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APEA- Psych Pharm EXAM 2025 COMPLETE EXAM QUESTIONS AND VERIFIED ANSWERS |RECENTLY TESTING REAL EXAM QUESTIONS| 100% SOLVED!!

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APEA- Psych Pharm EXAM 2025 COMPLETE EXAM QUESTIONS AND VERIFIED ANSWERS |RECENTLY TESTING REAL EXAM QUESTIONS| 100% SOLVED!!

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APEA- Psych Pharm
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APEA- Psych Pharm










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Institution
APEA- Psych Pharm
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APEA- Psych Pharm

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Uploaded on
November 30, 2025
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Written in
2025/2026
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APEA- Psych Pharm EXAM 2025 COMPLETE
EXAM QUESTIONS AND VERIFIED ANSWERS
|RECENTLY TESTING REAL EXAM QUESTIONS|
100% SOLVED!!

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Terms in this set (56)


Bupropion (Wellbutrin), seizure disorder.
used in the treatment of
depression, should NOT
be administered to a
patient with:
alcoholism.
attention deficit disorder.
hypertension.
seizure disorder.

First-generation tardive dyskinesia.
antipsychotics (e.g.,
haloperidol [Haldol]) are
associated with side
effects including:
sedation.
tardive dyskinesia.
weight gain.
weight loss.

,If methylphenidate is Bupropion (Wellbutrin)
contraindicated, which of
the following medications
is recommended for
treatment of ADHD in an
adult?
Bupropion (Wellbutrin)
Clonazepam (Klonopin)
Fluoxetine (Prozac)
Phenelzine (Nardil)

Therapeutic drug levels have variable bioavailability, are lipophilic and highly
and predictability of protein bound.
clinical response are
difficult to determine in
first-generation
antipsychotics (e.g.,
haloperidol [Haldol])
because they:
are poorly absorbed in
the intestines, hydrophilic
and highly protein bound.
are highly bioavailable
and poorly protein bound.
have a decreased first
pass effect, are
hydrophilic and poorly
protein bound.
have variable
bioavailability, are
lipophilic and highly
protein bound.

, Guanfacine (Intuniv), used alpha-2-adrenergic agonist.
in the treatment of
attention deficit disorder,
is classified as a(n):
alpha-2-adrenergic
agonist.
selective norepinephrine
reuptake inhibitor.
sympathomimetic/stimulan
t.
tricyclic antidepressant.

Suvorexant (Belsomra), blocking the signals of orexin chemicals in the
used in the treatment of hypothalamus.
insomnia, induces sleep
by:
activating melatonin
receptors.
blocking the signals of
orexin chemicals in the
hypothalamus.
increasing the catabolism
of tryptophan.
raising the levels of
gamma-aminobutyric acid
(GABA).
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