Exam #3 NUR 3125 – Neuro Latest
Tests with All Correct Solutions
Graded A+ 2026 Updated.
migraine headache - Answer trigeminal nerve activated -> releases neuropeptides ->
inflammation within meningeal vasculature
clinical manifestations: THROBBING, UNILATERAL, N/V, sensitivity to light/sound (photophobia),
visual disturbances, aura, speech disturbances
cluster headache clinical manifestations - Answer rapid onset, pain behind EYE (radiates),
conjunctival redness, lacrimation, nasal congestion, rhinorrhea, PTOSIS
tension type headache - Answer muscle tension in scalp and neck, oromandibular
dysfunction, STRESS, ANXIETY
clinical manifestations: HATBAND distribution around head, NOT associated with N/V
"band is around head"
myasthenia gravis etiology - Answer - women with peak incidence in young adulthood
- 70% have thymic disorder (thymus gland)
myasthenia gravis - Answer autoimmune disease caused by LOSS OF FUNCTIONING
ACETYLCHOLINE receptors in neuromuscular junction
- manifested by weakness and easy fatigability
myasthenia gravis clinical manifestations - Answer - myasthenic crisis: sudden weakness ->
during stress, ETOH, cold -> may require ventilation
- PTOSIS
, - autoimmune syndrome -> ACUTE, INFLUENZA-LIKE illness prior (have they been sick)
guillain-barre syndrome clinical manifestations - Answer weakness, numbness, paresthesia -
starts @ legs - ascending, bilaterally, can lead to paralysis
parkinson disease - Answer - progressive LOSS OF DOPAMINE-PRODUCING CELLS in the
substantia nigra which is within the basal ganglia
- LEWY BODIES present
- > 65 years
parkinson disease clinical manifestations - Answer TREMOR, rigidity, BRADYKINESIA, "PILL
ROLLING", cognitive dysfunction (little emotion in face)
acetylcholine - Answer STIMULATES muscle movement
dopamine - Answer INHIBITORY effect on movement
ALS (lou gehrig's disease) - Answer - middle-late adulthood: men >
- progressive neurodegenerative disorder that selectively affects motor function of upper and
lower motor neurons
ALS (lou gehrig's disease) clinical manifestations - Answer weakness, spasticity, stiffness,
IMPAIRED FINE MOTOR control, DYSPHAGIA, DYSARTHRIA, MUSCLE ATROPHY, causes
respiratory failure, INTELLECT/COGNITIVE FUNCTION INTACT & sensation
multiple sclerosis (MS) - Answer - lesions = hard, sharp-edged, DYEMYELINATED PATCHES
visible throughout WHITE MATTER of CNS -> PLAQUES
- found in optic nerves, periventricular white matter, brainstem, cerebellum
multiple sclerosis (MS) clinical manifestations - Answer blurred vision, speech & swallowing,
muscle strength, gait & coordination, paresthesia, vertigo, fatigue
spinal shock - Answer - primary injury results in areflexia: flaccid muscles, paralysis and
absence of sensation at and below level of injury
Tests with All Correct Solutions
Graded A+ 2026 Updated.
migraine headache - Answer trigeminal nerve activated -> releases neuropeptides ->
inflammation within meningeal vasculature
clinical manifestations: THROBBING, UNILATERAL, N/V, sensitivity to light/sound (photophobia),
visual disturbances, aura, speech disturbances
cluster headache clinical manifestations - Answer rapid onset, pain behind EYE (radiates),
conjunctival redness, lacrimation, nasal congestion, rhinorrhea, PTOSIS
tension type headache - Answer muscle tension in scalp and neck, oromandibular
dysfunction, STRESS, ANXIETY
clinical manifestations: HATBAND distribution around head, NOT associated with N/V
"band is around head"
myasthenia gravis etiology - Answer - women with peak incidence in young adulthood
- 70% have thymic disorder (thymus gland)
myasthenia gravis - Answer autoimmune disease caused by LOSS OF FUNCTIONING
ACETYLCHOLINE receptors in neuromuscular junction
- manifested by weakness and easy fatigability
myasthenia gravis clinical manifestations - Answer - myasthenic crisis: sudden weakness ->
during stress, ETOH, cold -> may require ventilation
- PTOSIS
, - autoimmune syndrome -> ACUTE, INFLUENZA-LIKE illness prior (have they been sick)
guillain-barre syndrome clinical manifestations - Answer weakness, numbness, paresthesia -
starts @ legs - ascending, bilaterally, can lead to paralysis
parkinson disease - Answer - progressive LOSS OF DOPAMINE-PRODUCING CELLS in the
substantia nigra which is within the basal ganglia
- LEWY BODIES present
- > 65 years
parkinson disease clinical manifestations - Answer TREMOR, rigidity, BRADYKINESIA, "PILL
ROLLING", cognitive dysfunction (little emotion in face)
acetylcholine - Answer STIMULATES muscle movement
dopamine - Answer INHIBITORY effect on movement
ALS (lou gehrig's disease) - Answer - middle-late adulthood: men >
- progressive neurodegenerative disorder that selectively affects motor function of upper and
lower motor neurons
ALS (lou gehrig's disease) clinical manifestations - Answer weakness, spasticity, stiffness,
IMPAIRED FINE MOTOR control, DYSPHAGIA, DYSARTHRIA, MUSCLE ATROPHY, causes
respiratory failure, INTELLECT/COGNITIVE FUNCTION INTACT & sensation
multiple sclerosis (MS) - Answer - lesions = hard, sharp-edged, DYEMYELINATED PATCHES
visible throughout WHITE MATTER of CNS -> PLAQUES
- found in optic nerves, periventricular white matter, brainstem, cerebellum
multiple sclerosis (MS) clinical manifestations - Answer blurred vision, speech & swallowing,
muscle strength, gait & coordination, paresthesia, vertigo, fatigue
spinal shock - Answer - primary injury results in areflexia: flaccid muscles, paralysis and
absence of sensation at and below level of injury
