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NR 507: Advanced Pathophysiology Midterm Exam Complete Question 1-100 April 2025

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NR 507: Advanced Pathophysiology Midterm Exam Complete Question 1-100 April 2025

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NR 507: Advanced Pathophysiology
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NR 507: Advanced Pathophysiology Midterm Exam
Complete Question 1-100 April 2025

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Practice questions for this set


Learn 1 /7 Study with Learn




- smooth muscle hypertrophy
- hypertrophy and hyperplasia of goblet cells
- epithelial cell metaplasia
- migration of more WBC to site
- thickening and rigidity of bronchial basement membrane



Choose an answer



what does long term exposure to irritants
1 Asthma 2
promote in bronchitis? (5)



3 alveolar hyperinflation 4 3 layers of the bronchioles



Don't know?




Terms in this set (167)


Chronic disease due to bronchoconstriction and an excessive inflammatory
Asthma
response in the bronchioles

, coughing
wheezing
What are 5 s/s of asthma shortness of breath
rapid breathing
chest tightness

-airway inflammation, bronchial hyper-reactivity and smooth muscle spasm
-excess mucus production and accumulation
Pathophysiology of asthma (5) -hypertrophy of bronchial smooth muscle
-airflow obstruction
-decreased alveolar ventilation

Bronchioles smaller passageways that originate from the bronchi that become the alveoli

innermost layer
3 layers of the bronchioles middle layer - lamina propria
outermost layer

lamina propria the middle layer of the bronchioles

structure of the lamina propria embedded with connective tissue cells and immune cells

purpose of the lamina propria white blood cells are present to help protect the airways

How does the lamina propria effect the the WBCs protective feature goes into overdrive causing an inflammatory
lungs in regards to asthma response that damages host tissue

What does the innermost layer of the columnar epithelial ells and mucus producing goblet cells
bronchioles contain

What does the outermost layer of the smooth muscle cells
bronchioles contain

what does the outermost layer of the control the airways ability to constrict and dilate
bronchioles do

When air is unable to move out of the alveolar like it should due to bronchial walls
alveolar hyperinflation
collapsing around possible mucus plug thus trapping air inside

the ongoing inflammatory process of asthma produces mucus and pus plug that
how does hyperinflation occur?
the bronchial walls collapse around

-expanded thorax and hypercapnia (retention of CO2)
Effect of hyperinflation of the alveolar
- respiratory acidosis

What are two anticholinergic drugs used tiotropium and ipratropium
for asthma

These drugs block the effects of the parasympathetic nervous system
What do anticholinergics do in the lungs?
- increasing bronchodilation

the parasympathetic system is stimulated by the vagal nerve to release
acetylcholine which binds to the cholinergic receptors of the respiratory tract to
cause bronchial constriction = decreased airflow
MOA of anticholinergic drugs for asthma

- blocking the cholinergic receptors prevents acetylcholine binding preventing
the bronchial constriction

bronchitis inflammation of the bronchial tubes

bronchial inflammation
hypersecretion of mucus
3 characteristics of bronchitis
chronic productive cough for at least 3 consecutive months for at least 2
successive years

The supply of oxygen to and removal of wastes from the cells and tissues of the
Perfusion
body as a result of the flow of blood through the capillaries.

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