NU 272 – Exam 3 Study Guide Chamberlain University Health & Illness Concepts
Overview with answers
energy |metabolism |- |answersrefers |to |the |chemical |processes |involved |in |converting |carbohydrates, |
fats, |and |proteins |from |the |foods |we |eat |into |the |energy |needed |for |cell |functions
Non-modifiable |risk |factors |- |answersgenetics
Gender
Ethnicity
Life |span |considerations |Infants |obesity |- |answersReview |of |family |history
Life |span |considerations |children/ |adolescents |- |answers-Goiter
-Family |History
-Most |endocrine |issues |with |children |are |autoimmune |or |tumor
Life |span |considerations |Pregnant |women |- |answers-Uterus |becomes |an |endocrine |gland
-gestational |diabetes. |
-Increased |risk |for |thyroid |hormone |production |abnormalities.
Life |consideration |older |adults |- |answers-Nodules |and |irregular |borders |of |Thyroid
-Assess |cognitive |ability |to |answer |questions
-Most |common |metabolic |disorder |osteoporosis
Obesity |Shape |- |answersUpper |body |Apple
Lower |body |Pear
Total |Daily |Energy |Expenditure |(TDEE) |- |answerssum |of |all |the |calories |used |to |perform |physical |
activity, |maintain |basal |metabolism, |and |digest, |absorb, |and |metabolize |food.
Causes |obesity |- |answersimbalance |between |calorie |intake |and |energy |expenditure, |genetics, |
metabolism, |behavior, |environment, |culture, |various |medical |conditions
Energy |nutrients |- |answerscarbohydrates, |fats, |proteins
Regulatory |Nutrients |- |answersvitamins, |minerals, |water
Complete |proteins |- |answersanimal |protein |or |plant |protein |+ |small |amount |of |animal |protein |or |two |
complementary |vegetable |proteins.
adipose |tissue |- |answers90% |of |body |energy |stored |here
, BMI |- |answers▸ratio |of |weight |(in |kilograms) |to |height |(in |meters).
▸also |provides |an |estimation |of |relative |risk |for |diseases |such |as |heart |disease, |diabetes, |and |
hypertension.
Underweight |BMI |- |answers<18.5
normal |BMI |- |answers18.5-24.9
Overweight |BMI |- |answers25-29.9
Obesity |BMI |- |answers30-35 |Class1
35-40 |class |2
Extreme |obesity |40+ |Class |3
Excessive |fat |in |abdomen |- |answersis |an |independent |predictor |of |risk |factors |and |mortality
Obesity |prevention |and |treatment |options |- |answers▸Dietary |modifications
▸Exercise
▸Stress |management
▸Bariatric |surgery
Obesity |treatment |at |what |BMI |- |answers25-30
and |30 |plus
▸Roux-en-Y |gastric |bypass |Surgery |- |answersRestrictive |and |Malabsorptive
sleeve |gastrectomy |- |answersRestrictive
Complications |weight |loss |surgery |- |answers▸Hemorrhage
▸DVT/PE
▸Dumping |Syndrome
▸Bile |reflux
Bowel |Obstruction
Cirrhosis |- |answersEnd |stage |chronic |liver |disease. |It |is |a |progressive, |irreversible |disorder, |eventually |
leading |to |liver |failure.
Most |common |types |of |cirrhosis |- |answersAlcoholic |cirrhosisbiliary |cirrhosis, |post |hepatic |cirrhosis
Biliary |Atresia |- |answersFailed |to |open |or |closed
Biliary |atresia |leads |to |- |answersLeads |to |cholestasis, |cirrhosis, |portal |hypertension, |end |stage |liver |
disease, |and |death |by |2 |years |if |not |treated
Overview with answers
energy |metabolism |- |answersrefers |to |the |chemical |processes |involved |in |converting |carbohydrates, |
fats, |and |proteins |from |the |foods |we |eat |into |the |energy |needed |for |cell |functions
Non-modifiable |risk |factors |- |answersgenetics
Gender
Ethnicity
Life |span |considerations |Infants |obesity |- |answersReview |of |family |history
Life |span |considerations |children/ |adolescents |- |answers-Goiter
-Family |History
-Most |endocrine |issues |with |children |are |autoimmune |or |tumor
Life |span |considerations |Pregnant |women |- |answers-Uterus |becomes |an |endocrine |gland
-gestational |diabetes. |
-Increased |risk |for |thyroid |hormone |production |abnormalities.
Life |consideration |older |adults |- |answers-Nodules |and |irregular |borders |of |Thyroid
-Assess |cognitive |ability |to |answer |questions
-Most |common |metabolic |disorder |osteoporosis
Obesity |Shape |- |answersUpper |body |Apple
Lower |body |Pear
Total |Daily |Energy |Expenditure |(TDEE) |- |answerssum |of |all |the |calories |used |to |perform |physical |
activity, |maintain |basal |metabolism, |and |digest, |absorb, |and |metabolize |food.
Causes |obesity |- |answersimbalance |between |calorie |intake |and |energy |expenditure, |genetics, |
metabolism, |behavior, |environment, |culture, |various |medical |conditions
Energy |nutrients |- |answerscarbohydrates, |fats, |proteins
Regulatory |Nutrients |- |answersvitamins, |minerals, |water
Complete |proteins |- |answersanimal |protein |or |plant |protein |+ |small |amount |of |animal |protein |or |two |
complementary |vegetable |proteins.
adipose |tissue |- |answers90% |of |body |energy |stored |here
, BMI |- |answers▸ratio |of |weight |(in |kilograms) |to |height |(in |meters).
▸also |provides |an |estimation |of |relative |risk |for |diseases |such |as |heart |disease, |diabetes, |and |
hypertension.
Underweight |BMI |- |answers<18.5
normal |BMI |- |answers18.5-24.9
Overweight |BMI |- |answers25-29.9
Obesity |BMI |- |answers30-35 |Class1
35-40 |class |2
Extreme |obesity |40+ |Class |3
Excessive |fat |in |abdomen |- |answersis |an |independent |predictor |of |risk |factors |and |mortality
Obesity |prevention |and |treatment |options |- |answers▸Dietary |modifications
▸Exercise
▸Stress |management
▸Bariatric |surgery
Obesity |treatment |at |what |BMI |- |answers25-30
and |30 |plus
▸Roux-en-Y |gastric |bypass |Surgery |- |answersRestrictive |and |Malabsorptive
sleeve |gastrectomy |- |answersRestrictive
Complications |weight |loss |surgery |- |answers▸Hemorrhage
▸DVT/PE
▸Dumping |Syndrome
▸Bile |reflux
Bowel |Obstruction
Cirrhosis |- |answersEnd |stage |chronic |liver |disease. |It |is |a |progressive, |irreversible |disorder, |eventually |
leading |to |liver |failure.
Most |common |types |of |cirrhosis |- |answersAlcoholic |cirrhosisbiliary |cirrhosis, |post |hepatic |cirrhosis
Biliary |Atresia |- |answersFailed |to |open |or |closed
Biliary |atresia |leads |to |- |answersLeads |to |cholestasis, |cirrhosis, |portal |hypertension, |end |stage |liver |
disease, |and |death |by |2 |years |if |not |treated
