ANCC PMHNP Cert Exam Review Questions And Answers Verifed 100% Correct

ANCC PMHNP Cert Exam Review Questions And Answers Verifed 100% Correct What is the adverse effect of Depakote on the liver? - ANSWER can cause liver damage, and the risk is more likely to occur during the first 6 months of taking the medication. Signs might include nausea or vomiting, loss of appetite, abdominal pain, dark-colored urine, facial swelling, and yellowing of the skin or white of eyes What would you do for a patient on Depakote with +4 protein in the urine? - ANSWER Rule out UTI, no adjustment for medication is needed What happens when you take carbamazepine (tegertal) and erythromycin together? - ANSWER It will decrease the effect of the antibiotic by inhibition What happens when you take zyprexa and smoke? - ANSWER nicotine will increase the excretion of the drug thereby inhibiting it's effect ICP in Infants S/S - ANSWER Bulging fontanels, high-pitch cry, irritability, restlessness *ICP* *CHILDREN* signs and symptoms - ANSWER • Headache • Vomiting (usually projectile) • Seizures • Diplopia (Dbl vision), blurred vision Neuro cry - ANSWER high-pitched and very grating on the ears due to their neurological sxs being overwhelmed Neuroleptic Malignant Syndrome - ANSWER Adverse reaction to antipsychotics with severe "lead pipe" rigidty, FEVER, and mental status changes. FEVER: Fever Encephalopathy Vitals unstable Elevated enzymes Rigidity of muscles NMS treatment - ANSWER Dantrolene D2 agonists (e.g., bromocriptine). lead poisoning - ANSWER A medical condition caused by toxic levels of the metal lead in the blood Lead poisoning treatment - ANSWER chelation therapy Amygdala's role in emotion - ANSWER - Aggression center - Role in storing implicit emotional memories - Stimulation (+) anger/violence or fear/anxiety - Damage (-) mellow; hyperorality; hypersexuality; dis-inhibited behavior Amygdala role in aggression - ANSWER Small volume increase aggression. Frontal Lobe Atrophy - ANSWER As neurons die in the frontal and temporal regions, these lobes atrophy, or shrink. Gradually, this damage causes difficulties in thinking and behaviors normally controlled by these parts of the brain. ... Scientists think that FTLD (frontal temporal lobe dementia) is the most common cause of dementia in people younger than age 60 Four dopaminergic pathways - ANSWER mesocortical mesolimbic nigrostriatal tuberoinfundibular mesocortical pathway - ANSWER a path through which dopaminergic projections travel to reach the neocortex mescolimbic pathway - ANSWER The mesolimbic pathway, sometimes referred to as the reward pathway, is a dopaminergic pathway in the brain. The pathway connects the ventral tegmental area in the midbrain, to the ventral striatum of the basal ganglia in the forebrain. The ventral striatum includes the nucleus accumbens and the olfactory tubercle. Nigrostriatal pathway - ANSWER the dopaminergic tract from the substantia nigra to the striatum Tuberoinfundibular pathway - ANSWER from hypothalamus to anterior pituitary -> DA released from these neurons inhibit prolactin secretion -> when DA receptors here are blocked prolactin levels rise cause galactorrhea) primary prevention - ANSWER Efforts to prevent an injury or illness from ever occurring. secondary prevention - ANSWER Efforts to limit the effects of an injury or illness that you cannot completely prevent, screening exams. tertiary prevention - ANSWER actions taken to contain damage once a disease or disability has progressed beyond its early stages menopause hormone therapy - ANSWER most effective therapy for moderate -severe vasomotor symptoms - use quality of life scales to identify degree of vasomotor symptom severity *should not be used for protection against CVD or dementia congestive heart failure and depression - ANSWER MDD is highly prevalent in these patients Thyroid levels while on lithium - ANSWER Because of the high incidence of thyroid dysfunction that occurs during lithium treatment, patients should have a careful thyroid physical examination and determination of serum thyroid-stimulating hormone (TSH) and antithyroid peroxidase antibody titers before lithium treatment is begun. Patients with normal thyroid function initially should be reevaluated every 6 to 12 months for several years, and thyroid dysfunction should be treated if diagnosed. The development of thyroid dysfunction does not typically require discontinuation of lithium. If thyroid function is abnormal at the initial evaluation, lithium can still be given if necessary, but the thyroid dysfunction should be treated. SSRIs MOA - ANSWER block reuptake of serotonin by inhibiting the presynaptic reuptake SNRIs mechanism - ANSWER Inhibit 5-HT and norepinephrine reuptake. Lithium mechanism of action - ANSWER Not established, possibly related to inhibition of phosphoinosital cascade It inhibits excitatory neurotransmitters such as dopamine and glutamate, and promotes GABA-mediated neurotransmission. Carbamazepine mechanism - ANSWER Block voltage-dependent sodium channels Structural Therapy - ANSWER A therapeutic approach directed at changing or realigning the organization of a family to modify dysfunctional patterns and clarify boundaries. Developed by Salvador Minuchin.