UWSA1 Block 2 – Questions Correctly Answered By
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Terms in this set (56)
degradation of type 2 osteoarthritis
collagen and
proteoglycans within replaced by water
articular cartilage
sympathetic
sweating.. symp?
parasympathetic?
post ganglionic cholinergic neurons
Serum concentrations: concentrations in seminiferous tubules and epididymis
Testosterone and LH = are regulated by androgen binding protein
normal
- if it isnt made = low testosterone in this area
testosterone concentration
in seminiferous tubules and - androgen binding protein made at sertoli cells
epididymis is low
- thalassemia
- Iron deficiency
microcytic anemias - Anemia of chronic disease
- Sideroblastic
- Lead poisoning
inhibits excessive cartilage proliferation
mutation? inhibits too much
FGFR3 normal function?
symptoms: macrocephaly, shortened limbs/digits,
midface hypoplasia
bullous: Staph Aureus
bullous vs non bullous
causes of impetigo
non bullous: Staph Aureus, Strep pyogens
, 1) known antigen added to well
2) patients serum is added
-- antigen specific antibodies bind the antigen
3) plate is washed = remove unbound antibodies
indirect ELISA 4) anti-human IgG antibodies coupled to an enzyme
(peroxidase) and added to bind to the antigen bound
antibodies
5) plate is washed
6) substrate added to signal for color change
portal hypertension, where - esophageal varices: left gastric vein and azygous
is the congestion - splenomegaly: splenic vein and
- esophageal varices - ascites: paraumbilical vein and smal epigastric veins
- splenomegaly
- ascites
pseudohypoparathyroidism:
- mom imprinting
- Albright hereditary osteodystrophy (short 4th/5th
digits, short, obesity)
pseudohypoparathyroidism - end organ resistance
- high PTH
vs - Gs gene mutation alpha subunit
pseudopseudohypoparath Pseudopseudohypoparathyroidism
yroidism - father imprinting
- McCune albright
- Gq gene mutation
- no end organ resistance
- normal PTH
sweat glands: CFTR reabsorbs Cl- and enhances Na+
Cystic Fibrosis sweat
- mutation: increased Na+ and Cl- in sweat
glands vs
intestinal/respiratory
resp/intestinal: CFTR secretes chloride and inhibits Na+
epithelium
- mutation: low luminal CL-, increased Na+ absorption
Nitrates and beta blockers EDV:
BP: decrease
EDV Contractility:
BP HR:
Contractility Ejection time:
HR
Ejection TIme
Expert
Save
Terms in this set (56)
degradation of type 2 osteoarthritis
collagen and
proteoglycans within replaced by water
articular cartilage
sympathetic
sweating.. symp?
parasympathetic?
post ganglionic cholinergic neurons
Serum concentrations: concentrations in seminiferous tubules and epididymis
Testosterone and LH = are regulated by androgen binding protein
normal
- if it isnt made = low testosterone in this area
testosterone concentration
in seminiferous tubules and - androgen binding protein made at sertoli cells
epididymis is low
- thalassemia
- Iron deficiency
microcytic anemias - Anemia of chronic disease
- Sideroblastic
- Lead poisoning
inhibits excessive cartilage proliferation
mutation? inhibits too much
FGFR3 normal function?
symptoms: macrocephaly, shortened limbs/digits,
midface hypoplasia
bullous: Staph Aureus
bullous vs non bullous
causes of impetigo
non bullous: Staph Aureus, Strep pyogens
, 1) known antigen added to well
2) patients serum is added
-- antigen specific antibodies bind the antigen
3) plate is washed = remove unbound antibodies
indirect ELISA 4) anti-human IgG antibodies coupled to an enzyme
(peroxidase) and added to bind to the antigen bound
antibodies
5) plate is washed
6) substrate added to signal for color change
portal hypertension, where - esophageal varices: left gastric vein and azygous
is the congestion - splenomegaly: splenic vein and
- esophageal varices - ascites: paraumbilical vein and smal epigastric veins
- splenomegaly
- ascites
pseudohypoparathyroidism:
- mom imprinting
- Albright hereditary osteodystrophy (short 4th/5th
digits, short, obesity)
pseudohypoparathyroidism - end organ resistance
- high PTH
vs - Gs gene mutation alpha subunit
pseudopseudohypoparath Pseudopseudohypoparathyroidism
yroidism - father imprinting
- McCune albright
- Gq gene mutation
- no end organ resistance
- normal PTH
sweat glands: CFTR reabsorbs Cl- and enhances Na+
Cystic Fibrosis sweat
- mutation: increased Na+ and Cl- in sweat
glands vs
intestinal/respiratory
resp/intestinal: CFTR secretes chloride and inhibits Na+
epithelium
- mutation: low luminal CL-, increased Na+ absorption
Nitrates and beta blockers EDV:
BP: decrease
EDV Contractility:
BP HR:
Contractility Ejection time:
HR
Ejection TIme
