UWSA1 – Questions With Objective Solutions
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Terms in this set (451)
if you're giving parenteral via central line
nutrition (PN) for more than
48 hrs, how does it have to
be delivered?
why must TPN be given high osmotic load
through a central line?
greatest risk for PN? central line associated bloodstream infection (CLABSI)
coag negative staph - bold
common bugs causing
staph aureas
CLABSI?
GN like klebsiella and pseudomonas
common fungal cause of candida
CLABSI?
is a patient requiring PN at no, you chose this answer
risk for cholelithiasis after 7-
10 days?
how long does PN need to 2 weeks
be used for cholelithiasis to
become a risk?
is hyperglycemia common yes, these pt are twice as common to have hyperG
in PN?
are blood stream infections yes
are common and serious
complication of PN?
59 year old with non small superior vena cava syndrome
cell lung cancer has
headaches that worsen
leaning forward, JVD,
conjunctival injection; what
is dx?
, no; heart works fine
is there peripheral edema
in SVC syndrome?
two cancers causing SVC lung and lyphoma
syndrome?
radiation - answer
treatment for SVC endovenous stenting
syndrome?
both as paliaitive measures
pericardiocentesis is useful
for malignancy when there
is obviously systemic
venous congestion; this
question said no peripehral
or abdomianl edema,
suggesting it was localized
to the svc; pic
56 year old has 6 weeks of polymyositis
proximal muscle weakness;
proximal and symmetric in
bilateral UE nd LE; CK,
LDH, AST 250 ALT 140 are
elvated; waht is dx?
what is pain like in minimal
polymyositis?
what are both poly and malignancy
dermatomyositis associated
with?
what does bx show in endomysial infiltrate
polymyositis? patchy necrosis
is AST a muscle enzyme yes, threw you off cause it was elevated
too?
is ALS weakness distal or distal
proximal usually?
what is CK like in ALS? normal
what is CK like in MG? normal
,what cause of weakness myotonic MD
will have a slowed
relaxation phase of muscle
contraction?
weakness with family hx is myotonic MD
likely?
is there weakness in no
polymyalgia rheumatica?
creatine kinase
LDH
muscle enzymes
aldolase
AST
found in many tissues- skeletal muscle, heart, kidneys,
Where is AST found?
brain
can hypoT cause weakness, yes, but will have hypoT sxs too
pain, and enzyme
elevation?
can ALT and AST both be yes - they gave a noraml Tbili to show you it wasnt liver
elevated in muscle origin too
necrosis?
41 year old has minute temporal lobe epilepsy
episodes where hes
unresponsive and has
fumbling hand movements;
what is dx?
what kind of seizure causes focal seizures (which can proceed geralized)
sensory eg olfactory or
psych sxs eg fear?
what are sensory and psych auras
sxs with focal seizures
called?
what kind of seizures was focal with impaired awareness (previously known as
this guy having who was generalized partial)
just staring and doing stuff
with his hands?
, most common way focal with impaired awareness
temporal lobe epilepsy
presents?
examples of automatisms? lip smacking or hand movements
epileptiform focus over the
EEG finding in temporal temproal region
lobe epilepsy?
is there postictal confusion yes
in temporal lobe epilepsy?
Is there a postictal period no
in absence seizures?
are TIAs assocaited with negative
negative eg weakness,
droop or positive eg
movement sxs?
common location of focal temporla lobe
with imparied awareness
epilpeys?
two key features of automatisms and post ictal confusion
temporal lobe epilepsy?
lady with DM has freuqent neurogenic bladder
dribbling and leakage at
night; she has decreased
vibratory sense in both
ankles; post void volume is
300 ml; what is dx?
what kind of incontinence is OVERFLOW incontinence
neurogenic bladder?
does SANS or PANS PANS
control bladder voiding?
first line neurogenic bethanacol (musc agonist)
bladder drug?
timed voids
lifestyle treatment for
suprapubic pressure
neurogenic bladder ?
double voiding
Save
Terms in this set (451)
if you're giving parenteral via central line
nutrition (PN) for more than
48 hrs, how does it have to
be delivered?
why must TPN be given high osmotic load
through a central line?
greatest risk for PN? central line associated bloodstream infection (CLABSI)
coag negative staph - bold
common bugs causing
staph aureas
CLABSI?
