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TNCC TEST 2025 ACTUAL FINAL EXAM TEST BANK 300 QUESTIONS AND CORRET DETAILED ANSWER

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TNCC TEST 2025 ACTUAL FINAL EXAM TEST BANK 300 QUESTIONS AND CORRET DETAILED ANSWER

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Nursing Pediatrics
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Nursing Pediatrics











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Institution
Nursing Pediatrics
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Nursing Pediatrics

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TNCC TEST 2025 ACTUAL FINAL EXAM TEST BANK 300
QUESTIONS AND CORRET DETAILED ANSWERS WITH
RATIONALES (VERIFIED ANSWERS) |ALREADY GRADED
A+|100% COMPLETE VERIFIED PASS
A adult patient with a knife injury to the neck has an intact airway and is hemodynamically stable. They complain of difficulty swallowing and speaking. In the
primary survey, further assessment is indicated next for which of the following conditions?
a. Damage to the cervical spine
b. An expanding pneumothorax
c. Laceration of the carotid artery
d. Injury to the thyroid gland - ansa. Damage to the cervical spine

Breathing and Ventilation Assessment - ansInspect: spontaneous breathing, symmetrical rise and fall, depth/pattern/rate of respirations, accessory muscle use,
diaphragmatic breathing, skin color (normal, pale, flushed, cyanotic), contusions/abrasions/deformities (signs of underlying injury), open pneumothoraces
(sucking chest wound), JVD, tracheal position, signs of inhalation injury

Auscultate: presence, absence and equality of breath sounds at 2nd intercostal space midclavicular line and bases at the fifth intercostal space anterior axillary
line

Palpate: bony structures, possible rib fractures, SQ emphysema, soft tissue injury, JV pulsations at suprasternal notch or supraclavicular area

Life-threatening pulmonary injuries requiring immediate intervention: open pneumothorax, tension pneumothorax, flail chest, hemothorax.

Breathing and Ventilation Intervention - ansBreathing absent: jaw-thrust maneuver, oral airway adjunct, assist ventilation with bag-mask device, prepare for
definitive airway

Breathing present: NRB. Determine if ventilation effective: etCO2 35-45, SpO2 94% or higher. If ineffective: assist with bag-mask and determine need for
definitive airway

C (Primary Survey) - ansCirculation and Control of Hemorrhage

Cardiogenic Shock - ansResults from pump failure in the presence of adequate intravascular volume. There is a lack of cardiac output and end-organ perfusion
secondary to a decrease in myocardial contractility and/or valvular insufficiency.

Acute causes - myocardial infarction, dysrhythmias or toxicologic pathologies. Heart failure is a chronic cause.

Blunt cardiac injury may present similar to MI.

Excess of volume administration or increased after load can result in pulmonary edema and increased myocardial ischemia.

Inotropic support to improve contractility.

Circulation and Control of Hemorrhage Assessment - ansInspect: Uncontrolled external bleeding, skin color

Auscultate: Muffled heart sounds - may indicate pericardial tamponade

Palpate: carotid and/or femoral pulses for rate, rhythm, strength

Circulation and Control of Hemorrhage Interventions - ansControl and treat external bleeding: apply direct pressure, elevate bleeding extremity, apply pressure
over arterial sites, consider use of a tourniquet.

2 large bore IVs, if unable consider IO, obtain labs and crossmatch.

Initiate IVF of warmed isotonic crystalloid solution. Consider blood products after 2L.

**Large volumes of fluid lead to dilution coagulopathy which worsens metabolic acidosis and may cause hypothermia. Component therapy, including
administering RBC, plasma and platelets is a balanced approach so that O2 delivery is optimized, acidosis corrected and coagulopathy prevented.

Classifications of Shock - ansHypovolemic - decrease in the amount of circulating blood volume

Obstructive - obstruction in either the vasculature or heart

Cardiogenic - pump failure in the presence of adequate intravascular volume

Distributive - maldistribution of an adequate circulating blood volume (septic, anaphylactic, neurogenic)

,TNCC TEST 2025 ACTUAL FINAL EXAM TEST BANK 300
QUESTIONS AND CORRET DETAILED ANSWERS WITH
RATIONALES (VERIFIED ANSWERS) |ALREADY GRADED
A+|100% COMPLETE VERIFIED PASS

Corneal Abrasion - ansDamage to the corneal epithelium. Easy to evaluate with fluorescein.

