TNCC 9TH EDITION EXAM LATEST 2023-2024 COMPLETE 500 QUESTIONS AND CORRECT DETAILED ANSWERS WITH
RATIONALES (VERIFIED ANSWERS) |ALREADY GRADED A+
- ANSPrehospital shock index pg. 85
Injury: shearing or tearing. Diagnosed with MRI.
Widespread microscopic hemorrhage. - ANSDiffuse Axonal Injury (Cannot recover from
this; shearing/tearing portion DOES NOT heal)
Injury: shearing or tearing. Diagnosed with MRI.
Widespread microscopic hemorrhage. - ANSDiffuse Axonal Injury (Cannot recover from
this; shearing/tearing portion DOES NOT heal)
Shock: Spinal cord injury at any level. Transient loss of reflex below
the level of injury. Variable duration. S & S: flaccidity, loss of reflexes, bowel/bladder
dysfunction. - ANSSpinal
Shock: Spinal cord injury at any level. Transient loss of reflex below
the level of injury. Variable duration. S & S: flaccidity, loss of reflexes, bowel/bladder
dysfunction. - ANSSpinal
Shock: Spinal cord injury at T6 or above. Temporary loss of
vasomotor tone and sympathetic innervation. Temporary duration usually <72 hours. S
& S: hypotension, bradycardia, loss of ability to sweat below level of injury. -
ANSNeurogenic
Shock: Spinal cord injury at T6 or above. Temporary loss of
vasomotor tone and sympathetic innervation. Temporary duration usually <72 hours. S
& S: hypotension, bradycardia, loss of ability to sweat below level of injury. -
ANSNeurogenic
: Impairs thrombin production and platelet function
: Impairs thrombin production
: Results in depletion of clotting factors through
hemodilution and the impaired ability to produce clotting factors. - ANSTrauma Triad of
Death
1. Hypothermia
2. Metabolic Acidosis
3. Coagulopathy
: Impairs thrombin production and platelet function
: Impairs thrombin production
: Results in depletion of clotting factors through
hemodilution and the impaired ability to produce clotting factors. - ANSTrauma Triad of
Death
1. Hypothermia
2. Metabolic Acidosis
3. Coagulopathy
,TNCC 9TH EDITION EXAM LATEST 2023-2024 COMPLETE 500 QUESTIONS AND CORRECT DETAILED ANSWERS WITH
RATIONALES (VERIFIED ANSWERS) |ALREADY GRADED A+
???;.][''''''''''''''''''' - ANSthis card was created by a cat. enjoy.
.. activation: .... are found in the carotid sinus and along the aortic arch, are sensitive to
the degree of stretch in the arterial wall. When the receptors sense a decrease in
stretch, they stimulate the sympathetic nervous system to release Epi, norepi, causing
stimulation of cardiac activity and constriction of blood vessels, which causes a rise in
heart rate and diastolic blood pressure - ANSBaroreceptors:
... describes the concept of under treatment of pain. - ANSOligoanalgesia
... describes the concept of under treatment of pain. - ANSOligoanalgesia
... describes the concept of under treatment of pain. - ANSOligoanalgesia
... is a principle that confuses on prevention rather than intervention. - ANSDamage
control resuscitation
... is a principle that confuses on prevention rather than intervention. - ANSDamage
control resuscitation
... is a principle that confuses on prevention rather than intervention. - ANSDamage
control resuscitation
... is a test that requires fluid to be sent to the lab and is considered the gold standard
for identifying CSF - ANSBeta2-Transferrin
... is a test that requires fluid to be sent to the lab and is considered the gold standard
for identifying CSF - ANSBeta2-Transferrin
... is a test that requires fluid to be sent to the lab and is considered the gold standard
for identifying CSF - ANSBeta2-Transferrin
... is a triad of assessment findings; widening pulse pressure, bradycardia, and
diminished respiratory effort - ANSCushing ( it is an attempt to increase MAP against
elevated ICP, ultimately trying to cause a rise in CPP)
... is a triad of assessment findings; widening pulse pressure, bradycardia, and
diminished respiratory effort - ANSCushing ( it is an attempt to increase MAP against
elevated ICP, ultimately trying to cause a rise in CPP)
... is a triad of assessment findings; widening pulse pressure, bradycardia, and
diminished respiratory effort - ANSCushing ( it is an attempt to increase MAP against
elevated ICP, ultimately trying to cause a rise in CPP)
... is damaged brain tissue usually caused by blunt trauma - ANSCerebral contusion
,TNCC 9TH EDITION EXAM LATEST 2023-2024 COMPLETE 500 QUESTIONS AND CORRECT DETAILED ANSWERS WITH
RATIONALES (VERIFIED ANSWERS) |ALREADY GRADED A+
... is damaged brain tissue usually caused by blunt trauma - ANSCerebral contusion
... is damaged brain tissue usually caused by blunt trauma - ANSCerebral contusion
... is defined as the pressure gradient across the brain tissue, or the difference between