GN like klebsiella and pseudomonas
common fungal cause of candida
CLABSI?
is a patient requiring PN at no, you chose this answer
risk for cholelithiasis after 7-
10 days?
how long does PN need to 2 weeks
be used for cholelithiasis to
become a risk?
is hyperglycemia common yes, these pt are twice as common to have hyperG
in PN?
are blood stream infections yes
are common and serious
complication of PN?
59 year old with non small superior vena cava syndrome
cell lung cancer has
headaches that worsen
leaning forward, JVD,
conjunctival injection; what
is dx?
, no; heart works fine
is there peripheral edema
in SVC syndrome?
two cancers causing SVC lung and lyphoma
syndrome?
radiation - answer
treatment for SVC endovenous stenting
syndrome?
both as paliaitive measures
pericardiocentesis is useful
for malignancy when there
is obviously systemic
venous congestion; this
question said no peripehral
or abdomianl edema,
suggesting it was localized
to the svc; pic
56 year old has 6 weeks of polymyositis
proximal muscle weakness;
proximal and symmetric in
bilateral UE nd LE; CK,
LDH, AST 250 ALT 140 are
elvated; waht is dx?
what is pain like in minimal
polymyositis?
what are both poly and malignancy
dermatomyositis associated
with?
what does bx show in endomysial infiltrate
polymyositis? patchy necrosis
is AST a muscle enzyme yes, threw you off cause it was elevated
too?
is ALS weakness distal or distal
proximal usually?
what is CK like in ALS? normal
what is CK like in MG? normal
,what cause of weakness myotonic MD
will have a slowed
relaxation phase of muscle
contraction?
weakness with family hx is myotonic MD
likely?
is there weakness in no
polymyalgia rheumatica?
creatine kinase
LDH
muscle enzymes
aldolase
AST
found in many tissues- skeletal muscle, heart, kidneys,
Where is AST found?
brain
can hypoT cause weakness, yes, but will have hypoT sxs too
pain, and enzyme
elevation?
can ALT and AST both be yes - they gave a noraml Tbili to show you it wasnt liver
elevated in muscle origin too
necrosis?
41 year old has minute temporal lobe epilepsy
episodes where hes
unresponsive and has
fumbling hand movements;
what is dx?
what kind of seizure causes focal seizures (which can proceed geralized)
sensory eg olfactory or
psych sxs eg fear?
what are sensory and psych auras
sxs with focal seizures
called?
what kind of seizures was focal with impaired awareness (previously known as
this guy having who was generalized partial)
just staring and doing stuff
with his hands?
, most common way focal with impaired awareness
temporal lobe epilepsy
presents?
examples of automatisms? lip smacking or hand movements
epileptiform focus over the
EEG finding in temporal temproal region
lobe epilepsy?
is there postictal confusion yes
in temporal lobe epilepsy?
Is there a postictal period no
in absence seizures?
are TIAs assocaited with negative
negative eg weakness,
droop or positive eg
movement sxs?
common location of focal temporla lobe
with imparied awareness
epilpeys?
two key features of automatisms and post ictal confusion
temporal lobe epilepsy?
lady with DM has freuqent neurogenic bladder
dribbling and leakage at
night; she has decreased
vibratory sense in both
ankles; post void volume is
300 ml; what is dx?
what kind of incontinence is OVERFLOW incontinence
neurogenic bladder?
does SANS or PANS PANS
control bladder voiding?
first line neurogenic bethanacol (musc agonist)
bladder drug?
timed voids
lifestyle treatment for
suprapubic pressure
neurogenic bladder ?
double voiding
