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NURS 5315 EXAM 5 (UTA) NEWEST 2026 ACTUAL EXAM TEST BANK| NURS5315 ADVANCED PATHOPHYSIOLOGY EXAM 5 REVIEW WITH COMPLETE REAL EXAM QUESTIONS AND CORRECT VERIFIED ANSWERS/ ALREADY GRADED A+

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NURS 5315 EXAM 5 (UTA) NEWEST 2026 ACTUAL EXAM TEST BANK| NURS5315 ADVANCED PATHOPHYSIOLOGY EXAM 5 REVIEW WITH COMPLETE REAL EXAM QUESTIONS AND CORRECT VERIFIED ANSWERS/ ALREADY GRADED A+

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NURS 5315 EXAM 5 (UTA) NEWEST 2026 ACTUAL EXAM
TEST BANK| NURS5315 ADVANCED PATHOPHYSIOLOGY
EXAM 5 REVIEW WITH COMPLETE REAL EXAM
QUESTIONS AND CORRECT VERIFIED ANSWERS/
ALREADY GRADED A+
Exam Overview
NURS 5315 – Advanced Pathophysiology at the University of Texas at Arlington
(UTA) builds on prior knowledge of anatomy and physiology to develop advanced
understanding of physiologic and pathophysiologic concepts across the
lifespan . Exam 5 focuses on clinical concepts related to the Renal, Reproductive,
and Gastrointestinal (GI) systems, as well as other systems covered in the latter
portion of the course .
Exam Details at a Glance:
Aspect Detail
Course NURS 5315 Advanced Pathophysiology
Institution UT Arlington (UTA)
Exam 5
Focus Areas Renal, Reproductive, GI, Hematologic, Cardiovascular,
Respiratory, Endocrine
Format Multiple-choice, NCLEX-style questions with rationales
Recent 202/2027 academic year
Update


Core Content Areas Covered (Exam 5)
1. Renal & Reproductive Systems
 Renal physiology and pathophysiology (nephron components: Glomerulus,
Proximal Tubule, Loop of Henle, Distal Tubule)
 Electrolyte imbalances and action potential alterations

,  Fluid volume disorders
2. Gastrointestinal System
 GI pathophysiology (hepatoportal circulation, osmotic diarrhea, pancreatic
disorders)
 Hepatic and biliary disorders
3. Hematologic & Cardiovascular Systems
 Anemias (macrocytic, microcytic, pernicious, folate deficiency)
 Sickle cell disease
 Immunity types
 Valvular heart diseases
4. Endocrine & Neurologic Systems
 Adrenal disorders (Addison disease, Cushing syndrome)
 Thyroid disorders
 Neurologic conditions
5. Cellular Adaptation & Injury
 Reactive Oxygen Species (ROS) and antioxidants
 Action potentials and electrolyte effects
 Cellular adaptations (atrophy, hypertrophy, hyperplasia, dysplasia,
metaplasia)


: Renal & Electrolyte Pathophysiology
1. A patient diagnosed with syndrome of inappropriate antidiuretic hormone
secretion (SIADH) is at highest risk for which electrolyte imbalance?

 A) Hypernatremia

 B) Hyperkalemia

,  C) Hyponatremia

 D) Hypercalcemia

Rationale: Excess ADH causes water retention, diluting serum sodium
concentrations and leading to hyponatremia. This is a hallmark finding in SIADH .



2. Which laboratory finding is most characteristic of iron deficiency anemia
secondary to chronic gastrointestinal bleeding?

 A) Elevated ferritin and macrocytosis

 B) Low hemoglobin with microcytic hypochromic cells

 C) Increased vitamin B12 and reticulocyte count

 D) Elevated hematocrit with normocytic erythrocytes

Rationale: Iron deficiency anemia produces small, pale red blood cells (microcytic,
hypochromic) due to impaired hemoglobin synthesis .



3. A patient with Addison disease would most likely exhibit which laboratory
finding?

 A) Hypernatremia and hypokalemia

 B) Hyponatremia and hyperkalemia

 C) Elevated cortisol levels

 D) Increased aldosterone secretion

, Rationale: Adrenal insufficiency causes decreased aldosterone, leading to sodium
loss and potassium retention. This results in hyponatremia and hyperkalemia .



4. Hypokalemia affects action potentials by which mechanism?

 A) Hypopolarization (more positive), making cells more excitable

 B) Hyperpolarization (more negative), making cells less excitable

 C) No effect on resting membrane potential

 D) Decreasing permeability to sodium

Rationale: Hypokalemia causes potassium to leave the cell, leaving behind
negative anions. The cell hyperpolarizes (becomes more negative) and is therefore
less excitable. Clinical consequences include weakness, smooth muscle atony,
paresthesia, and cardiac dysrhythmias .



5. Hyperkalemia affects action potentials by which mechanism?

 A) Hypopolarization (more positive), making cells more excitable

 B) Hyperpolarization (more negative), making cells less excitable

 C) Decreasing permeability to sodium

 D) No effect on resting membrane potential

Rationale: Increased potassium enters the cell, making it more positive.
Depolarization occurs, taking the resting membrane potential closer to threshold

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