Correct Answers 2025-2026 Updated
Set.
resuscitation phase of burns - Answer cardiopulmonary instability, life-threatening airway and
breathing problems, hypovolemia (until about day 4)
- o2 alterations from CO poisoning, upper airway obstruction, chemical pneumonitis
- focus on ABCs
- assess breath sounds, administer O2, monitor HbCO levels, elevate HOB, prepare for
intubation
- fluid resuscitation using parkland formula. can give colloids one capillary leak seals to decrease
edema - Answer interventions for resuscitative phase
acute care phase of burns - Answer - Lasts until complete wound closure (begins day 4 and
ends in about 2 weeks)
- Inflammatory response with large fluid shifts
- Focus on wound cleaning and infection prevention. Infection most common cause of death
after resuscitation phase
- Pain management, prevent hypothermia
rehab phase of burns - Answer focus on healing, scar management, and psychosocial issues
- contractures, pressure ulcers, DVT
biosynthetics (biobrane) - Answer porous allowing for absorption of topical antibiotics, adhere
to wound fibrin
synthetic (tegaderm) - Answer semi occlusive, coverage of donor sites, fluid may collect
hydrocolloidal (duoderm) - Answer occlusive, partial thickness wound, cannot see wound,
odor and exudate, but rapid healing and decreased pain
4 ml x TBSA x weight (kg)
, - face and skin may appear cherry red
- look for impaired oxygenation or upper airway obstruction
- singed eyebrows and nose hair, soot in throat - Answer how do we know to intubate with
burns?
- need O2 even if O2 stat is okay
- o2 can't bind to hgb
- CO poisoning can cause headache, CNS disturbances, unresponsiveness
- need high flow 100% o2 - Answer carbon monoxide poisoning considerations
o Hyperkalemia from breakdown of muscles and tissue, or hypokalemia because of overdilution
of vascular space and hyponatremia - Answer electrolyte concerns in burns
chemical burns - Answer - acids, alkalis, compounds. either pH or concentration determines
injury
- overall treatment is copious flushing or irrigation of water to the burn
- remove source without injuring rescuer and make sure scene is safe
- much more serious than what is observed because damage to inner tissues
- monitor ECG, cardiac dysrhythmias common - Answer electrical burn considerations
initial stage of shock - Answer - decreased tissue perfusion leads to decreased O2 and
decreased cardiac output and increased lactic acid
- at this stage many people don't seek treatment
compensatory stage of shock - Answer - HR increased to try to increase cardiac output and get
blood to where it needs to go. SNS kicks in and lactic acid continues to increase
- need to treat aggressively in this stage
- SNS puts out epi, vasoconstriction, increased HR and contractility, bronchodilation
- RAAS (vasoconstriction and retention of Na and H20)
- BP low due to low volume
- O2 down