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Exam (elaborations)

NR 667 VISE Assignment – Comprehensive Case Analysis, Clinical Evaluation & Verified Answers

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Master the NR 667 VISE Assignment with this complete, detailed, and professionally structured study resource. Includes clinical case analysis, VISE evaluation components, assessment criteria, evidence-based decision-making, differential diagnosis support, and verified answers aligned with the NR 667 course requirements. Perfect for AGACNP, FNP, PMHNP, and APRN students who need clear guidance for completing the VISE assignment accurately and confidently. This resource helps improve clinical judgment, documentation skills, and overall assignment performance.

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NR 667
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December 4, 2025
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2025/2026
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NR 667 VISE Assignment

-No known cause in 90% of cases of primary HTN
-Secondary causes: renal failure, kidney disease, renal artery stenosis, Cush
Etiology: Hypertension syndrome, hyper/hypo thyroidism, increased ICP, sleep apnea, oral
contraceptives, steroids, cocaine, NSAIDs, decongestants, sympathomimet
alcohol, antidepressants, caffeine

-Modifiable: smoking, DM, high cholesterol, obesity (single most important
factor in children), physical inactivity, poor diet, excessive sodium intake,
Risk Factors: Hypertension excessive alcohol consumption
-Non-modifiable: CKD, family hx, increased age (>55 men, > 65 women), low
socioeconomic status, low educational status, male sex, OSA, stress, pregn

-Most are asymptomatic; occipital headache, headache upon waking, blurr
vision, fundoscopic exam (AV nicking, exudates, papilledema), left vent.
Assessment: Hypertension
hypertrophy, pregnancy w/HTN and proteinuria, edema, and excessive we
gain

-Secondary HTN, white coat HTN (artificial elevation d/t medical environm
Differential Diagnosis: Hypertension
anxiety)

, NR 667 VISE Assignment
-Urinalysis = proteinuria
-Electrolytes, creatinine, calcium
-Fasting lipid profile and BS
Final Diagnosis: Hypertension
-ECG
-Measure BP twice, 5 mins apart
-Patient should be seated; use proper cuff size and application

-Maintaining healthy weight and BMI
-Smoking cessation
-Regular aerobic exercise
Prevention: Hypertension -Alcohol in moderation (< 1 oz/day)
-Stress management
-Medication compliance
-Assess for and treat OSA

, NR 667 VISE Assignment
-Stage 1: Risk score < 10% =lifestyle modification
-Stage 2: lifestyle + medication
-DASH eating plan: high fruit, veggies, grains; low fat dairy, fish, poultry, be
nuts
-Reduce dietary sodium to 2,300mg/day, increase K+
Non-pharm management: Hypertension
-Reduce sat. fat intake
-Body weight reduction; 1kg of weight reduction = 1 mm/hg bp reduction
-150 mins of aerobic exercise and/or 3 sessions of isometric resistance per
-Treat other underlying diseases
-Check bp 2x/week during pregnancy

-Start medication for primary prevention of CVD if pt. has ASCVD risk ≥ 10%
stage 1 HTN or if ASCVD is < 10% with bp >140/90
-Stage 2: start 2 bp-lowering medications
Pharmacological management: -African Americans: 2+ medications recommended; thiazide and CCBs are t
Hypertension most effective
*DO NOT use ACE and ARB concurrently
-Beta blockers are NOT first line
-Thiazides, CCBs, ACEIs, and ARBs can be used alone or in combo

-Can use beta blockers (labetalol), methyldopa, CCBs (nifedipine)
Pregnancy considerations: Hypertension
-AVOID ARBs and ACEIs

, NR 667 VISE Assignment
-Inquire about adherence and any side effects
Follow-up: Hypertension
-Reassess monthly until patient reaches goal, then every 3-6 months as nee

-Only 54% of treated patients are at goal treatment; expect complications i
Expected course: Hypertension under treated
-Most patients require more than one medication to reach goal bp

-Stroke, CAD, MI, renal failure, heart failure, eclampsia (seizures), pulmonar
Possible Complications: Hypertension
edema, hypertensive crisis, hypertensive retinopathy, ED

-Inherited disorder, high dietary intake, obesity, sedentary lifestyle, DM,
hypothyroidism, anabolic steroid use, hepatitis, cirrhosis, uremia, nephrotic
Etiology: Hyperlipidemia
syndrome, stress, drug-induced (thiazide diuretics, beta blockers, cyclospo
alcohol, caffeine, metabolic syndrome

-Family history, physical inactivity, smoking, age (men > 45, women > 55 or
Risk factors: Hyperlipidemia premature menopause without estrogen replacement), obesity, diet high in
fat, DM

-Few physical findings; xanthomata (fat deposits in the skin), xanthelasma (y
Assessment findings: Hyperlipidemia plaques on the eyelid), corneal arcus prior to age 50 (arc of cholesterol aro
the iris), bruits, angina pectoris, MI, stroke

Differential diagnosis: Hyperlipidemia -Secondary causes: hypothyroidism, pregnancy, DM, non-fasting state

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