NUR 239 Final Exam – Questions With Clear
Final Solutions
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Terms in this set (73)
- beta cells directly lowers blood glucose levels
- promotes glucose uptake by cells, glucose
What is the action of storage
insulin? - prevents glycogen breakdown
- inhibits gluconeogenesis
- promotes fat storage
- alpha cells, increase BG levels
What is the action of
- increased amino acids transport to ivermectin for
glucagon?
glucose production
What are the effects of
insulin on metabolism?
- blood glucose levels between normal &
What is pre diabetes?
diagnostic level
- 5-10% of people diagnosed with diabetes
What is Type 1 diabetes?
- usually younger
- 90-95% of patients with diabetes
What is Type 2 diabetes?
- after 40 years of age also children / teens
- 18% of pregnancies
What is gestational - 2nd or 3rd trimester of pregnancy
diabetes? - hormones from placenta inhibit action of insulin
- high risk for complications with baby & later T2D
What are associated or - pancreatic, endocrine disorders, drug induced
other conditions or (steroids)
syndromes with
diabetes?
, - 3 polys
- polyuria: BG levels exceed what can be filtered by
What are common glomeruli of kidneys, amount reabsorbed by tubules
clinical manifestations of - polydipsia: intracellular dehydration; water pulled
diabetes? out of body cells, early sign that is often overlooked
- polyphagia: body feels like its starving because it
can't use insulin to unlock cells & let glucose in
- absolute deficiency of insulin, results from
destruction of pancreatic beta cells, high blood
glucose levels
- causes: autoimmune, abnormal immune response,
What is T1D?
T cells destroy beta cells, genetic susceptibility,
environmental
- results: decreased insulin production, increased
glucose production by liver, blood glucose level
- sudden onset, severe symptoms initially, difficult to
control
- sudden weight loss despite normal or increased
appetite
What are S&S of T1D? - loss of body tissue because of lack of insulin
forces body to use fat & proteins as source of
energy
- wasting way due to starvation affect
- leads to acidosis -> DKA
- slow, insidious onset, less severe symptoms initially,
less difficult to control
- causes: environmental, obesity, genetic tendency,
What is T2DM? metabolic syndrome
- relative insulin deficiency
- no ketones -> hyperglycemic, hyperosmolar
syndrome
- ABD obesity: waist >35 inches in women, > 40 in
men
What is metabolic - triglycerides > 150 mg/dL
syndrome & increased - HDL <50 mg/dL women, < 40 mg/dL men
risk for T2D? - BP > 130/85
- FBS > 100 mg/dL
- must have 3-5 of these
Final Solutions
Save
Terms in this set (73)
- beta cells directly lowers blood glucose levels
- promotes glucose uptake by cells, glucose
What is the action of storage
insulin? - prevents glycogen breakdown
- inhibits gluconeogenesis
- promotes fat storage
- alpha cells, increase BG levels
What is the action of
- increased amino acids transport to ivermectin for
glucagon?
glucose production
What are the effects of
insulin on metabolism?
- blood glucose levels between normal &
What is pre diabetes?
diagnostic level
- 5-10% of people diagnosed with diabetes
What is Type 1 diabetes?
- usually younger
- 90-95% of patients with diabetes
What is Type 2 diabetes?
- after 40 years of age also children / teens
- 18% of pregnancies
What is gestational - 2nd or 3rd trimester of pregnancy
diabetes? - hormones from placenta inhibit action of insulin
- high risk for complications with baby & later T2D
What are associated or - pancreatic, endocrine disorders, drug induced
other conditions or (steroids)
syndromes with
diabetes?
, - 3 polys
- polyuria: BG levels exceed what can be filtered by
What are common glomeruli of kidneys, amount reabsorbed by tubules
clinical manifestations of - polydipsia: intracellular dehydration; water pulled
diabetes? out of body cells, early sign that is often overlooked
- polyphagia: body feels like its starving because it
can't use insulin to unlock cells & let glucose in
- absolute deficiency of insulin, results from
destruction of pancreatic beta cells, high blood
glucose levels
- causes: autoimmune, abnormal immune response,
What is T1D?
T cells destroy beta cells, genetic susceptibility,
environmental
- results: decreased insulin production, increased
glucose production by liver, blood glucose level
- sudden onset, severe symptoms initially, difficult to
control
- sudden weight loss despite normal or increased
appetite
What are S&S of T1D? - loss of body tissue because of lack of insulin
forces body to use fat & proteins as source of
energy
- wasting way due to starvation affect
- leads to acidosis -> DKA
- slow, insidious onset, less severe symptoms initially,
less difficult to control
- causes: environmental, obesity, genetic tendency,
What is T2DM? metabolic syndrome
- relative insulin deficiency
- no ketones -> hyperglycemic, hyperosmolar
syndrome
- ABD obesity: waist >35 inches in women, > 40 in
men
What is metabolic - triglycerides > 150 mg/dL
syndrome & increased - HDL <50 mg/dL women, < 40 mg/dL men
risk for T2D? - BP > 130/85
- FBS > 100 mg/dL
- must have 3-5 of these
