Nurs 5315 UTA exam 1
-blastoma - ANS-from blasts or immature cells- neuroblastoma
-oma - ANS-benign tumor- lipoma
acid base balance impact on K+ - ANS-hydrogen enters mobile, permitting potassium to
break out for the duration of acidosis, visa versa for alkalosis
Adrenergic dealers impact on K+ - ANS-albuterol, beta blockers, and alpha adrenergic
antagonists motive K+ movement into the cell. Alpha adrenergic receptors shift K+ out of the
cell
Allele - ANS-Paired genes on autosomal chromosomes
Alpha Fetoprotein Origin - ANS-Liver and germ cellular tumors
Antidiuretic hormone - ANS-Secreted by means of pituitary gland in response to water
deficit, Na extra, or hypotension. Causes kidneys to reabsorb water, growing plasma extent
Atrophy - ANS-E. Cells lower in size
P. Still functional
Physiologic: thymus gland in early early life
Pathological: disuse
Autosomal chromosomes - ANS-first 22 of 23 chromosomes
Beta Human Chorionic gonadotropin origin - ANS-germ mobile tumors
Breast ca metastasis - ANS-Liver, lungs, mind, bones
calcium and albumin - ANS-When blood levels of albumin are low, serum calcium levels will
also be low as there's less protein to be had for binding
calcium and ionized calcium - ANS-acidosis increases ionized Ca+
alkalosis decreases ionized Ca+ and can motive signs and symptoms of hypocalcemia
calcium and phosphorus courting - ANS-inverse courting- if one increases= different one
decreases, rehulated through PTH, Vit D, calcitonin
Carcino- - ANS-from epithelial tissue- renal cell carcinoma
Carcinoembryonic Antigen - ANS-GI, pancreas, lung, breast tumors
Carcinoma in situ - ANS-preinvasive epithelial malignant tumors of glandular or squamous
cells- cervix
Carrier - ANS-Person who has a diseased gene however is phenotypically everyday
Caseous Necrosis - ANS-lung sickness- commonly TB- tissue seems like clumped cheese
Cell lysis impact on K+ - ANS-intracellular K+ is released into bloodstream
Coagulative Necrosis - ANS-kidneys, coronary heart, adrenals- secondary to hypoxia
Codominance - ANS-Both alleles show off (blood type AB)
Colorectal ca metastasis - ANS-Liver, lungs
Dominant - ANS-Trait visible in phenotype
Dysplasia - ANS-E. Cells alternate in size, shape, agency
P. AKA odd hyperplasia, a disorderly proliferation
Physiologic: N/A
Pathologic: squamous dysplasia of cervix from HPV
dystrophic calcification - ANS-accumulation of Ca in lifeless or loss of life tissues
calcium salt clump and harden- intrude with cellular structure and feature
r/t pulmonary TB, atherosclerosis, injured coronary heart valves, persistent pancreatitis
, Edema - ANS-Accumulation of fluid within the interstitial space- venous obstruction, Na and
water retention
C.M. May be localized or based, tightness of skin, facial swelling, rales, decreased wound
healing, improved hazard of pressure sores, weight gain
Effective arterial blood volume - ANS-The quantity of blood within the arterial space- ECF
changes will motive modifications inside the EABV inside the equal path
Ethanol - ANS-E. Temper altering drug, long time consequences on liver and nutritional
popularity
P. Metabolized by way of liver, generates free radicals
C.M. CNS melancholy, nutrient deficiencies-Mag, Vit B6, thiamine, PO4, irritation and fatty
infiltration of liver, hepatomegaly, leads to liver failure irreversible
Euvolemic Hypernatremia - ANS-general frame water loss, typically from DI
C.M. Extreme polyuria and moderate hypernatremia, weight loss, weak pulses, tachycardia,
postural hypotension, fever, stressed
Exercise effect on K+ - ANS-mobile ATP is dwindled, establishing K+ channels and allowing
K+ to leave cell
Fat Necrosis - ANS-breast, pancreas, stomach structures- creates soaps
