mental health Rasmussen final questions with verified
answers
0.4-1 Ans✓✓✓ therapeutic level of lithium
signs
slight hand tremor
mild thirst
weight gain
nausea
advanced Ans✓✓✓ early, advanced or severe lithium toxicity?
1.5-2 mEq/L
course hand tremor
persistant GI upset
mental confusion
muscle hyper irritability
EEG changes
incoordination
hold meds, Blood level drawn, adjust dose, administer emetic ,gastric lavage,
mannitol, aminophylline hasten lithium excretion
alcohol Ans✓✓✓ early signs of withdrawal develop within a few hours after
cessation they peak after 24 to 48 hours and then rapidly and dramatically
disappear
,the person may appear hyperalert, manifest jerky movements and irritability,
startle easily, and experience subjective distress often described as "shaking
inside." Grand mal seizures may appear 7 to 48 hours after cessation
•Autonomic hyperactivity (e.g., tachycardia, diaphoresis, elevated blood pressure)
•Severe disturbance in sensorium (e.g., disorientation, clouding of consciousness)
•Perceptual disturbances (e.g., visual or tactile hallucinations)
•Fluctuating levels of consciousness (e.g., ranging from hyperexcitability to
lethargy)
•Delusions (paranoid), agitated behaviors, and fever (temperatures of 100° to
103° F)
alzheimers Ans✓✓✓ a complex disease that begins to damage the brain long
before the symptoms appear. AD affects processes that keep the neurons healthy,
such as (1) communication, (2) metabolism, and (3) repair. In a healthy brain
neurons are supported by microtubules, which guide nutrients and molecules
between the cell body and the axon terminals. A special protein called tau protein
is responsible for the stability of the microtubules. In AD tau protein is subjected
to chemical changes, which result in neurofibrillary tangles and cause
disintegration of the microtubules, thus collapsing the neuron's transport system.
This disintegration of the neuron transport system results in malfunction of
communication between neurons, and eventually leads to neural cell death. It is
the destruction and death of the cells that causes memory failure, personality
changes, problems in carrying out daily activities, and other features of the
disease
alzheimers meds Ans✓✓✓ galantamine hydrobromide (Razadyne), rivastigmine
tartrate (Exelon), and donepezil hydrochloride (Aricept)
, These are called cholinesterase inhibitors that help dely the progression of the
disease in the mild to moderate stage
alzheimers meds Ans✓✓✓ Memantine hydrochloride (Namenda), an N-methyl-d-
aspartate (NMDA), is an antagonist at the NMDA-glutamatergic ion channels. This
drug works by blocking the toxic effects associated with excess glutamate and
regulates glutamate activation. It is the first drug to target symptoms of AD during
the moderate to severe stages of the disorder
anorexia Ans✓✓✓ engage in self-starvation, express intense fear of gaining
weight, and have a disturbance in self-evaluation of weight and its importance.
often experience amenorrhea
• Weight loss more than 30% over 6 months • Rapid decline in weight • Inability
to gain weight with outpatient treatment • Severe hypothermia caused by loss of
subcutaneous tissue or dehydration (body temperature lower than 36° C or 96.8°
F) • Heart rate less than 40 beats per minute • Systolic blood pressure less than
70 mm Hg • Hypokalemia (less than 3 mEq/L) or other electrolyte disturbances
not corrected by oral supplementation • Electrocardiographic changes (especially
dysrhythmias)
Milelu therapy:
These modalities are designed to normalize eating patterns and to begin to
address the issues raised by the illness. The milieu of an eating disorder unit is
purposefully organized to assist the patient in establishing more adaptive
behavioral patterns, including normalization of eating.
answers
0.4-1 Ans✓✓✓ therapeutic level of lithium
signs
slight hand tremor
mild thirst
weight gain
nausea
advanced Ans✓✓✓ early, advanced or severe lithium toxicity?
1.5-2 mEq/L
course hand tremor
persistant GI upset
mental confusion
muscle hyper irritability
EEG changes
incoordination
hold meds, Blood level drawn, adjust dose, administer emetic ,gastric lavage,
mannitol, aminophylline hasten lithium excretion
alcohol Ans✓✓✓ early signs of withdrawal develop within a few hours after
cessation they peak after 24 to 48 hours and then rapidly and dramatically
disappear
,the person may appear hyperalert, manifest jerky movements and irritability,
startle easily, and experience subjective distress often described as "shaking
inside." Grand mal seizures may appear 7 to 48 hours after cessation
•Autonomic hyperactivity (e.g., tachycardia, diaphoresis, elevated blood pressure)
•Severe disturbance in sensorium (e.g., disorientation, clouding of consciousness)
•Perceptual disturbances (e.g., visual or tactile hallucinations)
•Fluctuating levels of consciousness (e.g., ranging from hyperexcitability to
lethargy)
•Delusions (paranoid), agitated behaviors, and fever (temperatures of 100° to
103° F)
alzheimers Ans✓✓✓ a complex disease that begins to damage the brain long
before the symptoms appear. AD affects processes that keep the neurons healthy,
such as (1) communication, (2) metabolism, and (3) repair. In a healthy brain
neurons are supported by microtubules, which guide nutrients and molecules
between the cell body and the axon terminals. A special protein called tau protein
is responsible for the stability of the microtubules. In AD tau protein is subjected
to chemical changes, which result in neurofibrillary tangles and cause
disintegration of the microtubules, thus collapsing the neuron's transport system.
This disintegration of the neuron transport system results in malfunction of
communication between neurons, and eventually leads to neural cell death. It is
the destruction and death of the cells that causes memory failure, personality
changes, problems in carrying out daily activities, and other features of the
disease
alzheimers meds Ans✓✓✓ galantamine hydrobromide (Razadyne), rivastigmine
tartrate (Exelon), and donepezil hydrochloride (Aricept)
, These are called cholinesterase inhibitors that help dely the progression of the
disease in the mild to moderate stage
alzheimers meds Ans✓✓✓ Memantine hydrochloride (Namenda), an N-methyl-d-
aspartate (NMDA), is an antagonist at the NMDA-glutamatergic ion channels. This
drug works by blocking the toxic effects associated with excess glutamate and
regulates glutamate activation. It is the first drug to target symptoms of AD during
the moderate to severe stages of the disorder
anorexia Ans✓✓✓ engage in self-starvation, express intense fear of gaining
weight, and have a disturbance in self-evaluation of weight and its importance.
often experience amenorrhea
• Weight loss more than 30% over 6 months • Rapid decline in weight • Inability
to gain weight with outpatient treatment • Severe hypothermia caused by loss of
subcutaneous tissue or dehydration (body temperature lower than 36° C or 96.8°
F) • Heart rate less than 40 beats per minute • Systolic blood pressure less than
70 mm Hg • Hypokalemia (less than 3 mEq/L) or other electrolyte disturbances
not corrected by oral supplementation • Electrocardiographic changes (especially
dysrhythmias)
Milelu therapy:
These modalities are designed to normalize eating patterns and to begin to
address the issues raised by the illness. The milieu of an eating disorder unit is
purposefully organized to assist the patient in establishing more adaptive
behavioral patterns, including normalization of eating.
