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Test Bank For Rubin's Pathology: Clinicopathologic Foundations of Medicine 7th Edition by Strayer ISBN:9781451183900, All 34 Chapters Covered, Verified Latest Edition

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Test Bank For Rubin's Pathology: Clinicopathologic Foundations of Medicine 7th Edition by Strayer ISBN:9781451183900, All 34 Chapters Covered, Verified Latest Edition

Institution
Rubin\\\'s Pathology
Course
Rubin\\\'s Pathology

Content preview

,Test Bank Rubin's Pathology: Clinicopathologic Foundations of Medicine 7th Edition
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Table of Contents:
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Chapter 1: Cell Adaptation, Injury and Death
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Chapter 2: Inflammation
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Chapter 3: Repair, Regeneration and Fibrosis
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Chapter 4: Immunopathology
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Chapter 5: Neoplasia
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Chapter 6: Developmental and Genetic Diseases
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Chapter 7: Hemodynamic Disorders
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Chapter 8: Environmental and Nutritional Pathology
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Chapter 9: Infectious and Parasitic Diseases
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Section II: Pathogenesis of Systemic Conditions Expandable section
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Chapter 10: Aging
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Chapter 11: Systemic Autoimmune Diseases
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Chapter 12: Sepsis
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Chapter 13: Obesity and Diabetes Mellitus
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Chapter 14: The Pathology of Pregnancy
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Chapter 15: The Amyloidoses
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Section III: Diseases of Individual Organ SystemsExpandable section
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Chapter 16: Blood Vessels
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Chapter 17: The Heart
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Chapter 18: The Respiratory System
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Chapter 19: The Gastrointestinal Tract
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Chapter 20: The Liver and Biliary System
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Chapter 21: The Pancreas
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Chapter 22: The Kidney
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Chapter 23: The Lower Urinary Tract and Male Reproductive System
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Chapter 24: The Female Reproductive System and Peritoneum
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Chapter 25: The Breast
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Chapter 26: Hematopathology
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Chapter 27: The Endocrine System
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Chapter 28: The Skin
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Chapter 29: The Head and Neck
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Chapter 30: Bones, Joints and Soft Tissue
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Chapter 31: Skeletal Muscle and Peripheral Nervous System
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Chapter 32: The Central Nervous System
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Chapter 33: The Eye
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Chapter 34: Forensic Pathology
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,Rubin's Pathology: Clinicopathologic Foundations of Medicine
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Chapter 1: Cell Adaptation, Injury and Death
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Ischemia and other toxic injuries increase the accumulation of intracellular calcium as a result
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1. of:
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A) release of stored calcium from the mitochondria.
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B) improved intracellular volume regulation. ff ff ff




C) decreased influx across the cell membrane. ff ff ff ff ff



D) attraction of calcium to fatty infiltrates. ff ff ff ff ff



The patient is found to have liver disease, resulting in the removal of a lobe of his liver.
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2. Adaptation to the reduced size of the liver leads to _
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A) metaplasia
B) organ atrophy ff



C) compensatory hyperplasia ff



D) physiologic hypertrophy ff




A person eating peanuts starts choking and collapses. His airway obstruction is
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partially cleared, but he remains hypoxic until he reaches the hospital. The prolonged
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3. cell hypoxia caused a cerebral infarction and resulting _ in the brain.
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A) caspase activation ff



B) coagulation necrosis ff



C) rapid phagocytosis ff



D) protein p53 deficiency ff ff



Bacteria and viruses cause cell damage by
ff ff ff ff ff ff , which is unique from the intracellular
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4. damage caused by other injurious agents.
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A) disrupting the sodium/potassium ATPase pump ff ff ff ff



B) interrupting oxidative metabolism processes ff ff ff



C) replicating and producing continued injury ff ff ff ff




D) decreasing protein synthesis and function ff ff ff ff



The patient has a prolonged interruption in arterial blood flow to his left kidney, causing
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5. hypoxic cell injury and the release of free radicals. Free radicals damage cells by:
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A) destroying phospholipids in the cell membrane. ff ff ff ff ff



B) altering the immune response of the cell.
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C) disrupting calcium storage in the cell. ff ff ff ff ff



D) inactivation of enzymes and mitochondria. ff ff ff ff

, 6. Injured cells have impaired flow of substances through the cell membrane as a result of:
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A) increased fat load. ff ff



B) altered permeability. ff



C) altered glucose utilization. ff ff



D) increased surface receptors. ff ff



7. Reversible adaptive intracellular responses are initiated by:
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A) stimulus overload. ff



B) genetic mutations. ff



C) chemical messengers. ff



D) mitochondrial DNA. ff



8. Injured cells become very swollen as a result of:
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A) increased cell protein synthesis. ff ff ff



B) altered cell volume regulation. ff ff ff



C) passive entry of potassium into the cell. ff ff ff ff ff ff



D) bleb formation in the plasma membrane.
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A diabetic patient has impaired sensation, circulation, and oxygenation of his feet. He steps
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on a piece of glass, the wound does not heal, and the area tissue becomes necrotic. The
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9. necrotic cell death is characterized by:
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A) rapid apoptosis. ff



B) cellular rupture. ff




C) shrinkage and collapse. ff ff



D) chronic inflammation. ff



A 99-year-old woman has experienced the decline of cell function associated with age. A
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10. group of theories of cellular aging focus on programmed:
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A) changes with genetic influences. ff ff ff



B) elimination of cell receptor sites. ff ff ff ff



C) insufficient telomerase enzyme. ff ff



D) DNA mutation or faulty repair.
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An 89-year-old female patient has experienced significant decreases in her mobility and
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stamina during a 3-week hospital stay for the treatment of a femoral head fracture. Which of
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the following phenomena most likely accounts for the patients decrease in muscle function
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11. that underlies her reduced mobility?
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A) Impaired muscle cell metabolism resulting from metaplasiaff ff ff ff ff ff



B) Dysplasia as a consequence of inflammation during bone remodeling
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C) Disuse atrophy of muscle cells during a prolonged period of immobility
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Institution
Rubin\\\'s Pathology
Course
Rubin\\\'s Pathology

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