CCRN Exam Questions and Answers
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SIADH - ✔✔too much water, dilutional hyponatremia. Decreased
osmolarity=hypoosmolar. Decreased urinary output.
CSF normal protein, glucose, WBCs, specific gravity, - ✔✔Protein <100,
Glucose: 70 WBCs: 4 cells/mm2 Specific gravity 1.007
Poikothermia - ✔✔fluctuation of core body temperature of more than 2° C
due to changes in ambient room temperature
pathophysiology of a seizure - ✔✔neurons in the cerebral cortex fire at the
same time in a paroxysmal burst.
System driven outcome - ✔✔include length of stay, readmission rate, and
resource utilization.
Arterial supply to the brain: vertebrobasilar, common carotid, meningeal
arteries - ✔✔The vertebrobasilar arteries supply the posterior portion of the
brain. The common carotid arteries supply the anterior area of the brain.
The meningeal arteries supply the superior portion of the brain.
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Pheochromocytoma - ✔✔adrenal medulla,hi epi/norepi. s/s: hypertension,
sweating, headache, palpitations, apprehension, nausea/vomiting, tremor,
pallor, abdominal pain, chest pain, and hyperglycemia.
Acute radiation syndrome - ✔✔large doses of ionizing radiation ,
Circulatory collapse, increased intracranial pressure, vasculitis, and
meningitis causing death within 3 days
Complications of SIADH - ✔✔seizure activity
Treatment of SIADH (avoid what solutions?) - ✔✔Fluid restriction
3% nacl (1500 osmolarity over 25cc/hr or less)
Dont do hypotonic solutions!
Asses for fluid overload
hypertonic solutions - ✔✔D5LR; D5 1/2 NS; D5NS
hypotonic solutions - ✔✔0.5% NS (HNS or 0.45% NS); 2.5% dextrose in
0.45% NS (D2.5 45% NS)
Osmolality and Sodium - ✔✔275-295= normal osmolality. Sodium=135-
145. Usually 2X of Na
Causes of SIADH - ✔✔Viral PNA
Oat cell carcinoma
Head problems
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Increased serum osmolality
Anesthesia and analgesics
Stress
Diabetes insipidus (urine specific gravity?) - ✔✔No ADH, can't keep water,
increased UOP. Hypernatremic, hyperosmolar, increased urinary output (6-
24L a day of clear urine)
urine specific gravity 1.001-1.005
Severe hypovolemia
Causes of diabetes insipidus (what medication?) - ✔✔Head problem
Dilantin (DI)
Treatment of diabetes insipidus (medication, fluid, monitoring x2) -
✔✔Pitressin/vasopressin (same as ADH)
Give fluids (increase intravascular volume)
Monitor urine specific gravity
EKG monitor for ischemia
Hypoglycemia s/s - ✔✔Tachycardia, palpitations, diaphoresis, irritable,
restlessness
Confusion, lethargy, slurred speech, seizure, coma, death. IF YOU ARE IN
A BETA ADRENERGIC BLOCKER, you only see the CNS symptoms
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DKA (BS, breathing, acid vs K) - ✔✔Blood Sugar 400 to 900, Dehydration,
No insulin, Ketones, Kussmaul breathing
Whenever high acid =hi K. For every drop of 0.1 in pH =increase by 0.6 of
K
HHNK (who gets, BS, breaths) - ✔✔old age, diet controlled diabetics, TPN
patients, who get a lot of inteavascular sugar, and pancreatitis as pancreas
is eating itself, does not work properly.
Blood sugar 1000-2000, severe dehydration, (6 to 10 Liters behind.
Patient still makes insulin, so it can occur over months, preventing the
breakdown of fats which causes no acidosis, Shallow breaths.
Treatment DKA - ✔✔insulin (a lot)
A fair amount fluids first saline and then D5 1/2 NS
Treatment HHNK - ✔✔Only a little insulin
A lot if fluids
Leukopenia - ✔✔Abnormally low WBC count < 5000.
Caused by viral illness, bone marrow disorder or medications such as
chemotherapy, HIV regimens, lupus and its meds, antibiotics such as
bactrim and immunosuppressive meds. Patients present with malaise,
chills, fever.