the pressures of the cerebral artery and venous vessels. - ANSCPP; CPP = map - icp
... is defined as the pressure gradient across the brain tissue, or the difference between
the pressures of the cerebral artery and venous vessels. - ANSCPP; CPP = map - icp
... is defined as the pressure gradient across the brain tissue, or the difference between
the pressures of the cerebral artery and venous vessels. - ANSCPP; CPP = map - icp
... is the initial post traumatic inflammatory response. ... ... activates this response and
... are sent to the injury sites, activating signaling pathways that mobilize inflammatory
cells. - ANSImmune response; tissue hypoxia;neutrophils
... is the initial post traumatic inflammatory response. ... ... activates this response and
... are sent to the injury sites, activating signaling pathways that mobilize inflammatory
cells. - ANSImmune response; tissue hypoxia;neutrophils
... is the initial post traumatic inflammatory response. ... ... activates this response and
... are sent to the injury sites, activating signaling pathways that mobilize inflammatory
cells. - ANSImmune response; tissue hypoxia;neutrophils
... is the shifting of brain tissue with displacement into another compartment as the
result of bleeding or edema. - ANSHerniation
... is the shifting of brain tissue with displacement into another compartment as the
result of bleeding or edema. - ANSHerniation
... is the shifting of brain tissue with displacement into another compartment as the
result of bleeding or edema. - ANSHerniation
... pain is persistent and usually lasts longer than 3 to 6 months. - ANSChronic
... pain is persistent and usually lasts longer than 3 to 6 months. - ANSChronic
... pain is persistent and usually lasts longer than 3 to 6 months. - ANSChronic
... pain originates from organs and may lead to referred pain. (Trauma) - ANSVisceral
... pain originates from organs and may lead to referred pain. (Trauma) - ANSVisceral
... pain originates from organs and may lead to referred pain. (Trauma) - ANSVisceral
, TNCC 9TH EDITION EXAM LATEST 2023-2024 COMPLETE 500 QUESTIONS AND CORRECT DETAILED ANSWERS WITH
RATIONALES (VERIFIED ANSWERS) |ALREADY GRADED A+
... pain originates from skin and musculoskeletal structures (burns) - ANSSomatic
... pain originates from skin and musculoskeletal structures (burns) - ANSSomatic
... pain originates from skin and musculoskeletal structures (burns) - ANSSomatic
... refers to a condition that occurs when the patient suffers a second milks TBI before
recovery from the first - ANSSecond impact syndrome
... refers to a condition that occurs when the patient suffers a second milks TBI before
recovery from the first - ANSSecond impact syndrome
... refers to a condition that occurs when the patient suffers a second milks TBI before
recovery from the first - ANSSecond impact syndrome
... results from a collection of blood forming between the dura Mater and the skull. This
is frequently associated with fractures of the temporal or parietal skull that lacerated the
.... - ANSEpidural hematoma ; middle meninges artery
... results from a collection of blood forming between the dura Mater and the skull. This
is frequently associated with fractures of the temporal or parietal skull that lacerated the
.... - ANSEpidural hematoma ; middle meninges artery
... results from a collection of blood forming between the dura Mater and the skull. This
is frequently associated with fractures of the temporal or parietal skull that lacerated the
.... - ANSEpidural hematoma ; middle meninges artery
... shock is from hemorrhage and is the leading cause of preventable deaths in trauma
patients. Can also be caused by plasma loss in ... - ANSHypovolemic; burns
... shock is from hemorrhage and is the leading cause of preventable deaths in trauma
patients. Can also be caused by plasma loss in ... - ANSHypovolemic; burns
... shock is from hemorrhage and is the leading cause of preventable deaths in trauma
patients. Can also be caused by plasma loss in ... - ANSHypovolemic; burns
... shock occurs as a result of maldistribution of an adequate circulation blood volume
with the loss of vascular tone or increased permeability. 3 examples. - ANSDistributive;
anaphylactic, septic and neurogenic
... shock occurs as a result of maldistribution of an adequate circulation blood volume
with the loss of vascular tone or increased permeability. 3 examples. - ANSDistributive;
anaphylactic, septic and neurogenic
RATIONALES (VERIFIED ANSWERS) |ALREADY GRADED A+
- ANSPrehospital shock index pg. 85
Injury: shearing or tearing. Diagnosed with MRI.