Findings: photophobia, tearing, pain, injected conjunctiva (redness), lid swelling, irritation

Treatment: Ophthalmic ABX, Cycloplegic agent to decrease spasms and pain, ophthalmic NSAIDS to decrease swelling, oral analgesics, Ophthalmic f/u in 24
hours.
(Do NOT patch - increases infection)

Corneal Foreign Body - ansRoutinely metal, plastic or wood.

Findings: photophobia, pain, injected conjunctiva (redness), lid swelling

Treatment: topical anesthetic, removal of foreign body, ophthalmic ABX, cycloplegics, oral analgesia

Corneal Laceration - ansInvolves one or more layers of the cornea. Visualized with a slit lamp.

Findings: similar to abrasion, pain out of proportion to findings, decreased vision

Treatment: treat small lacerations similar to an abrasion, larger lacerations need ophthalmology referral and possible surgery

Cycloplegic agent - ansCycloplegia is paralysis of the ciliary muscle of the eye, resulting in a loss of accommodation. Because of the paralysis of the ciliary
muscle, the curvature of the lens can no longer be adjusted to focus on nearby objects.

D (Primary Survey) - ansDisability (Neurologic Status)

Disability Assessment - ansAssess GCS on arrival and repeat per policy.

Assess pupils for equality, shape and reactivity (PERRL)

Disability interventions - ansEvaluate for need for CT. Assume AMS to be the result of CNS injury until proven otherwise.

Consider ABGs - AMS may be indicator of decreased cerebral perfusion, hypoventilation or acid-base imbalance.

Consider bedside glucose.

Distributive Shock - ansOccurs as a result of maldistribution of an adequate circulating volume with a loss of vascular tone or increased permeability.

Diffuse vasodilation lowers the systemic pressure, creating a relative hypovolemia or reduction of the mean systemic volume and venous return to the heart or
drop in preload, resulting in distributive shock.

Anaphylactic: release of inflammatory mediators, such as histamine, which contracts bronchial smooth muscle and increases vascular permeability and
vasodilation.

Septic Shock: systemic release of bacterial endotoxins, resulting in an increased vascular permeability and vasodilation

Neurogenic shock: loss of sympathetic nervous system control of vascular tone, which produces venous and arterial vasodilation. With the loss of sympathetic
nervous system input in spinal cord injury, unopposed vagal activity may result in decreased cardiac output through bradycardia.

TREATMENT: increase systemic resistance, controlled volume replacement. Vasoconstriction and in some cases (neurogenic) Atropine to counteract
bradycardia.

E (Primary Survey) - ansExposure and Environmental Control

Exposure and Environmental Control - ansCarefully and completely undress the patient. Inspect for uncontrolled bleeding and note any obvious injuries.

Prevent heat loss. Hypothermia combined with hypotension and acidosis is a potentially lethal combination in the injured patient. Consider: warm blankets, keep
ambient temperature warm, warm IVF, forced air warmers, radiant warming lights.

F (Primary Survey) - ansFull Set of VS & Family Presence

,TNCC TEST 2025 ACTUAL FINAL EXAM TEST BANK 300
QUESTIONS AND CORRET DETAILED ANSWERS WITH
RATIONALES (VERIFIED ANSWERS) |ALREADY GRADED
A+|100% COMPLETE VERIFIED PASS

G (Primary Survey) - ansGet Resuscitation Adjuncts:
(LMNOP)
L: Labs
M: Monitor cardiac rate and rhythm
N: Naso or orogastric tube consideration
O: Oxygenation - SpO2 and/or etCO2 monitor
P: Pain assessment and management

GCS - ansGCS

EYES
1: Does not open eyes
2: Opens eyes in response to pain
3: Opens eyes in response to voice
4: Opens eyes spontaneously

VERBAL
1. Makes no sounds
2. Makes sounds
3. Words
4. Confused, disoriented
5. Oriented, converses normally