Fatty Infiltration - ANS-intracellular accumulation of lipids inside the liver
liver fails to metabolize lipids. Typically from ETOH or excessive fats food plan. Can cause
cirrhosis
Fluid extent deficit - ANS-Dehydration- consumption isn't always enough for frame's desires
C.M. Poor skin turgor, dry mucous membranes, sunken eyes, sunken fontanelles, reduced
urine output, fatigue
Fluid volume extra - ANS-Fluid consumption exceeds body's wishes
C.M. Edema, rales, HTN, weight gain, bounding pulses, intake> output, JVD, restlessness or
anxiety
Free radical and ROS - ANS-E. Ordinary byproduct of ATP production, will weigh down the
mitochondria- exhaust intracellular antioxidants
P. Lipid peroxidation, damage proteins, fragment DNA
C.M. Development in Alzheimer's, heart disease, Parkinson's sickness, Amyotrophic Lateral
Sclerosis
Gangrenous Necrosis - ANS-Dry- darkish shrunk skin
Wet- internal organs- can lead to dying
Gas- from clostridium- antitoxins and hyperbaric remedy
Genotype - ANS-A map of ones specific genes
Gout - ANS-E. Disturbances in serum urate levels. Uncommon for < 30 years old.
P. Uric acid is deposited in the tissues of kidney, heart, earlobes, and joints.
C.M. Inflammation, painful joints. Result of diuretic use or diet high in cream sauces, red
wine, or red meat
Head and neck ca metastasis - ANS-Liver, bones, lymphatics
Heterozygous - ANS-When both a dominant and a recessive allele are present
Homozygous - ANS-Two dominant or recessive alleles
Hydrostatic pressure - ANS-Blood pressure- pushes fluid outside of the vessels, the force of
fluid against the walls of a compartment- venous obstruction, Na and water retention
Hyperplasia - ANS-E. Increase in cell number
P. Increased cellular division
Physiologic: liver regeneration
Pathologic: endometrial- usually r/t hormones
-blastoma - ANS-from blasts or immature cells- neuroblastoma
-oma - ANS-benign tumor- lipoma
acid base balance impact on K+ - ANS-hydrogen enters mobile, permitting potassium to
break out for the duration of acidosis, visa versa for alkalosis
Adrenergic dealers impact on K+ - ANS-albuterol, beta blockers, and alpha adrenergic
antagonists motive K+ movement into the cell. Alpha adrenergic receptors shift K+ out of the
cell
Allele - ANS-Paired genes on autosomal chromosomes
Alpha Fetoprotein Origin - ANS-Liver and germ cellular tumors
Antidiuretic hormone - ANS-Secreted by means of pituitary gland in response to water
deficit, Na extra, or hypotension. Causes kidneys to reabsorb water, growing plasma extent
Atrophy - ANS-E. Cells lower in size
P. Still functional
Physiologic: thymus gland in early early life
Pathological: disuse
Autosomal chromosomes - ANS-first 22 of 23 chromosomes
Beta Human Chorionic gonadotropin origin - ANS-germ mobile tumors
Breast ca metastasis - ANS-Liver, lungs, mind, bones
calcium and albumin - ANS-When blood levels of albumin are low, serum calcium levels will
also be low as there's less protein to be had for binding
calcium and ionized calcium - ANS-acidosis increases ionized Ca+
alkalosis decreases ionized Ca+ and can motive signs and symptoms of hypocalcemia
calcium and phosphorus courting - ANS-inverse courting- if one increases= different one
decreases, rehulated through PTH, Vit D, calcitonin
Carcino- - ANS-from epithelial tissue- renal cell carcinoma
Carcinoembryonic Antigen - ANS-GI, pancreas, lung, breast tumors
Carcinoma in situ - ANS-preinvasive epithelial malignant tumors of glandular or squamous
cells- cervix
Carrier - ANS-Person who has a diseased gene however is phenotypically everyday
Caseous Necrosis - ANS-lung sickness- commonly TB- tissue seems like clumped cheese
