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NURS 6620 FINAL EXAM 2025 COMPLETE QUESTIONS AND VERIFIED ANSWERS | ACTUAL EXAM| ACCURATE SOLUTIONS WITH RATIONALES/ALREADY GRADED A+

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NURS 6620 FINAL EXAM 2025 COMPLETE QUESTIONS AND VERIFIED ANSWERS | ACTUAL EXAM| ACCURATE SOLUTIONS WITH RATIONALES/ALREADY GRADED A+

Institution
NURS 6620
Course
NURS 6620

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NURS 6620 FINAL EXAM 2025
COMPLETE QUESTIONS AND
VERIFIED ANSWERS | ACTUAL
EXAM| ACCURATE SOLUTIONS
WITH RATIONALES/ALREADY
GRADED A+

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Terms in this set (43)

, b.Sodium chloride enters the cell.
In hypoxic injury, movement of fluid
and ions into the cell is associated
with acute failure of metabolism and
a loss of ATP production. Normally,
the pump that transports sodium
ions out of the cell is maintained by
the presence of ATP and ATPase, the
During cell injury active transport enzyme. In
caused by metabolic failure caused by
hypoxia, why hypoxia, reduced ATP and ATPase
does an increase levels permit sodium to accumulate
in the osmotic in the cell, whereas potassium
pressure within diffuses outward. The increase of
the cell occur? intracellular sodium increases
osmotic pressure, which draws more
water into the cell. (Transport
mechanisms are described in
Chapter 1.) The remaining options do
not accurately describe the cell
injury that results in increased
osmotic pressure caused by
hypoxia.

, c.Renal failure
Hyperkalemia should be
investigated when a history of renal
disease, massive trauma, insulin
A healthcare deficiency, Addison disease, use of
professional is potassium salt substitutes, or
caring for four metabolic acidosis exists.
patients. Which Hyperparathyroidism might lead to
patient should hyperphosphatemia. Vomiting is
the professional frequently associated with
assess for potassium depletion.
hyperkalemia or Hyperaldosteronism also can lead
hypermagnesmi to potassium wasting.
a? Renal failure usually causes
hypermagnesemia, in which
magnesium concentration is greater
than 2.5 mEq/L. Hypermagnesemia
is not a result of the other options.

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Institution
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Course
NURS 6620

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