ATI Med-Surg 2024 final
exam with verified answers
100% guarantee pass
normal sinus rhythm ECG
A. PR interval length
B. QRS length
C. QT interval - ANSWER A. 0.12-0.20 seconds
B. 0.08-0.12 seconds
C. 0.35-0.43 seconds
A-flutter - ANSWER rapid regular atrial depolarization that
produces sawtooth ECG pattern
treatment:
CCB/BB to reduce HR
anticoagulant
ibutalide (dysrhythmic for A-flutter)
cardioversion
radiofrequency catheter ablation (destruction of tiny parts
of heart that produce extra conduction)
, when to treat bradycardia (HR less than 60) - ANSWER
only if pt is symptomatic
meds that treat bradycardia (2) - ANSWER atropine
isoproterenol
non-med management for bradycardia - ANSWER
pacemaker
meds that manage A-fib, SVT, and V-tach *w/ pulse* (3) -
ANSWER amiodarone
adenosine
verapamil
non-med management for A-fib, SVT, and V-tach *w/
pulse* - ANSWER synchronized cardioversion
meds that manage V-fib and V-tach *without pulse* (3) -
ANSWER amiodarone
lidocaine
epinephrine
non-med management for V-fib and V-tach *without
pulse* - ANSWER defibrillation
what should be done if pt loses pulse during synchronized
cardioversion - ANSWER immediately begin
unsynchronized defibrillation
synchronized cardioversion function - ANSWER delivers
counter-shock to the heart synchronized to QRS complex
,used for A-fib, SVT, and V-tach w/ pulse
defibrillation function - ANSWER deliver unsynchronized
counter-shock to the heart, stopping all electrical activity
so SA node can reestablish rhythm
used for V-fib and V-tach without pulse
premature ventricular contraction (PVC) ECG - ANSWER
wide QRS complex and no P waves--ventricles contract
before atria
hyperkalemia ECG changes (4) - ANSWER tall T-wave
flat P-wave
prolonged PR interval
prolonged QRS complex
prolonged QT can put pt at risk for developing... -
ANSWER torsades de pointes (life threatening)
torsades de pointes ECG - ANSWER rapid irregular QRS
complexes which change in axis (life threatening)
pericardial effusion/cardiac tamponade ECG - ANSWER
low voltage (muffled heart sounds)
types of AV blocks (4) - ANSWER first degree
second degree type I (wencklebach)
second degree type II (mobitz)
third degree (complete)
, first degree heart block ECG - ANSWER conduction delay
at AV node which causes long PR interval
"with a first degree, PR may be close to 0.3!"
second degree heart block (type I) ECG
aka wenckebach block - ANSWER progressive PR
elongation then missing QRS complex
"longer, longer, longer, DROP, now you have a
wencklebach"
second degree heart block (type II) ECG
aka mobitz type II block - ANSWER every few beats entire
beat goes missing without any change in PR interval
"everything normal, then the beat goes shooo, mobitz type
II"
third degree heart block - ANSWER no atrial impulses are
transmitted to the ventricles--atria and ventricles beat
independently
depressed or inverted T waves indicate.. - ANSWER
tissue ischemia
cause of endocarditis - ANSWER infection/inflammation
of endocardium
exam with verified answers
100% guarantee pass
normal sinus rhythm ECG
A. PR interval length
B. QRS length
C. QT interval - ANSWER A. 0.12-0.20 seconds
B. 0.08-0.12 seconds
C. 0.35-0.43 seconds
A-flutter - ANSWER rapid regular atrial depolarization that
produces sawtooth ECG pattern
treatment:
CCB/BB to reduce HR
anticoagulant
ibutalide (dysrhythmic for A-flutter)
cardioversion
radiofrequency catheter ablation (destruction of tiny parts
of heart that produce extra conduction)
, when to treat bradycardia (HR less than 60) - ANSWER
only if pt is symptomatic
meds that treat bradycardia (2) - ANSWER atropine
isoproterenol
non-med management for bradycardia - ANSWER
pacemaker
meds that manage A-fib, SVT, and V-tach *w/ pulse* (3) -
ANSWER amiodarone
adenosine
verapamil
non-med management for A-fib, SVT, and V-tach *w/
pulse* - ANSWER synchronized cardioversion
meds that manage V-fib and V-tach *without pulse* (3) -
ANSWER amiodarone
lidocaine
epinephrine
non-med management for V-fib and V-tach *without
pulse* - ANSWER defibrillation
what should be done if pt loses pulse during synchronized
cardioversion - ANSWER immediately begin
unsynchronized defibrillation
synchronized cardioversion function - ANSWER delivers
counter-shock to the heart synchronized to QRS complex
,used for A-fib, SVT, and V-tach w/ pulse
defibrillation function - ANSWER deliver unsynchronized
counter-shock to the heart, stopping all electrical activity
so SA node can reestablish rhythm
used for V-fib and V-tach without pulse
premature ventricular contraction (PVC) ECG - ANSWER
wide QRS complex and no P waves--ventricles contract
before atria
hyperkalemia ECG changes (4) - ANSWER tall T-wave
flat P-wave
prolonged PR interval
prolonged QRS complex
prolonged QT can put pt at risk for developing... -
ANSWER torsades de pointes (life threatening)
torsades de pointes ECG - ANSWER rapid irregular QRS
complexes which change in axis (life threatening)
pericardial effusion/cardiac tamponade ECG - ANSWER
low voltage (muffled heart sounds)
types of AV blocks (4) - ANSWER first degree
second degree type I (wencklebach)
second degree type II (mobitz)
third degree (complete)
, first degree heart block ECG - ANSWER conduction delay
at AV node which causes long PR interval
"with a first degree, PR may be close to 0.3!"
second degree heart block (type I) ECG
aka wenckebach block - ANSWER progressive PR
elongation then missing QRS complex
"longer, longer, longer, DROP, now you have a
wencklebach"
second degree heart block (type II) ECG
aka mobitz type II block - ANSWER every few beats entire
beat goes missing without any change in PR interval
"everything normal, then the beat goes shooo, mobitz type
II"
third degree heart block - ANSWER no atrial impulses are
transmitted to the ventricles--atria and ventricles beat
independently
depressed or inverted T waves indicate.. - ANSWER
tissue ischemia
cause of endocarditis - ANSWER infection/inflammation
of endocardium
