BKAT Study
Anterior leads - ANS-V1 - V4. LAD lesion.
Aspiration precautions - ANS-HOB > 30, common handwashing, check feeding tube, investigate
for residuals, swallow have a look at, ETT cuff at appropriate tiers
Assess for GI bleed - ANS-Coffee floor emesis or drainage
Best vasopressors for septic shock - ANS-Neorepinephrine, Epinepherine, Vasopressin
Biphasic settings for defibrillation - ANS-a hundred and fifty J
Cardiac enzymes - ANS-Troponins, CK-MB, and CK
Causes of expanded CVP - ANS-RV failure, tricuspid stenosis or regurg, pericardial effusion,
constrictive pericarditis, advanced vena cava obstruction, fluid overload, hyperdynamic flow,
excessive PEEP setting
CAUTI prevention - ANS-Hand washing, aseptic insertion method, common pericare, no
structured loops, no kinked tubing
Changes in CK - ANS-Rise: 3-6 hours
Peak: 24 hours
Normal: 3-four days
Changes in CK-MB - ANS-Released after myocardial necrosis. Specific for myocardial damage.
Rise: 3-12 hours
Peak: 24 hours
Normal: 2-three days
Chest trauma headaches - ANS-Pneumothorax
CLABSI prevention - ANS-Hand hygiene, chlorhexidine skin prep, full-barrier precautions
(masks, affected person head turned away), avoid femoral vein, take out catheters as soon as
possible, every day assessment of catheters
Clinical presentation of DI - ANS-Polyuria, polydipsia, altered everyday bowel habits, signs of
dehydration
, Common conditions that motive a murmur - ANS-Aortic dissection, aortic regurgitation (each
acute & continual), mitral valve regurgitation (both acute & continual), mitral valve stenosis
Complications of chest trauma to lungs - ANS-Pneumothorax, PE, pleural effusion, ARDS
Complications of excessive cervical damage/spinal wire - ANS-Respiratory disorder: loss of
feature, loss of force.
Complications of lengthy bone fractures - ANS-Fat emboli, air emboli (PE)
Complications while the usage of thrombolytics - ANS-Allergic response, bleeding/hemorrhage,
stroke
Complications with acute renal failure - ANS-Increased K, extended fluid, HTN
Components of neuro examination - ANS-LOC, intellectual fame, cognitive characteristic, cranial
nerves, motor, sensory, coordination, and reflexes
Critical Illness and Insulin Resistance - ANS-•Insulin resistance sustained by inflammatory
mediators
•Endocrine exhaustion results in relative deficiency of insulin production
Describe "a" wave with CVP - ANS-Represents atrial contraction. Correlates to PR interval.
Describe "c" wave with CVP - ANS-Represents closure of tricuspid valve. Correlates to QRS
complicated.
Describe "v" wave with CVP - ANS-Represents atrial filling. Correlates to TP c programming
language.
Describe CVP waveform - ANS-Three peaks (a, c, v waves) & Two descents (x and y)
Describe Diabetes Insipidus (DI) - ANS-Due to ADH deficiency resulting in big urine output,
immoderate thirst, and hyperosmolality
Describe digoxin toxicity - ANS-Confusion, Irregular pulse, Loss of urge for food, Nausea,
vomiting, diarrhea, Palpitations, Vision changes (blind spots, blurred imaginative and prescient,
adjustments in how colorations appearance, or seeing spots), Decreased focus, Decreased
urine output, Difficulty respiration whilst mendacity down, Excessive midnight urination
Describe drug adjustments to be made for sufferers in renal failure - ANS-Decrease the dose or
increase the for the duration of between doses (no longer cleared as well via the kidneys)
Anterior leads - ANS-V1 - V4. LAD lesion.
Aspiration precautions - ANS-HOB > 30, common handwashing, check feeding tube, investigate
for residuals, swallow have a look at, ETT cuff at appropriate tiers
Assess for GI bleed - ANS-Coffee floor emesis or drainage
Best vasopressors for septic shock - ANS-Neorepinephrine, Epinepherine, Vasopressin
Biphasic settings for defibrillation - ANS-a hundred and fifty J
Cardiac enzymes - ANS-Troponins, CK-MB, and CK
Causes of expanded CVP - ANS-RV failure, tricuspid stenosis or regurg, pericardial effusion,
constrictive pericarditis, advanced vena cava obstruction, fluid overload, hyperdynamic flow,
excessive PEEP setting
CAUTI prevention - ANS-Hand washing, aseptic insertion method, common pericare, no
structured loops, no kinked tubing
Changes in CK - ANS-Rise: 3-6 hours
Peak: 24 hours
Normal: 3-four days
Changes in CK-MB - ANS-Released after myocardial necrosis. Specific for myocardial damage.
Rise: 3-12 hours
Peak: 24 hours
Normal: 2-three days
Chest trauma headaches - ANS-Pneumothorax
CLABSI prevention - ANS-Hand hygiene, chlorhexidine skin prep, full-barrier precautions
(masks, affected person head turned away), avoid femoral vein, take out catheters as soon as
possible, every day assessment of catheters
Clinical presentation of DI - ANS-Polyuria, polydipsia, altered everyday bowel habits, signs of
dehydration
, Common conditions that motive a murmur - ANS-Aortic dissection, aortic regurgitation (each
acute & continual), mitral valve regurgitation (both acute & continual), mitral valve stenosis
Complications of chest trauma to lungs - ANS-Pneumothorax, PE, pleural effusion, ARDS
Complications of excessive cervical damage/spinal wire - ANS-Respiratory disorder: loss of
feature, loss of force.
Complications of lengthy bone fractures - ANS-Fat emboli, air emboli (PE)
Complications while the usage of thrombolytics - ANS-Allergic response, bleeding/hemorrhage,
stroke
Complications with acute renal failure - ANS-Increased K, extended fluid, HTN
Components of neuro examination - ANS-LOC, intellectual fame, cognitive characteristic, cranial
nerves, motor, sensory, coordination, and reflexes
Critical Illness and Insulin Resistance - ANS-•Insulin resistance sustained by inflammatory
mediators
•Endocrine exhaustion results in relative deficiency of insulin production
Describe "a" wave with CVP - ANS-Represents atrial contraction. Correlates to PR interval.
Describe "c" wave with CVP - ANS-Represents closure of tricuspid valve. Correlates to QRS
complicated.
Describe "v" wave with CVP - ANS-Represents atrial filling. Correlates to TP c programming
language.
Describe CVP waveform - ANS-Three peaks (a, c, v waves) & Two descents (x and y)
Describe Diabetes Insipidus (DI) - ANS-Due to ADH deficiency resulting in big urine output,
immoderate thirst, and hyperosmolality
Describe digoxin toxicity - ANS-Confusion, Irregular pulse, Loss of urge for food, Nausea,
vomiting, diarrhea, Palpitations, Vision changes (blind spots, blurred imaginative and prescient,
adjustments in how colorations appearance, or seeing spots), Decreased focus, Decreased
urine output, Difficulty respiration whilst mendacity down, Excessive midnight urination
Describe drug adjustments to be made for sufferers in renal failure - ANS-Decrease the dose or
increase the for the duration of between doses (no longer cleared as well via the kidneys)
