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Exam (elaborations)

Test Bank for Advanced Physiology and Pathophysiology Essentials for Clinical Practice 2nd Edition Tkacs

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Test Bank for Advanced Physiology and Pathophysiology Essentials for Clinical Practice 2nd Edition Tkacs Test Bank for Advanced Physiology and Pathophysiology Essentials for Clinical Practice 2nd Edition Tkacs Test Bank for Advanced Physiology and Pathophysiology Essentials for Clinical Practice 2nd Edition Tkacs Test Bank for Advanced Physiology and Pathophysiology Essentials for Clinical Practice 2nd Edition Tkacs Test Bank for Advanced Physiology and Pathophysiology Essentials for Clinical Practice 2nd Edition Tkacs Test Bank for Advanced Physiology and Pathophysiology Essentials for Clinical Practice 2nd Edition Tkacs Test Bank for Advanced Physiology and Pathophysiology Essentials for Clinical Practice 2nd Edition Tkacs

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Institution
Advanced Physiology And Pathophysiology, 2e
Course
Advanced Physiology And Pathophysiology, 2e











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Written for

Institution
Advanced Physiology And Pathophysiology, 2e
Course
Advanced Physiology And Pathophysiology, 2e

Document information

Uploaded on
September 20, 2024
Number of pages
138
Written in
2024/2025
Type
Exam (elaborations)
Contains
Questions & answers

Subjects

  • isbn 9780826167552
  • advance

Content preview

,AdvancedggjPhysiology ggjandggjPathophysiology ggjEssentials ggjfor ggj ggClinical
ggjPracticegj1st ggjEdition ggjTkacs ggjTest ggjBank ggj-Questions ggj ggjwith ggjAnswerjKey

ggjAt ggjthe ggjend ggjof ggjEveryggjChapter,ggjAll ggjUpdated




Chapter gg1. ggTHE gg FOUNDATIONAL gg CONCEPTS gg OF
gg CLINICALgPRACTICE




1. The g g characteristic, g g localized g g cardinal g g signs g g of g g acute
g g inflammation g g include:

A) fever.
B) fatigue.
C) redness.
D) granuloma.


2. The g g vascular, g g hemodynamic g g stage g g of g g acute g g inflammation
g g is g g initiated g g byggmomentary ggvasoconstriction ggfollowed ggby
ggvasodilation ggthat ggcauses gglocalized:

A) bleeding.
B) congestion.

C) pale g g skin.
D) coolness.


3. The g g cellular g g stage g g of g g acute g g inflammation g g is g g marked g g by
g g the g g movement g g ofggleukocytes gginto ggthe ggarea. ggWhich ggof ggthe
ggfollowing ggcells ggarrives g g early ggin gggreat g g numbers?

A) Basophils
B) Lymphocytes
C) Neutrophils
D) Platelets

,4. The g g phagocytosis g g process g g involves g g three g g distinct g g steps.
g g What g g is g g thegginitial g g step g g in g g the g g process?

A) Engulfment
B) Intracellular g g killing

C) Antigen g g margination

D) Recognition g g and g g adherence




5. Which g g of g g the g g following g g mediators g g of g g inflammation
g g causes g g increasedggcapillary g g permeability g g and g g pain?

A) Serotonin
B) Histamine

C) Bradykinin
D) Nitric g g oxide



6. Inflammatory g g exudates g g are g g a g g combination g g of g g several
g g types. g g Which g g ofggthe g g following g g exudates g g is g g composed
g g of g g enmeshed g g necrotic g g cells?

A) Serous
B) Fibrinous

C) Suppurative
D) Membranous


7. The g g acute-phase g g systemic g g response g g usually g g begins g g within
g g hours g g of g g theggonset g g of g g inflammation g g and g g includes:

A) fever g g and g g lethargy.

B) decreased g g C-reactive g g protein.

C) positive g g nitrogen g g balance.

D) low g g erythrocyte g g sedimentation g g rate.




8. In g g contrast g g to g g acute g g inflammation, g g chronic g g inflammation
g g is g g characterizedggby g g which g g of g g the g g following g g phenomena?

, A) Profuse g g fibrinous g g exudation

B) A g g shift g g to g g the g g left g g of g g granulocytes

C) Metabolic g g and g g respiratory g g alkalosis

D) Lymphocytosis gg and g g activated g g macrophages


9. Exogenous g g pyrogens g g (interleukin-1) g g and g g the g g presence g g of
g g bacteria g g in g g theggblood g g lead g g to g g the g g release g g of
g g endogenous g g pyrogens g g that:

A) stabilize g g thermal g g control g g in g g the g g brain.

B) produce g g leukocytosis g g and gg anorexia.
C) block g g viral g g replication g g in g g cells.

D) inhibit g g prostaglandin g g release.



10 An g g older g g adult g g patient g g has g g just g g sheared g g the g g skin
g g on g g her g g elbow g g whileggattempted
. to ggboost ggherself ggup ggin ggbed, ggan ggevent ggthat gghas
ggprecipitated ggacute gginflammation ggin ggthe ggregion ggsurrounding
ggthe ggwound. ggWhich ggof ggthe ggfollowing g g events g g will g g occur
g g during g g the g g vascular g g stage g g of g g the g g patients
inflammation?
A) Outpouring g g of g g exudate g g into g g interstitial g g spaces

B) Chemotaxis

C) Accumulation g g of g g leukocytes g g along g g the g g epithelium

D) Phagocytosis g g of g g cellular g g debris




11 Which g g of g g the g g following g g individuals g g most g g likely g g has g g the
g g highest g g risk g g of
. experiencing g g chronic g g inflammation?

A) A g g patient gg who g g has g g recently g g been g g diagnosed g g with g g type
gg2 g g diabetes

B) A g g patient g g who g g is g g a g g carrier g g of g g an g g antibiotic-resistant
g g organism

C) A g g patient g g who g g is g g taking g g oral g g antibiotics g g for g g an g g upper
g g respiratory g g infection

D) A g g patient g g who g g is g g morbidly g g obese g g and g g who g g has g g a
g g sedentary g g lifestyle

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