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Rasmussen Pathophysiology Exam 2 (2025) | 100% Verified Questions & Correct Answers

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Rasmussen Pathophysiology Exam 2 (2025) | 100% Verified Questions & Correct Answers

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Rasmussen Pathophysiology Exam 2 (2025) | 100% Verified Questions &
Correct Answers




1. If a patient with type 1 diabetes has increased adipose tissue due to obesity,
what potential effect might this have on their glucose levels?

It will decrease blood glucose levels by enhancing insulin sensitivity.

It may stabilize blood glucose levels by increasing insulin
production.

It may lead to higher blood glucose levels due to reduced glucose
absorption by adipose tissue.

It will have no effect on blood glucose levels.

2. Describe how excessive ADH affects blood osmolality and urine osmolality
in SIADH.

Excessive ADH causes dehydration and increased serum osmolality.

Excessive ADH causes increased serum osmolality and decreased
urine osmolality.

Excessive ADH leads to water reabsorption in the renal tubules,
resulting in low serum osmolality and high urine osmolality.

Excessive ADH has no effect on serum or urine osmolality.

3. What autoimmune condition is primarily responsible for causing primary
hypothyroidism?

Addison's disease

Graves' disease

,Hashimoto's disease

, Cushing's syndrome

4. Describe the role of bicarbonate ions in the kidney tubules' response to
fever.

Kidney tubules secrete bicarbonate ions to increase acidity.

Kidney tubules reabsorb more bicarbonate ions to help neutralize
excess acids.

Kidney tubules convert bicarbonate into glucose for energy.

Kidney tubules do not affect bicarbonate levels during fever.

5. All of the following are associated with hypothyroidism, except:

lethargy

weight loss

hair loss

cold intolerance

6. Which hormone is produced by the anterior pituitary gland that stimulates
the adrenal cortex?

Luteinizing hormone (LH)

Thyroid-stimulating hormone (TSH)

Adrenocorticotropic hormone (ACTH)

Growth hormone (GH)

7. A patient with uncontrolled diabetes presents with excessive hunger
despite recent meals. What underlying mechanism could explain this
symptom?

, Inadequate insulin action leading to unutilized glucose.

Dehydration resulting in increased thirst and hunger.

Increased metabolism due to hyperthyroidism.

Excessive production of glucagon causing increased appetite.

8. If a patient presents with symptoms of mental deterioration and blindness,
what infectious disease should be considered as a possible cause?

Tuberculosis

Hepatitis

HIV/AIDS

Syphilis

9. Describe the potential long-term effects of untreated childhood gigantism.

Untreated childhood gigantism only affects height without other
health implications.

Untreated childhood gigantism results in increased muscle strength
and endurance.

Untreated childhood gigantism has no significant long-term effects.

Untreated childhood gigantism can lead to lower life expectancy
and weak limbs.

10. If a patient is diagnosed with a primary endocrine disorder affecting the
thyroid gland, what would be the most likely cause of their symptoms?

Insufficient stimulation from the pituitary gland

Excessive hormone production from the adrenal glands

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