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NUR 430 Final Exam Questions With Correct Answers Updated Set.

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resuscitation phase of burns - Answer cardiopulmonary instability, life-threatening airway and breathing problems, hypovolemia (until about day 4) - o2 alterations from CO poisoning, upper airway obstruction, chemical pneumonitis - focus on ABCs - assess breath sounds, administer O2, monitor HbCO levels, elevate HOB, prepare for intubation - fluid resuscitation using parkland formula. can give colloids one capillary leak seals to decrease edema - Answer interventions for resuscitative phase acute care phase of burns - Answer - Lasts until complete wound closure (begins day 4 and ends in about 2 weeks) - Inflammatory response with large fluid shifts - Focus on wound cleaning and infection prevention. Infection most common cause of death after resuscitation phase - Pain management, prevent hypothermia rehab phase of burns - Answer focus on healing, scar management, and psychosocial issues - contractures, pressure ulcers, DVT biosynthetics (biobrane) - Answer porous allowing for absorption of topical antibiotics, adhere to wound fibrin synthetic (tegaderm) - Answer semi occlusive, coverage of donor sites, fluid may collect hydrocolloidal (duoderm) - Answer occlusive, partial thickness wound, cannot see wound, odor and exudate, but rapid healing and decreased pain 4 ml x TBSA x weight (kg) give 50% of calculated fluid need in 8 hours, 25% in seconds 8 hours, and 25% in third eight hours - Answer parkland formula and how it is separated - facial burns

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NUR 430 Final Exam Questions With
Correct Answers 2025-2026 Updated
Set.
resuscitation phase of burns - Answer cardiopulmonary instability, life-threatening airway and
breathing problems, hypovolemia (until about day 4)

- o2 alterations from CO poisoning, upper airway obstruction, chemical pneumonitis



- focus on ABCs

- assess breath sounds, administer O2, monitor HbCO levels, elevate HOB, prepare for
intubation

- fluid resuscitation using parkland formula. can give colloids one capillary leak seals to decrease
edema - Answer interventions for resuscitative phase



acute care phase of burns - Answer - Lasts until complete wound closure (begins day 4 and
ends in about 2 weeks)

- Inflammatory response with large fluid shifts

- Focus on wound cleaning and infection prevention. Infection most common cause of death
after resuscitation phase

- Pain management, prevent hypothermia



rehab phase of burns - Answer focus on healing, scar management, and psychosocial issues

- contractures, pressure ulcers, DVT



biosynthetics (biobrane) - Answer porous allowing for absorption of topical antibiotics, adhere
to wound fibrin



synthetic (tegaderm) - Answer semi occlusive, coverage of donor sites, fluid may collect



hydrocolloidal (duoderm) - Answer occlusive, partial thickness wound, cannot see wound,
odor and exudate, but rapid healing and decreased pain



4 ml x TBSA x weight (kg)

, - face and skin may appear cherry red

- look for impaired oxygenation or upper airway obstruction

- singed eyebrows and nose hair, soot in throat - Answer how do we know to intubate with
burns?



- need O2 even if O2 stat is okay

- o2 can't bind to hgb

- CO poisoning can cause headache, CNS disturbances, unresponsiveness

- need high flow 100% o2 - Answer carbon monoxide poisoning considerations



o Hyperkalemia from breakdown of muscles and tissue, or hypokalemia because of overdilution
of vascular space and hyponatremia - Answer electrolyte concerns in burns



chemical burns - Answer - acids, alkalis, compounds. either pH or concentration determines
injury

- overall treatment is copious flushing or irrigation of water to the burn



- remove source without injuring rescuer and make sure scene is safe

- much more serious than what is observed because damage to inner tissues

- monitor ECG, cardiac dysrhythmias common - Answer electrical burn considerations



initial stage of shock - Answer - decreased tissue perfusion leads to decreased O2 and
decreased cardiac output and increased lactic acid

- at this stage many people don't seek treatment



compensatory stage of shock - Answer - HR increased to try to increase cardiac output and get
blood to where it needs to go. SNS kicks in and lactic acid continues to increase

- need to treat aggressively in this stage



- SNS puts out epi, vasoconstriction, increased HR and contractility, bronchodilation

- RAAS (vasoconstriction and retention of Na and H20)

- BP low due to low volume


- O2 down

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