Widespread microscopic hemorrhage. - ANSDiffuse Axonal Injury (Cannot recover from
this; shearing/tearing portion DOES NOT heal)
Injury: shearing or tearing. Diagnosed with MRI.
Widespread microscopic hemorrhage. - ANSDiffuse Axonal Injury (Cannot recover from
this; shearing/tearing portion DOES NOT heal)
Shock: Spinal cord injury at any level. Transient loss of reflex below
the level of injury. Variable duration. S & S: flaccidity, loss of reflexes, bowel/bladder
dysfunction. - ANSSpinal
Shock: Spinal cord injury at any level. Transient loss of reflex below
the level of injury. Variable duration. S & S: flaccidity, loss of reflexes, bowel/bladder
dysfunction. - ANSSpinal
Shock: Spinal cord injury at T6 or above. Temporary loss of
vasomotor tone and sympathetic innervation. Temporary duration usually <72 hours. S
& S: hypotension, bradycardia, loss of ability to sweat below level of injury. -
ANSNeurogenic
Shock: Spinal cord injury at T6 or above. Temporary loss of
vasomotor tone and sympathetic innervation. Temporary duration usually <72 hours. S
& S: hypotension, bradycardia, loss of ability to sweat below level of injury. -
ANSNeurogenic
: Impairs thrombin production and platelet function
: Impairs thrombin production
: Results in depletion of clotting factors through
hemodilution and the impaired ability to produce clotting factors. - ANSTrauma Triad of
Death
1. Hypothermia
2. Metabolic Acidosis
3. Coagulopathy
: Impairs thrombin production and platelet function
: Impairs thrombin production
: Results in depletion of clotting factors through
hemodilution and the impaired ability to produce clotting factors. - ANSTrauma Triad of
Death
1. Hypothermia
2. Metabolic Acidosis
3. Coagulopathy
,TNCC 9TH EDITION EXAM LATEST 2023-2024 COMPLETE 500 QUESTIONS AND CORRECT DETAILED ANSWERS WITH
RATIONALES (VERIFIED ANSWERS) |ALREADY GRADED A+
???;.][''''''''''''''''''' - ANSthis card was created by a cat. enjoy.
.. activation: .... are found in the carotid sinus and along the aortic arch, are sensitive to
the degree of stretch in the arterial wall. When the receptors sense a decrease in
stretch, they stimulate the sympathetic nervous system to release Epi, norepi, causing
stimulation of cardiac activity and constriction of blood vessels, which causes a rise in
heart rate and diastolic blood pressure - ANSBaroreceptors:
... describes the concept of under treatment of pain. - ANSOligoanalgesia
... describes the concept of under treatment of pain. - ANSOligoanalgesia
... describes the concept of under treatment of pain. - ANSOligoanalgesia
... is a principle that confuses on prevention rather than intervention. - ANSDamage
control resuscitation
... is a principle that confuses on prevention rather than intervention. - ANSDamage
control resuscitation
... is a principle that confuses on prevention rather than intervention. - ANSDamage
control resuscitation
... is a test that requires fluid to be sent to the lab and is considered the gold standard
for identifying CSF - ANSBeta2-Transferrin
... is a test that requires fluid to be sent to the lab and is considered the gold standard
for identifying CSF - ANSBeta2-Transferrin
... is a test that requires fluid to be sent to the lab and is considered the gold standard
for identifying CSF - ANSBeta2-Transferrin
... is a triad of assessment findings; widening pulse pressure, bradycardia, and
diminished respiratory effort - ANSCushing ( it is an attempt to increase MAP against
elevated ICP, ultimately trying to cause a rise in CPP)
... is a triad of assessment findings; widening pulse pressure, bradycardia, and
diminished respiratory effort - ANSCushing ( it is an attempt to increase MAP against
elevated ICP, ultimately trying to cause a rise in CPP)
... is a triad of assessment findings; widening pulse pressure, bradycardia, and
diminished respiratory effort - ANSCushing ( it is an attempt to increase MAP against
elevated ICP, ultimately trying to cause a rise in CPP)
... is damaged brain tissue usually caused by blunt trauma - ANSCerebral contusion
,TNCC 9TH EDITION EXAM LATEST 2023-2024 COMPLETE 500 QUESTIONS AND CORRECT DETAILED ANSWERS WITH
RATIONALES (VERIFIED ANSWERS) |ALREADY GRADED A+
... is damaged brain tissue usually caused by blunt trauma - ANSCerebral contusion
... is damaged brain tissue usually caused by blunt trauma - ANSCerebral contusion
... is defined as the pressure gradient across the brain tissue, or the difference between
