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Summary NURS 5315 SHOCK NEWEST FINAL SUMMARIES UPDATED FOR 2025 ACTUAL QUESTIONS WITH 100% VERIFIED SOLUTIONS Shock Shock is defined as a physiologic state characterized by

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NURS 5315 SHOCK NEWEST FINAL SUMMARIES UPDATED FOR 2025 ACTUAL QUESTIONS WITH 100% VERIFIED SOLUTIONS Shock Shock is defined as a physiologic state characterized by

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NURS 5315 SHOCK NEWEST FINAL SUMMARIES UPDATED FOR 2025 ACTUAL
QUESTIONS WITH 100% VERIFIED SOLUTIONS 2023-2025
Shock

Shock is defined as a physiologic state characterized by inadequate tissue perfusion to meet
metabolic demand and tissue oxygenation.

metabolic demand

what cardiac output is normally equal to

How can shock be characterized?

By inadequate peripheral and end organ perfusion. Also it can be characterized by low cardiac
output and decreased level of consciousness.

What types of shock can cause a high cardiac output?

Shock thats caused by sepsis or anaphylaxis cardiac out put can be high

All types of shock can do what to organs?

Can result in impared function of vital organs such as the brain (Decreased level of
consciousness) and kidneys ( Low urine output and ineffective filtering)

What will occur if adequate o2 delivery to the tissues is not maintained?

Organ dysfunction

What are the major functions of the cardiopulmonary system?

Delivers oxygen to body tissues

Removes metabolic by products of cellular metabolism

Tissue hypoxia

This is present when a region of the body or an organ is deprived of adequate O2 supply.

What does cardiac output determine and vascular resistance determine?

Determines adequate blood flow to the tissues

Cardiac output

The volume of blood pumped by the heart per minute is the product of stroke volume and heart
rate

,Cardia output=Stroke volume X Heart Rate

Stroke volume

The volume of blood pumped by the ventricles with each contraction

Cardia output=Stroke volume X Heart Rate

According to this formula if the heart rate decreases, stroke volume must increase
commensurately to maintain the cardiac output.

How can cardiac output increase?

By an increase in heart rate, in stroke volume or both

What are the limits of increased cardia output produced by increasing heart rate?

If the rate is too fast, as can happen with tachyarrhythmias, stroke volume can fall bc there is
inadequate time to fill the heart.




As shock develops, compensatory mechanisms attempt to?

Attempt to maintain O2 delivery to vital organs

vasoconstriction

the constriction of blood vessels, which increases blood pressure.

Name the compensatory mechanisms

Tachycardia

Increased systemic vascular resistance (SVR) (vasoconstriction)

Increased strength of cardiac contraction (Contractility)

Increase in venous smooth muscle tone

The body's first action to maintain cardiac output is to?

Is to increase heart rate, or tachycardia, which can increase cardiac output to a limited degree

, When O2 delivery to the tissues is compromised what happens?

Blood flow is redirected or shunted from nonvital organs and tissues ( like skin, skeletal muscles,
gut, kidneys) to vital organs like brain, heart

What is another mechanism to maintain stroke volume and cardiac output?

Is increased strength of cardiac contractions, or contractility, with more complete emptying of
the ventricles

What else can support stroke volume?

Can be supported by increased venous smooth muscle tone, improving venous return to the
heart and preload

How is blood pressure determined

product of Cardiac output and Systemic Vascular Resistance (SVR)

As cardiac output decreases blood pressure is maintained by?

By an increase in SVR

pulse pressure

The difference between the systolic and diastolic blood pressure often narrows bc increased SVR
raises the diastolic pressure.

systolic pressure

Blood pressure in the arteries during contraction of the ventricles.

diastolic pressure

occurs when the ventricles are relaxed; the lowest pressure against the walls of an artery

If cardiac output is inadequate, tissue perfusion is?

Signs of poor tissue perfusion?

Is compromised, even if blood pressure is normal.

Lactic acidosis and end organ dysfunction will be present even if blood pressure is normal

When SVR cannot increase further blood pressure?

Blood pressure begins to decline. Then O2 delivery to organs is severely compromised
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