NURS 341 EXAM 2 – Questions With Fully Explained
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Sherpath: Diabetes Mellitus Chapter 21 Test Case Study 4 (Short
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Terms in this set (82)
what are the local response hallmarck signs 1. redness
of inflammation (6)? 2. heat
3. swelling
4. loss of function
5. PAIN
6. purulent exudate
what are the systemic response hallmarck 1. leukocytosis - increase WBC
signs of inflammation (7)? 2. malaise - restless
3. nausea
4. fatigue
5. increased HR and RR - compensate for low BP
6. fever
7. elevated CRP and ESR
what is inflammation? a predictable, nonspecific response to cellular injury and
beings with mast cell granulation
what is the vascular response to what happens to the vessels
inflammation?
vasodilation and increased capillary permeability
exudate
what is the cellular response to brings infection fighters and wound healers to the site of injury
inflammation? which beings at the release of CHEMOTACTIC factors
involves: margination, diapedisis, neutrophils,
monocytes/macrophages, lymphocytes
what is margination of the cellular accumulations of neutrophils on the walls of capillaries
response?
what is diapedesis of the cellular response? cells leaving the inside of the vessel and moving out to the
area of injury
, what are neutrophils of the cellular NON-SPECIFIC
response?
first ones on the site of infection to take care of the injury no
matter what is causing it
what are monocytes/macrophages of the NON-SPECIFIC
cellular response?
first ones on the site of infection to take care of the injury no
matter what is causing it
important for phagocytosis
what are lymphocytes of the cellular SPECIFIC
response?
T or B lymphocytes show for specific antigens
what are the different types of exudate (4)? 1. serous - straw watery
2. sanguineous - bloody
3. serosanguineous - watery straw and bloody
4. purulent - yellow, pus SIGN OF INFECTION
what is needed for effective wound healing? 1. adequate nutrition - protein
2. adequate blood supply - to bring oxygen to the cells
what medications supress the inflammatory 1. antihistamines
response? 2. corticosteroids
3. NSAIDS
what is mast cell degranulation? the start of inflammatory process
large cells send messages to other tissues in response to
cellular injury the burst open and release chemical mediators
(histamine, kinins, prostaglandins, chemotactic factors)
what is histamine in mast cell degranulation? casues vasodilation and increased permeability and more
mast cells degranulation
this is NOT good for someone with major injuries because it
causes a lot of vasodilation and blood pressure to go down
which impaires oxygenation and perfusion
how does histamine effect oxygenation? it causes increased capillary permeability and gas exchange
occurs in the capillary membrane. SO when they are leaky the
fill with fluid and impair gas exchange.
how does histamine effect perfusion? it causes a lot of vasodilation and brings the blood pressure
down. someone can't have good perfusion without adequate
blood pressure.
what are kinins in mast cell degranulation? cause even more vasodilation and increased capillary
permeability
what are prostaglandins in mast cell responsible for pain
degranulation?
pain medications block this
what are chemotactic factors in mast cell direct infection fighters and wound healers (WBC) to the site
degranulation? of injury
Solutions
Save
Students also studied
Sherpath: Diabetes Mellitus Chapter 21 Test Case Study 4 (Short
30 terms 24 terms 22 terms
jesssbee Preview kenanderson2017 Preview Brittany182
Terms in this set (82)
what are the local response hallmarck signs 1. redness
of inflammation (6)? 2. heat
3. swelling
4. loss of function
5. PAIN
6. purulent exudate
what are the systemic response hallmarck 1. leukocytosis - increase WBC
signs of inflammation (7)? 2. malaise - restless
3. nausea
4. fatigue
5. increased HR and RR - compensate for low BP
6. fever
7. elevated CRP and ESR
what is inflammation? a predictable, nonspecific response to cellular injury and
beings with mast cell granulation
what is the vascular response to what happens to the vessels
inflammation?
vasodilation and increased capillary permeability
exudate
what is the cellular response to brings infection fighters and wound healers to the site of injury
inflammation? which beings at the release of CHEMOTACTIC factors
involves: margination, diapedisis, neutrophils,
monocytes/macrophages, lymphocytes
what is margination of the cellular accumulations of neutrophils on the walls of capillaries
response?
what is diapedesis of the cellular response? cells leaving the inside of the vessel and moving out to the
area of injury
, what are neutrophils of the cellular NON-SPECIFIC
response?
first ones on the site of infection to take care of the injury no
matter what is causing it
what are monocytes/macrophages of the NON-SPECIFIC
cellular response?
first ones on the site of infection to take care of the injury no
matter what is causing it
important for phagocytosis
what are lymphocytes of the cellular SPECIFIC
response?
T or B lymphocytes show for specific antigens
what are the different types of exudate (4)? 1. serous - straw watery
2. sanguineous - bloody
3. serosanguineous - watery straw and bloody
4. purulent - yellow, pus SIGN OF INFECTION
what is needed for effective wound healing? 1. adequate nutrition - protein
2. adequate blood supply - to bring oxygen to the cells
what medications supress the inflammatory 1. antihistamines
response? 2. corticosteroids
3. NSAIDS
what is mast cell degranulation? the start of inflammatory process
large cells send messages to other tissues in response to
cellular injury the burst open and release chemical mediators
(histamine, kinins, prostaglandins, chemotactic factors)
what is histamine in mast cell degranulation? casues vasodilation and increased permeability and more
mast cells degranulation
this is NOT good for someone with major injuries because it
causes a lot of vasodilation and blood pressure to go down
which impaires oxygenation and perfusion
how does histamine effect oxygenation? it causes increased capillary permeability and gas exchange
occurs in the capillary membrane. SO when they are leaky the
fill with fluid and impair gas exchange.
how does histamine effect perfusion? it causes a lot of vasodilation and brings the blood pressure
down. someone can't have good perfusion without adequate
blood pressure.
what are kinins in mast cell degranulation? cause even more vasodilation and increased capillary
permeability
what are prostaglandins in mast cell responsible for pain
degranulation?
pain medications block this
what are chemotactic factors in mast cell direct infection fighters and wound healers (WBC) to the site
degranulation? of injury