the pressures of the cerebral artery and venous vessels. - ANSCPP; CPP = map - icp
... is defined as the pressure gradient across the brain tissue, or the difference between
the pressures of the cerebral artery and venous vessels. - ANSCPP; CPP = map - icp
... is defined as the pressure gradient across the brain tissue, or the difference between
the pressures of the cerebral artery and venous vessels. - ANSCPP; CPP = map - icp
... is the initial post traumatic inflammatory response. ... ... activates this response and
... are sent to the injury sites, activating signaling pathways that mobilize inflammatory
cells. - ANSImmune response; tissue hypoxia;neutrophils
... is the initial post traumatic inflammatory response. ... ... activates this response and
... are sent to the injury sites, activating signaling pathways that mobilize inflammatory
cells. - ANSImmune response; tissue hypoxia;neutrophils
... is the initial post traumatic inflammatory response. ... ... activates this response and
... are sent to the injury sites, activating signaling pathways that mobilize inflammatory
cells. - ANSImmune response; tissue hypoxia;neutrophils
... is the shifting of brain tissue with displacement into another compartment as the
result of bleeding or edema. - ANSHerniation
... is the shifting of brain tissue with displacement into another compartment as the
result of bleeding or edema. - ANSHerniation
... is the shifting of brain tissue with displacement into another compartment as the
result of bleeding or edema. - ANSHerniation
... pain is persistent and usually lasts longer than 3 to 6 months. - ANSChronic
... pain is persistent and usually lasts longer than 3 to 6 months. - ANSChronic
... pain is persistent and usually lasts longer than 3 to 6 months. - ANSChronic
... pain originates from organs and may lead to referred pain. (Trauma) - ANSVisceral
... pain originates from organs and may lead to referred pain. (Trauma) - ANSVisceral
... pain originates from organs and may lead to referred pain. (Trauma) - ANSVisceral
, TNCC 9TH EDITION EXAM LATEST 2023-2024 COMPLETE 500 QUESTIONS AND CORRECT DETAILED ANSWERS WITH
RATIONALES (VERIFIED ANSWERS) |ALREADY GRADED A+
... pain originates from skin and musculoskeletal structures (burns) - ANSSomatic
... pain originates from skin and musculoskeletal structures (burns) - ANSSomatic
... pain originates from skin and musculoskeletal structures (burns) - ANSSomatic
... refers to a condition that occurs when the patient suffers a second milks TBI before
recovery from the first - ANSSecond impact syndrome
... refers to a condition that occurs when the patient suffers a second milks TBI before
recovery from the first - ANSSecond impact syndrome
... refers to a condition that occurs when the patient suffers a second milks TBI before
recovery from the first - ANSSecond impact syndrome
... results from a collection of blood forming between the dura Mater and the skull. This
is frequently associated with fractures of the temporal or parietal skull that lacerated the
.... - ANSEpidural hematoma ; middle meninges artery
... results from a collection of blood forming between the dura Mater and the skull. This
is frequently associated with fractures of the temporal or parietal skull that lacerated the
.... - ANSEpidural hematoma ; middle meninges artery
... results from a collection of blood forming between the dura Mater and the skull. This
is frequently associated with fractures of the temporal or parietal skull that lacerated the
.... - ANSEpidural hematoma ; middle meninges artery
... shock is from hemorrhage and is the leading cause of preventable deaths in trauma
patients. Can also be caused by plasma loss in ... - ANSHypovolemic; burns
... shock is from hemorrhage and is the leading cause of preventable deaths in trauma
patients. Can also be caused by plasma loss in ... - ANSHypovolemic; burns
... shock is from hemorrhage and is the leading cause of preventable deaths in trauma
patients. Can also be caused by plasma loss in ... - ANSHypovolemic; burns
... shock occurs as a result of maldistribution of an adequate circulation blood volume
with the loss of vascular tone or increased permeability. 3 examples. - ANSDistributive;
anaphylactic, septic and neurogenic
... shock occurs as a result of maldistribution of an adequate circulation blood volume
with the loss of vascular tone or increased permeability. 3 examples. - ANSDistributive;
anaphylactic, septic and neurogenic