MOTOR
1. Makes no movements
2. Extension to painful stimuli (decerebrate)
3. Abnormal flexion to painful stimuli (decorticate)
4. Withdrawal to painful stimuli
5. Localizes painful stimuli
6. Obeys commands

H (Secondary Survey) - ansHistory
Prehospital Report (MIST)
M: MOI
I: Injuries sustained
S: Signs and symptoms in the field
T: Treatment in field

Patient History (SAMPLE):
S: Symptoms
A: Allergies and tetanus status
M: Medications
P: Past medical history
L: Last oral intake
E: Events and Environmental factors related to injury.

H: Head and Face
Head to Toe Assessment (secondary survey) - ansSOFT TISSUE:

Inspect: lacerations, puncture wounds, abrasions, contusions, edema, ecchymosis, impaled objects.

Palpate: areas of tenderness, step-offs, crepitus

BONY DEFORMITIES:

Inspect: asymmetry of facial expressions, exposed tissue or bone for brain matter

Palpate: depressions, angulations, tenderness

, TNCC TEST 2025 ACTUAL FINAL EXAM TEST BANK 300
QUESTIONS AND CORRET DETAILED ANSWERS WITH
RATIONALES (VERIFIED ANSWERS) |ALREADY GRADED
A+|100% COMPLETE VERIFIED PASS
Hepatic Injuries - ansIn blunt trauma the liver may lacerate from increased abdominal pressure.

Hematoma - bleeding contained within the capsule

Laceration - the capsule is disrupted

Findings: Cullen sign (ecchymosis around the umbilicus or RUQ), tenderness, guarding or rigidity RUQ, 9-12 rib FXs, elevated LFT

Graded I-VI, I = minor trauma

Nonoperative management is standard of care in hemodynamically stable patient. Observed with serial abdominal exams.

Findings of contrast extravasation may be embolized by IR.

For surgical patients - fluid resuscitation is essential. Risks of surgery include disruption of the natural tamponade process due to the evacuation of large
amounts of blood resulting in hypovolemia.

Hypovolemic Shock - ansCaused by a decrease in the amount of circulating blood volume.

In trauma typically results from hemorrhage, but can result in a precipitous loss of volume, ie vomiting or diarrhea.

Burn trauma can result in hypovolemic shock from damage to the cell membranes leading to plasma and protein leakage. of body water, results in inadequate
perfusion.

Hyperventilation can cause increased intrathoracic pressure resulting in compression of the heart and decreased cardiac output.

Initial Assessment - ans1. Preparation and Triage
2. Primary Survey
3. Reevaluation
4. Secondary Survey
5. Reevaluation Adjuncts
6. Reevaluation and Post Resuscitation Care
7. Definitive Care or Transport

Intraocular Foreign Body - ans*TRUE EMERGENCY AND EARLY INTERVENTION IS ESSENTIAL.

Findings: compromised visual acuity, misshapen pupils, pain

Treatment: elevate HOB, ophthalmology, immobilize foreign body, patch UNAFFECTED eye to limit concomitant eye movement, globe closure ASAP, systemic
and ophthalmic ABX, analgesics.

Postop infection, retinal detachment and vision loss are common complications.

lid injury - ans

Liver - ansLargest solid organ of the body. RUQ, 6th to 10th ribs. Encased by Glisson capsule with blood vessels, lymphatics and nerves. Filters 1.7L of blood
per minute.

The liver filters out toxins, takes the nutrients and returns the blood to the heart via the hepatic veins.

Hepatocyte cells are capable of regeneration allowing the liver to repair its own tissue.

Functions: Store and metabolize lipids, transport nutrients, produce glucose and bilirubin, convert ammonia to urea, secrete electrolytes, lipids, lecithin,
cholesterol and bile. Metabolizes vitamin K and produces thrombin and fibrinogen (all necessary for clotting).

Obstructive Shock - ansResults from hypo perfusion of the tissue due to an obstruction in either the vasculature or heart.

Tension pneumothorax - increased thoracic pressure leads to displacement of the vena cava, obstruction to atrial filling, decreased preload and decreased
cardiac output.

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