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Summary Alevel OCR Biology A notes on Communicable Diseases

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Alevel OCR Biology A notes on Communicable Diseases from an A* biology student. Plus diagrams.

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· endemic disease always present

·
epidemic:l arge 4 void
in cases


large
·
pandemic:e pidemic becomes
scale international.
+




communicable Diseases
-Infections diseases caused -




by pathogens, transmissible (malria, HIV/AIDS, TB)

diseases degenerative diseases lung
-




non-infections -
long term cancer, cystic fibrosis)

-



Bacteria prohcryotes,
=




mostly pathoogenic

>
M. Tuberculosis: human TB, N. Meningitidis:h uman bacterial meningitis, Potato ring nor




Viruses
-




cellular structure, infect lose cells genetic material
replicate proteins.
=


no
-
+





Tobacco virus= yellow plane leaves, Incluenza vins= human the, HIV= altech human immune
mosaic
system
-




Protoctista-
m icellular encryctes



Plasmodium falciparum hman malaria,
=
P.mestans potato
=



blight

Frugi= euhcryotic cells (all wall +
large central vacuoles, hyphae)


Cattle ringuorm athletes loor=
+

sin surface, Black Banana lingal disease
Sigaroha=

-Direct physical
transmission: compact, close
proximity, spares


-Indirect transmission vec tor
=


(insects, reproduce large
in numbers)


of pathogens susceptible individuals,
-




Transmission depends on presence + sale animals are reinstant




[

factors affecting -high population dense areas injection rates
=




direct trans-
mission disease
Carring
-



monocultures large albreak
-




factor affecting

indirect transmission E biology of rectors weather
+ +

climate:population of rection



Gemale mosquitoes favor high rainfall, temps
+

humidity:m alcria more common
tropics
in subtropics
+





4 iod malaria epidemics due to
climatic + environmental changes

nuce of poverlytransmission of wman diseases (thygeire), human
movement-migration:population 4 connected


-Passive defence mechanisms:
physical barrier (waxy article, allose all wall, closed stomara, ban, semparian strip),

chemical defences:
pathogens cant grow on
plans space due acidid
to condition



(Toxins, stilly reim in back, enzyme inhibitor=
tannins, receptor molecules, compands enavaging

of competing microorganism).
growth

-




Active defence mechanisms -


cell all
walls:
signalling vital, hypersensitivity:
rapid death of tissue around infection site,


physical barrier=
reinforced cell wall (callose +
liguins, plasmodesmata narrowing, ingrowth

into xylemn ussels, phloem blockage.

-cell signalling (digest altoloses signals phytodexis
cellulase enzymes resulting molecules to all surface receptors:release
-
-
=




->long
term.

=salicylic acid= t o winfected
migrates creas: activate defence mechanisms:
systematic acquired resistance



=ethylene =
plant communication, ethylene vaporises: other leaves on same plan stimulated:recretion

, Humans




t
Pathogen
Method of Transmission
Type




Bacterium




virus




Indirect lemale mosquitos
Protoctist
-




Direct-contact injected
with cattle
Cattle
ringworm

Lingus items
Athlete's Direct-conserer with touched
by insected

assumeebrum
lost
+




individuals.
I




plants


Pathogen Disease method of
Host Pathogen transmission
Type caused


C. midiganese Direct-contact w/ other
Bacterium ring not Potato, tomato
infected tubers.



Direct-connect w/hal
ViWS TobaccoMosaic Tobacco Tobacco mosaic

virus
of insected plants
viws (TMV)
Indirect-vector (oplich)
-




Bananas M. Gijaneis Direct -


spares
Black Sigatoha
Cunque
-




Direct -


Spores
Protoctist late
blight potato, tomato P. infestans

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