Summary Lifestyle Health and Risk

waver Carbohydrates Lipids
a glucose a
glucose
CH - OH
Aft-di-polar
CH - OH CH - OH CH - OH
I condensation
molecule I I I




eY
-

Oi
o
onYL
H -



Lipids
-

ins
-
H
HH Galy acid

Y Ybit
:


I
jo
in
- I I
most
bit
H
Hi
↳

H H
gr
OH



hydrogenDandeene E
I
I
-
I I
soluble
ji

·
↑ it hydrolysis it ji ↑ it ji fary acid
I ↓ Re
Hf
+
monosaccharide 1
glycoside
e
-
H2o removed
, -




-

acid
disaccharide falty H
Properties 2 (1 4)
a
glucose maltose
hydrophobic
=
x ,



B
glucose galactore
lactose
DI saccharides
dipolar
+ =




-
Cohesive because they are
a glucose
+ tructure :
sucrose

surface cohesion flow
good for transport Formation of
triglyceride
-




lots of glucose (1 4)
a ,
=

amylose
-Solvent good at
transporting around the
body glucose (1 AND 6)
4 1 lots of side branches amylopectin
IPOLY
H ① H
d
a
saccharides
+
=
, ,




glycogen (1 , 4 AND 1 6) + LOTS of side branches :


glycogen I
I
HO R
,
- -




H -

C -


OH H C -


0
mass transport system STARCH mixture of amylose +
amylopectin ②
=




11
-compact :


good for
storage H-C OH HO-C R H C 0
-
-
- -




side branches
glucose
released quickly
- :



-

raw materials
exchange organs from
Carry -

insoluble :

doesn't let water in
good for storage
H C OH
O ③
lungs/digestive system
-



to
-




e
g. .
body cells , main energy fore in
plants H0-C
19
-

R
H -

c -

0

-Remove metabolic waste e
g CO2 i H
↓
.



GLYCOGEN
.


a
glucose (1 , 4 and 6) IOB of side branches
fatty acids
=


1 +
,



In mammals mass transport system =
-
side branches quick energy release
:




glycerol
H20 released triglycer
,
-compact storage
=




circulatory system .
-

insoluble : doesn't cause cels to well by osmosis
large molecule lots of energy sto red
:

.




main crore in animals
aort a energy
ven
cuperior
car a -- ↑
↓ E pulmonary
ARTERIES
LIFESTYLE HEALTH AND RISK
↓
↑W artery
I - ·
-




W
left atrium
-




I
↓/~
j
↳ Risk factors
R thick muscular walls of CVD

-
- endothelium
Damage inflammatory response
-

to
:



↳ -[
-
pulmonary Memmm u m

folded endothelium (expands
↓
veins White blood cells lipids clump together
-




- +
Diet (high saturated fat
-
-


/
7 j2
right ~
under pressure) Time =

more build up =

plaque= atheroma =cholesterol
:
at heromal
small lumen
-


atrium
L Plaque partially blocks lumen :
restricted blood How -High
I
blood pressure
endothelium)
VEINS :
high blood pressure . damage to

(carbon monoxide
Smoking
-


left
Hardening of arteries atherosclerosis
:


- I
right thin oxygen in blood/tissu
umm reduces
ventricle muscular walls
rentricle
-values (backflow) : heart attack/stroke Nicot
B lood clot formation
.




makes platelets sticky bloo
11
=



RHS deoxygenated lumen
large
=
-
numm e
clot Also reduces antioxidan
prothrombin
.



Dr
inferior LHS
=




oxygenated =celldamage
=

Atheroma)
CAPILLARIES
ven a cara -
Genetics

aorta
one cell thich (diffusion) - THROmOPI e
-Age (plaque builds up over

Librinogen


-
/Y
- -Gender (men



i ↑ ~↑
likely
-I↑
metabolic
W exchange
L more
- :




-


- =
&
pulmonary- bestrogen levels
=




R
-
arvery ↳ semi-lunar THROMBIN I
good' cholesterol)
values




I I-
e e
perior
W - pulmonary atria ventricles
-

L
vena cara




"
I arvery Librin
L
Treatment of CVD
&

->
W
W ⑪ ② ③ + platelets #

a S red
rea blood
-1
I ~
CeIIS
cellS -Antihypertensives= reduc

= +