Cell lysis impact on K+ - ANS-intracellular K+ is released into bloodstream
Coagulative Necrosis - ANS-kidneys, coronary heart, adrenals- secondary to hypoxia
Codominance - ANS-Both alleles show off (blood type AB)
Colorectal ca metastasis - ANS-Liver, lungs
Dominant - ANS-Trait visible in phenotype
Dysplasia - ANS-E. Cells alternate in size, shape, agency
P. AKA odd hyperplasia, a disorderly proliferation
Physiologic: N/A
Pathologic: squamous dysplasia of cervix from HPV
dystrophic calcification - ANS-accumulation of Ca in lifeless or loss of life tissues
calcium salt clump and harden- intrude with cellular structure and feature
r/t pulmonary TB, atherosclerosis, injured coronary heart valves, persistent pancreatitis
, Edema - ANS-Accumulation of fluid within the interstitial space- venous obstruction, Na and
water retention
C.M. May be localized or based, tightness of skin, facial swelling, rales, decreased wound
healing, improved hazard of pressure sores, weight gain
Effective arterial blood volume - ANS-The quantity of blood within the arterial space- ECF
changes will motive modifications inside the EABV inside the equal path
Ethanol - ANS-E. Temper altering drug, long time consequences on liver and nutritional
popularity
P. Metabolized by way of liver, generates free radicals
C.M. CNS melancholy, nutrient deficiencies-Mag, Vit B6, thiamine, PO4, irritation and fatty
infiltration of liver, hepatomegaly, leads to liver failure irreversible
Euvolemic Hypernatremia - ANS-general frame water loss, typically from DI
C.M. Extreme polyuria and moderate hypernatremia, weight loss, weak pulses, tachycardia,
postural hypotension, fever, stressed
Exercise effect on K+ - ANS-mobile ATP is dwindled, establishing K+ channels and allowing
K+ to leave cell
Fat Necrosis - ANS-breast, pancreas, stomach structures- creates soaps
Fatty Infiltration - ANS-intracellular accumulation of lipids inside the liver
liver fails to metabolize lipids. Typically from ETOH or excessive fats food plan. Can cause
cirrhosis
Fluid extent deficit - ANS-Dehydration- consumption isn't always enough for frame's desires
C.M. Poor skin turgor, dry mucous membranes, sunken eyes, sunken fontanelles, reduced
urine output, fatigue
Fluid volume extra - ANS-Fluid consumption exceeds body's wishes
C.M. Edema, rales, HTN, weight gain, bounding pulses, intake> output, JVD, restlessness or
anxiety
Free radical and ROS - ANS-E. Ordinary byproduct of ATP production, will weigh down the
mitochondria- exhaust intracellular antioxidants
P. Lipid peroxidation, damage proteins, fragment DNA
C.M. Development in Alzheimer's, heart disease, Parkinson's sickness, Amyotrophic Lateral
Sclerosis
Gangrenous Necrosis - ANS-Dry- darkish shrunk skin
Wet- internal organs- can lead to dying
Gas- from clostridium- antitoxins and hyperbaric remedy
Genotype - ANS-A map of ones specific genes
Gout - ANS-E. Disturbances in serum urate levels. Uncommon for < 30 years old.
P. Uric acid is deposited in the tissues of kidney, heart, earlobes, and joints.
C.M. Inflammation, painful joints. Result of diuretic use or diet high in cream sauces, red
wine, or red meat
Head and neck ca metastasis - ANS-Liver, bones, lymphatics
Heterozygous - ANS-When both a dominant and a recessive allele are present
Homozygous - ANS-Two dominant or recessive alleles
Hydrostatic pressure - ANS-Blood pressure- pushes fluid outside of the vessels, the force of
fluid against the walls of a compartment- venous obstruction, Na and water retention
Hyperplasia - ANS-E. Increase in cell number
P. Increased cellular division
Physiologic: liver regeneration
Pathologic: endometrial- usually r/t hormones
