BLOOD GLUCOSE
HORMONES :
produced in
glands ,
carried MECHANISM OF HORMONE ACTION -
second messenger model
in blood plasma to target cells , effective in -
used by glucagon and adrenaline in control of blood glucose
low concentrations but have widespread and Mechanism involving adrenaline
long lasting effects ☒
Adrenaline binds to a transmembrane protein receptor ,
this
ROLE OF PANCREAS :
produces digestive enzymes causes the
protein to change shape in the membrane
and hormones for regulating blood glucose in
•
Change in
.
shape activates adesyl cyclase enzyme converting
islets of langerhans Includes larger a- cells ATP to cyclic AMP le Amn
producing glucagon and B-cells for insulin ) •
CAMP (secondary messenger ) birds to protein kinase
enzyme
lulls ) activating it
ROLE OF LIVER : contains hepatocytes changing its shape i.
where hormones produced in the pancreas • active protein kinase catalyses conversion of glycogen to glucose
have an effect which moves out of the liver cell by facilitated diffusion
Glycogen glucose glycogen to store
s.si → and into the blood through channel proteins
Glycogenlysis glycogen glucose diffuse into blood
: → ADRENALINE : produced at times of stress or excitement
Gluconeogenesis glucose from other
:
sources in the adrenal glands this raises
,
blood glucose cone by : .
SOURCES OF BLOOD GLUCOSE : diet /absorbed •
attaching to protein receptors on
target cells membrane
from hydrolysis of carbohydrates glycogenlysis ,
•
activating enzymes causing breakdown of glycogen to glucose
in muscles and liver or
gluconeogenesis in the line
HORMONE INTERACTION IN GLUCOSE CONC .
-
Insulin and glucagon act antagonistically 1 opposites) ,
Glucose is a substrate needed for
insulin towers glucose cone .
yet glucagon increases it respiration if cone .
falls too low
in self -
regulating negative feedback where glucose cells will be deprived of energy
IF potential
conc .
determines how much insulin or
glucagon rs conc . raises too high water
produced allowing highly sensitive control of cone .
of blood would be lowered causing
-
Blood glucose cone . is not constant but fluctuates dehydration
around an optimum point due negative feedback
to
only when conc . rises or falls beyond certaina INSULIN ANL GLUCAGON →
point is hormone secretion charged
HORMONES :
produced in
glands ,
carried MECHANISM OF HORMONE ACTION -
second messenger model
in blood plasma to target cells , effective in -
used by glucagon and adrenaline in control of blood glucose
low concentrations but have widespread and Mechanism involving adrenaline
long lasting effects ☒
Adrenaline binds to a transmembrane protein receptor ,
this
ROLE OF PANCREAS :
produces digestive enzymes causes the
protein to change shape in the membrane
and hormones for regulating blood glucose in
•
Change in
.
shape activates adesyl cyclase enzyme converting
islets of langerhans Includes larger a- cells ATP to cyclic AMP le Amn
producing glucagon and B-cells for insulin ) •
CAMP (secondary messenger ) birds to protein kinase
enzyme
lulls ) activating it
ROLE OF LIVER : contains hepatocytes changing its shape i.
where hormones produced in the pancreas • active protein kinase catalyses conversion of glycogen to glucose
have an effect which moves out of the liver cell by facilitated diffusion
Glycogen glucose glycogen to store
s.si → and into the blood through channel proteins
Glycogenlysis glycogen glucose diffuse into blood
: → ADRENALINE : produced at times of stress or excitement
Gluconeogenesis glucose from other
:
sources in the adrenal glands this raises
,
blood glucose cone by : .
SOURCES OF BLOOD GLUCOSE : diet /absorbed •
attaching to protein receptors on
target cells membrane
from hydrolysis of carbohydrates glycogenlysis ,
•
activating enzymes causing breakdown of glycogen to glucose
in muscles and liver or
gluconeogenesis in the line
HORMONE INTERACTION IN GLUCOSE CONC .
-
Insulin and glucagon act antagonistically 1 opposites) ,
Glucose is a substrate needed for
insulin towers glucose cone .
yet glucagon increases it respiration if cone .
falls too low
in self -
regulating negative feedback where glucose cells will be deprived of energy
IF potential
conc .
determines how much insulin or
glucagon rs conc . raises too high water
produced allowing highly sensitive control of cone .
of blood would be lowered causing
-
Blood glucose cone . is not constant but fluctuates dehydration
around an optimum point due negative feedback
to
only when conc . rises or falls beyond certaina INSULIN ANL GLUCAGON →
point is hormone secretion charged
