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NR 507 Endocrine System, NR507 Midterm, NR 507 Final EXAM NEWEST 2025 QUESTIONS AND CORRECT DETAILED ANSWERS (VERIFIED ANSWERS) |ALREADY GRADED A+

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NR 507 Endocrine System, NR507 Midterm, NR 507 Final EXAM NEWEST 2025 QUESTIONS AND CORRECT DETAILED ANSWERS (VERIFIED ANSWERS) |ALREADY GRADED A+

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NR 507 Endocrine System, NR507 Midterm, NR
507 Final EXAM NEWEST 2025 QUESTIONS AND
CORRECT DETAILED ANSWERS (VERIFIED
ANSWERS) |ALREADY GRADED A+

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Terms in this set (352)


False.
The only way for a
hormone to work is The endocrine cell can also secrete hormones that
through the blood stream. target cells directly close it. When this occurs, this
type of secretion is known as paracrine signaling.

True.
Lipid-derived hormones
cross the cell membrane
This statement is true. Lipid-derived hormones cross
and bind to receptors in
the cell membrane and bind to receptors in the cell's
the cell's cytoplasm
cytoplasm.

True.
The anterior lobe of the
pituitary produces its own
This statement is true. The anterior lobe of the
hormones
pituitary produces its own hormones

To be considered a lipid False.
hormone, it must be
bound to a protein and it It must be bound to a protein and it bind to receptors
must bind to receptors on the cell membrane rather than inside the cell
inside the cell membrane membrane.

,The pineal gland is True.
responsible for regulating
the sleep-wake cycle

False.
The posterior lobe of the
pituitary produces its own The posterior pituitary receives its hormones
hormones (oxytocin and ADH) from the hypothalamus. The
anterior pituitary produces its own hormones.

Aldosterone promotes This statement is true.
sodium reabsorption and
potassium excretion in the
kidneys.

Elevated Thyroid Hormone
Suppressed TSH
Hyperthyroidism
Enlarged liver
Hand tremors

Decreased thyroid hormone and
Elevated TSH
Hypothyroidism
Fatigue
Diminished deep tendon reflexes

Onset 1<10-20 years
Associated with diabetic ketoacidosis
Symptoms: polyuria, polyphagia, polydipsia


Autoimmune:Genetic and environmental factors,
resulting in gradual process of autoimmune
Type 1 DM
destruction in genetically susceptible individuals
Nonautoimmune:Unknown
Strong association with HLA-DQA and HLA-DQB
genes


acute complications: Diabetic ketoacidosis

, Usually > 40 years of age
Associated with hyperosmolar nonketotic coma
Symptoms: weakness, weight loss, infections


Results from genetic susceptibility (polygenic)
combined with environmental determinants and other
risk factors
Type 2 DM
Inherited defects in beta-cell mass and function
combined with peripheral tissue insulin resistance
Associated with long-duration obesity


strong genetic association


Acute complications: Hyperosmolar nonketotic coma

alpha cells responsible for secreting glucagon

responsible for secreting insulin and amylin
beta cells
inhibits glucagon secretion

delta cells responsible for secreting gastrin and somatostatin

secrete pancreatic polypeptide that stimulates gastric
F (PP) Cells
secretions and antagonizes cholecystokinin.

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