Describe smooth -Not striated
-Spindle shaped muscle cells each
(involuntary) muscle containing a nucleus (uninucleate)
-Peristalsis, dilation/constriction of
(striated, structure, pupils, vasoconstriction/dilation
functions + -Innervated by autonomic NS
-Contraction is slow but muscle
additional info.): (x5) slow to tire
Describe cardiac -Striated
-Muscle cells form fibres with several
(involuntary) muscle nuclei (multinucleate)
-Sarcolemmas fuse to form intercalated
(striated, structure, disks
-Responsible for heartbeat (myogenic)
functions + -Innervated by autonomic NS
-Contracts continuously + powerfully
additional info.): (x6) without fatigue
-Striated
Describe skeletal -Muscle cells form fibres with
(voluntary) muscle several nuclei (multinucleate)
-Movement of skeleton at joints
(striated, structure, (function in antagonistic pairs)
functions + -Innervated by somatic NS
-Contracts quickly + powerfully
additional info.): (x5) but fatigues quickly
-Sarcolemma (plasma
What are the membrane)
specific organelles -Sarcoplasm
found only in (cytoplasm)
muscle cells? (x3) -Sarcoplasmic reticulum
(specialised ER)
, Go through the -AP arrives down motor neurone = causes polarisation =
voltage-gated Ca2+ channels open = Ca2+ enters
presynaptic knob = cause vesicles of ACh to fuse with
events at a
presynaptic membrane
-ACh released via exocytosis + diffuses across synaptic
cleft = bind to receptors on sarcolemma = open Na+
channels = Na+ enter cell = depolarises membrane +
neuromuscular initiates AP
-Depolarisation of sarcolemma spreads down T-tubules
(allow AP through centre of fibre)
-Ca2+ channels open + Ca2+ diffuses out of SR = sliding
junction: (x5) filament theory
-AChE breaks down ACh = recycled
-Motor end plate = where
What is a motor neurone ends + joins
muscle
end plate + a -Motor unit = motor
motor unit? neurone + all the muscle
fibres it connects to
Electro
How can -Pancuronium bromide = competes with
ACh for receptors on sarcolemma =
chemicals block binds to + blocks them = sarcolemma
doesn't depolarise = no muscular
contraction
contraction of a -Action can be reversed = inhibit AChE =
conc. ACh increases = out-competes
muscle? (example) Pancuronium bromide
-Spindle shaped muscle cells each
(involuntary) muscle containing a nucleus (uninucleate)
-Peristalsis, dilation/constriction of
(striated, structure, pupils, vasoconstriction/dilation
functions + -Innervated by autonomic NS
-Contraction is slow but muscle
additional info.): (x5) slow to tire
Describe cardiac -Striated
-Muscle cells form fibres with several
(involuntary) muscle nuclei (multinucleate)
-Sarcolemmas fuse to form intercalated
(striated, structure, disks
-Responsible for heartbeat (myogenic)
functions + -Innervated by autonomic NS
-Contracts continuously + powerfully
additional info.): (x6) without fatigue
-Striated
Describe skeletal -Muscle cells form fibres with
(voluntary) muscle several nuclei (multinucleate)
-Movement of skeleton at joints
(striated, structure, (function in antagonistic pairs)
functions + -Innervated by somatic NS
-Contracts quickly + powerfully
additional info.): (x5) but fatigues quickly
-Sarcolemma (plasma
What are the membrane)
specific organelles -Sarcoplasm
found only in (cytoplasm)
muscle cells? (x3) -Sarcoplasmic reticulum
(specialised ER)
, Go through the -AP arrives down motor neurone = causes polarisation =
voltage-gated Ca2+ channels open = Ca2+ enters
presynaptic knob = cause vesicles of ACh to fuse with
events at a
presynaptic membrane
-ACh released via exocytosis + diffuses across synaptic
cleft = bind to receptors on sarcolemma = open Na+
channels = Na+ enter cell = depolarises membrane +
neuromuscular initiates AP
-Depolarisation of sarcolemma spreads down T-tubules
(allow AP through centre of fibre)
-Ca2+ channels open + Ca2+ diffuses out of SR = sliding
junction: (x5) filament theory
-AChE breaks down ACh = recycled
-Motor end plate = where
What is a motor neurone ends + joins
muscle
end plate + a -Motor unit = motor
motor unit? neurone + all the muscle
fibres it connects to
Electro
How can -Pancuronium bromide = competes with
ACh for receptors on sarcolemma =
chemicals block binds to + blocks them = sarcolemma
doesn't depolarise = no muscular
contraction
contraction of a -Action can be reversed = inhibit AChE =
conc. ACh increases = out-competes
muscle? (example) Pancuronium bromide
