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APEA- Pharm Urology Exam with Questions and Verified Answers

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APEA- Pharm Urology Exam with Questions and Verified Answers

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APEA- Pharm Urology
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APEA- Pharm Urology
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APEA- Pharm Urology

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June 12, 2025
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APEA- Pharm Urology Exam with Questions and Verified Answers


In patients with chronic kidney disease (CKD), regardless of race or
diabetes status, the initial recommended therapy for hypertension to
improve kidney outcomes is:
aldosterone antagonists.
angiotensin-converting enzyme inhibitors.
beta-blockers.
calcium channel blockers.
angiotensin-converting enzyme inhibitors.




A 48-year-old patient with diabetes, hypertension, and renal insufficiency
is taking an angiotensin receptor blocker (ARB). ARBs act by:
blocking the receptor sites of angiotensinogen in the liver.
blocking the angiotensin I receptors in the cardiomyocytes and fibroblasts.
inhibiting the binding of angiotensin II to angiotensin I receptor sites on
vascular smooth muscles.
inhibiting the conversion of angiotensin I to angiotensin II, blocking the
renin-angiotensin system.
inhibiting the binding of angiotensin II to angiotensin I receptor sites on
vascular smooth muscles.




Fluoroquinolones, such as ciprofloxacin (Cipro), are a preferred choice in
the treatment of acute pyelonephritis because they:
have daily dosing regimens.
are less likely to result in antimicrobial resistance.
are effective in 3-day treatment regimens.
produce high concentrations in the renal medulla.
produce high concentrations in the renal medulla.




Patients receiving thiazide diuretics should be monitored for hypokalemia
and:
hyperuricemia.
hypermagnesemia.
hypernatremia.
hypocalcemia.
hyperuricemia.

, APEA- Pharm Urology Exam with Questions and Verified Answers


Extreme caution should be exercised when administering imipramine (Tofranil)
to a child with:
ataxia.
enuresis.
hypothyroidism.
epilepsy.
epilepsy.


imipramine shown to lower seizure threshold




In the treatment of urge incontinence, anticholinergics relax smooth
muscles in the bladder by:
blocking alpha adrenergic receptors.
interfering with the release of potassium.
inhibiting muscarinic activity of acetylcholine.
interfering with the release of calcium.
inhibiting muscarinic activity of acetylcholine.




Desmopressin acetate (DDAVP) used in the treatment of nocturnal enuresis
should be avoided in patients with:
attention deficit/hyperactivity disorder.
bradycardia.
diabetes.
hyponatremia.
hyponatremia.




Desmopressin acetate (DDAVP), used to treat nocturnal enuresis,:
decreases peripheral vascular resistance and urinary output.
decreases water permeability of the distal convoluted tubules and
collecting duct cells in the kidneys.
increases sodium absorption across the ascending loop of Henle and
decreases permeability of urea and osmolality.
increases water reabsorption in the renal collecting ducts and results in
an increased urine osmolality.
increases water reabsorption in the renal collecting ducts and results in
an increased urine osmolality.

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