NURS 5315 ENDOCRINE EXAM
QUESTIONS AND ANSWERS 2026
- 2027
Thyroid hormones - ANSWERS-T3, T4 and calcitonin
Pancreas hormones - ANSWERS-Insulin and glucagon
Adrenal Glands hormones - ANSWERS-Made up of the cortex which
secretes steroids such as cortisone and aldosterone and the medulla
which secretes catecholamines such as epi and norepi
Primary thyroid disorders - ANSWERS-result in alterations of thyroid
hormone (TH) levels with secondary feedback effects on pituitary
thyroid stimulating hormone (TSH)
Subclinical Thyroid disease - ANSWERS-Thyroid disease that
presents with minimal to no symptoms but with abnormal lab values
Secondary Thyroid disorders - ANSWERS-conditions that results
from the dysfunction of the pituitary gland TSH production
Thyrotoxicosis - ANSWERS-(hyperthyroidism) a condition that
results from any cause of increased TH levels. Will how low TSH
levels and high T4 level. S&S: increased metabolic rate, heat
, intolerance, goiter, menstrual irrregularities, weight loss, diaphoresis,
fine tremor, tachycardia, frequent bowel movements, restlessness,
short attention span, hair loss, anorexia, exophthalmos, pretibial
edema, and heart failure.
Thyrotoxic Crisis(thyroid storm) - ANSWERS-worsening
hyperthyroid state triggered by an igniting even such as infection,
trauma, cardiopulmonary disorder, burns, seizures surgery, or
spontaneously. S&S: extreme restlessness and agitation, delirium,
seizures, coma, severe tachycardia, heart failure, hyperthermia,
delirium, volume depletion, NVD and death if not treated.
Grave's disease - ANSWERS-Most common cause of
hyperthyroidism and is an autoimmune disorder. Antibodies attach to
the thyroid cells and mimic the function of TSH which results in an
increased secretion of T3 and T4 and overrides the negative feedback
mechanisms which regulate TSH secretion. The stimulation of the
receptors by the antibodies results in the development of goiter. May
also experience exophthalmos, periorbital edema, and extraocular
muscle weakness leading to strabismus and diplopia
Hyperthyroidism from nodular thyroid disease - ANSWERS-
Follicular hypertrophy of the thyroid cells is responsible for the
formation of the thyroid nodules which secrete extra hormones.
Nodules develop bc of normal changes during pregnancy or puberty
or as a result of an autoimmune issue, viral infection or genetic
influence. Symptoms develop slowly and will not display
exophthalmos or pretibial myxedema
QUESTIONS AND ANSWERS 2026
- 2027
Thyroid hormones - ANSWERS-T3, T4 and calcitonin
Pancreas hormones - ANSWERS-Insulin and glucagon
Adrenal Glands hormones - ANSWERS-Made up of the cortex which
secretes steroids such as cortisone and aldosterone and the medulla
which secretes catecholamines such as epi and norepi
Primary thyroid disorders - ANSWERS-result in alterations of thyroid
hormone (TH) levels with secondary feedback effects on pituitary
thyroid stimulating hormone (TSH)
Subclinical Thyroid disease - ANSWERS-Thyroid disease that
presents with minimal to no symptoms but with abnormal lab values
Secondary Thyroid disorders - ANSWERS-conditions that results
from the dysfunction of the pituitary gland TSH production
Thyrotoxicosis - ANSWERS-(hyperthyroidism) a condition that
results from any cause of increased TH levels. Will how low TSH
levels and high T4 level. S&S: increased metabolic rate, heat
, intolerance, goiter, menstrual irrregularities, weight loss, diaphoresis,
fine tremor, tachycardia, frequent bowel movements, restlessness,
short attention span, hair loss, anorexia, exophthalmos, pretibial
edema, and heart failure.
Thyrotoxic Crisis(thyroid storm) - ANSWERS-worsening
hyperthyroid state triggered by an igniting even such as infection,
trauma, cardiopulmonary disorder, burns, seizures surgery, or
spontaneously. S&S: extreme restlessness and agitation, delirium,
seizures, coma, severe tachycardia, heart failure, hyperthermia,
delirium, volume depletion, NVD and death if not treated.
Grave's disease - ANSWERS-Most common cause of
hyperthyroidism and is an autoimmune disorder. Antibodies attach to
the thyroid cells and mimic the function of TSH which results in an
increased secretion of T3 and T4 and overrides the negative feedback
mechanisms which regulate TSH secretion. The stimulation of the
receptors by the antibodies results in the development of goiter. May
also experience exophthalmos, periorbital edema, and extraocular
muscle weakness leading to strabismus and diplopia
Hyperthyroidism from nodular thyroid disease - ANSWERS-
Follicular hypertrophy of the thyroid cells is responsible for the
formation of the thyroid nodules which secrete extra hormones.
Nodules develop bc of normal changes during pregnancy or puberty
or as a result of an autoimmune issue, viral infection or genetic
influence. Symptoms develop slowly and will not display
exophthalmos or pretibial myxedema
