NUR 2030 EXAM 4 QUESTIONS & ANSWERS(RATED A+)

NUR 2030 EXAM 4 QUESTIONS &
ANSWERS(RATED A+)
Side effect of another disorder - ANSWER- hormonal disorders:
hyperparathyroidism, exogenous glucocorticoids

Upper GI tract - ANSWEResophagus, stomach and small intestine
- digestion of food and fluid
- absorption of essential nutrients, vitamins and minerals
- alterations in upper GI function can cause indigestion, malabsorption, malnutrition,
or dehydration

Lower GI tract - ANSWERlarge intestine
- absorption of fluid and electrolytes
- alterations in lower GI function can cause dehydration, diarrhea or constipation

Stomach - ANSWER- primary function is to break down & digest food
- sphincters

Upper esophageal sphincter - ANSWERprevents aspiration of contents

Lower esophageal sphincter (LES) - ANSWERseparates gastric contents from
esophagus, gastric contents are acidic and can harm esophageal epithelium

Pyloric sphincter - ANSWERregulates movement of chyme out of stomach to
duodenum

Sphincter of Oddi - ANSWERregulates the movement of bile and pancreatic juice
from accessory organs

What are the 4 layers of the GI tract? - ANSWER1. mucosa
2. submucosa
3. muscularis
4. serosa

Small intestine - ANSWERmotility- peristalsis & segmentation
- villi and microvilli= increase surface area increasing absorptive cavity

Villi - ANSWERthousands of tiny fingerlike projections that contain cells, whose
functions are to release digestive enzymes, secrete mucus and absorb nutrients

Microvilli - ANSWERare located on the epithelial cells of the vili. This double set of
villi is known as the brush border

Villi and Microvilli - ANSWER- double the surface area significantly increasing the
absorptive capacity of the small intestine

, Large intestine - ANSWER- motility peristalsis & haustration
1. absorption of water and electrolytes (Na, CL, K)
2. Bacterial breakdown of proteins that were not digested or absorbed in the small
intestine. These resulting amino acids are broken down by bacteria, which leaves
ammonia. Ammonia is carried to the liver and converted to urea
3. bacteria metabolize bile salts and facilitate absorptions of bile
4. store feces until defecation

gastroesophageal reflux disease (GERD) - ANSWER- decreased closure of the
lower esophageal sphincter (LES) which allows acidic gastric contents to reflux up
into the esophagus and creates inflammation reaction
- decrease pressure at LES and increase pressure in stomach

Gastroesophageal Reflux Disease (GERD) causes - ANSWER- weak diaphragm or
circular muscle
- herniation
- delayed gastric emptying
- alcohol, smoking
- increased PNS

Gastroesophageal Reflux Disease (GERD) - ANSWER- acid of the stomach
damages the esophageal epithelium and imitates inflammation
- high acid contents causes erosion
- high acid environment changes squamous epithelium of esophagus to columnar
epithelium
- Barrett's esophagus
- can lead to esophageal cancer
- columnar epithelium is more resistant to acid

GI bleeding - ANSWER- hematemesis
- melena
- hematochezia
- occult blood

Hematemesis - ANSWER- vomiting blood
- red: current bleeding mostly from esophagus, medical emergency
- coffee grounds: blood from stomach that has mixed with stomach acid, then
vomited

Melena - ANSWERblack, tarry stool
- typically slow, chronic upper GI bleeding
- small intestine (PUD)

Hematochezia - ANSWER- bright, fresh blood from anus with stool
- lower GI bleeding from hemorrhoids, diverticulosis, colon cancer

Occult blood - ANSWER- small amount of blood, hidden in stool
- bleeding in any GI tract
- cannot see blood in stool but can test for it