- tension pneumothorax
- hemothorax
- tracheobronchial trauma
S+S of ARDS [ Ans: ] - severe dyspnea
- hypoxia
- decreased lung compliance
- diffuse pulmonary infiltrates
Etiology of ARDS [ Ans: ] - develops from a variety of
direct/indirect lung injuries
- most common cause of ARDS is sepsis
- neutrophils are attracted and release mediators which
produce changes in the lungs
- causes increased pulmonary capillary membrane
permeability and pulmonary artery vasoconstriction
exudative phase of ARDS [ Ans: ] - 1-7 days after direct
lung injury or host insult
