NU 578 Unit 3 Questions and Correct Detailed
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Quiz: Treatment of CHF
Ans: Digoxin (increases cardiac contractility), diuretics (increases NA and H2O excretion),
ACEI (decreases BP and BV), Vasodilators (decreases BP), dobutamine and dopamine and
PDE inhibitors (increase ventricular contractility)
Quiz: Digoxin
Ans: class: cardiac glycoside
increases myocardial contractile force and increases cardiac output
has a positive inotropic effect by inhibiting sodium potassium tump (this increases Calcium
into cell).
decreases HR
decreases AV nodal conduction
* USED FOR HF AND DYSRHYTHMIAS*
Quiz: Digoxin toxicity
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, Ans: warn patients about dig induced dysrhythmias and instruct to take meds exactly as
prescribed. toxicity symptoms are altered hr or rhythm, visual or gi disturbances (nausea,
anorexia, vomitting, fatigue, blurred vision, yellow tinge to vision). toxicity is made much
worse by hypokalemia or anything that decreases digoxin clearance.
Treatment: d/c digoxin, correct potassium, administer digibind
Quiz: Digoxin pharmacokinetics
Ans: good PO absorption but capsules are better than tablets. don't switch forms once
prescribed. dosage is based on lean body weight and is really excreted. therapeutic levels
are 0.5-0.8ng/ml.
half life is 1.5 days. 6 days required to reach plateau and 6 days to eliminate.
Quiz: Digoxin drug interactions
Ans: Antacids: decease absorption
diuretics: cause hypokalemia and increase toxicity
quinidine: displaces dig from tissues and decreases excretion
amiodarone: increases concentration of dig
verapamil: increase plasma levels of dig
sympathomimetics: increases chance of arrthymias
Quiz: How do ACEI work in CHF?
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, Ans: inhibitor of ACE decreases angiotensin II, decreases total peripheral resistance, and
decreases blood volume. decreased aldosterone decreases TPR and BV.
Block the production of angiotensin II, decrease the release of aldosterone, and suppress
degradation of kinins. arteriolar dilation improves blood flow in kidneys, venous dilation
reduces venous pressure, edema, preload, and suppression of aldosterone release
enhances excretion of sodium and water . contribute to cardiac remodeling!!! prolong life.
not in acute decompensated HF. excreted by kidneys
FIRST LINE TREATMENT IN CHF!!!!
Quiz: ACEI side effects
Ans: cough, increased potassium, diiness, angioedema, hypotension. can cause renal
failure in patients with bilateral renal artery stenosis. use with caution in patients taking
potassium supplements or k sparing diuretics, neutropenia
DDI: diuretics can intensify hypotension, antihypertensive agents can increase powered bp,
risk of hyperkalemia with k sparing diuretics and k supplements, NSaids and Lithium
decrease effects of ACEI
BBW: can cause fetal injury in pregnancy.zz
Quiz: Lisinopril
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Answers (Verified Answers) ||Already Graded
A+||Newest Version
Quiz: Treatment of CHF
Ans: Digoxin (increases cardiac contractility), diuretics (increases NA and H2O excretion),
ACEI (decreases BP and BV), Vasodilators (decreases BP), dobutamine and dopamine and
PDE inhibitors (increase ventricular contractility)
Quiz: Digoxin
Ans: class: cardiac glycoside
increases myocardial contractile force and increases cardiac output
has a positive inotropic effect by inhibiting sodium potassium tump (this increases Calcium
into cell).
decreases HR
decreases AV nodal conduction
* USED FOR HF AND DYSRHYTHMIAS*
Quiz: Digoxin toxicity
o
o
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in the number given.
, Ans: warn patients about dig induced dysrhythmias and instruct to take meds exactly as
prescribed. toxicity symptoms are altered hr or rhythm, visual or gi disturbances (nausea,
anorexia, vomitting, fatigue, blurred vision, yellow tinge to vision). toxicity is made much
worse by hypokalemia or anything that decreases digoxin clearance.
Treatment: d/c digoxin, correct potassium, administer digibind
Quiz: Digoxin pharmacokinetics
Ans: good PO absorption but capsules are better than tablets. don't switch forms once
prescribed. dosage is based on lean body weight and is really excreted. therapeutic levels
are 0.5-0.8ng/ml.
half life is 1.5 days. 6 days required to reach plateau and 6 days to eliminate.
Quiz: Digoxin drug interactions
Ans: Antacids: decease absorption
diuretics: cause hypokalemia and increase toxicity
quinidine: displaces dig from tissues and decreases excretion
amiodarone: increases concentration of dig
verapamil: increase plasma levels of dig
sympathomimetics: increases chance of arrthymias
Quiz: How do ACEI work in CHF?
o
o
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in the number given.
, Ans: inhibitor of ACE decreases angiotensin II, decreases total peripheral resistance, and
decreases blood volume. decreased aldosterone decreases TPR and BV.
Block the production of angiotensin II, decrease the release of aldosterone, and suppress
degradation of kinins. arteriolar dilation improves blood flow in kidneys, venous dilation
reduces venous pressure, edema, preload, and suppression of aldosterone release
enhances excretion of sodium and water . contribute to cardiac remodeling!!! prolong life.
not in acute decompensated HF. excreted by kidneys
FIRST LINE TREATMENT IN CHF!!!!
Quiz: ACEI side effects
Ans: cough, increased potassium, diiness, angioedema, hypotension. can cause renal
failure in patients with bilateral renal artery stenosis. use with caution in patients taking
potassium supplements or k sparing diuretics, neutropenia
DDI: diuretics can intensify hypotension, antihypertensive agents can increase powered bp,
risk of hyperkalemia with k sparing diuretics and k supplements, NSaids and Lithium
decrease effects of ACEI
BBW: can cause fetal injury in pregnancy.zz
Quiz: Lisinopril
o
o
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