NSG 3280 Exam 2 Questions and Answers
(2026/2027) | Review Pack | Real Exam
Practice | A+
• Epidermis characteristics. CORRECT ANSWER: Avascular outer skin
layer that produces keratin and contains melanocytes.
• Dermis characteristics. CORRECT ANSWER: Vascular skin layer
containing sebaceous/oil glands plus collagen and elastin.
• Subcutaneous tissue characteristics. CORRECT ANSWER: Deep layer
made of fat and connective tissue; provides energy storage.
• Aging skin: cell turnover. CORRECT ANSWER: Cell turnover decreases
with age, increasing infection risk and slowing healing.
• Aging skin: thickness vs sun exposure. CORRECT ANSWER: Advanced
age thins skin; chronic sun/UV exposure thickens skin through DNA
damage.
• Aging skin: vascularity. CORRECT ANSWER: Vascularity decreases with
age, so skin receives less oxygen from the blood.
• Aging skin: strength and elasticity. CORRECT ANSWER: Skin strength
and elasticity decrease with age.
• Aging skin: vascular fragility. CORRECT ANSWER: Vascular fragility
increases with age, increasing bruising and injury risk.
• Aging skin: melanin. CORRECT ANSWER: Melanin production decreases
with age, contributing to white/gray hair.
• Aging skin: sebum. CORRECT ANSWER: Sebum/oil production
decreases with age, causing dry skin.
• Aging skin: sweating. CORRECT ANSWER: Sweat production decreases
with age, impairing thermoregulation.
, • Primary skin lesions. CORRECT ANSWER: Lesions as they originally
appear on intact skin; remember primary = problem begins/pops up first.
• Secondary skin lesions. CORRECT ANSWER: Lesions modified from the
original appearance by scratching, spreading, scarring, breaking, infection,
or healing.
• Verrucae/warts. CORRECT ANSWER: Benign viral growths caused by
papillomavirus; may resolve spontaneously.
• HSV general concept. CORRECT ANSWER: Herpes simplex virus is
recurrent, spreads by direct contact or mucous membranes/sex, and lives
latent in nerves.
• HSV reactivation triggers. CORRECT ANSWER: Stress, decreased
immunity, menstruation, and sun/UV exposure can reactivate HSV.
• HSV prodrome. CORRECT ANSWER: Burning or tingling sensation 24-48
hours before lesions appear; occurs because latent virus reactivates in
nerves and irritates them.
• HSV lesion progression. CORRECT ANSWER: Burning/tingling and
redness progress to vesicles, then pustules, ulcers, and crusting; healing
may take about 10-14 days.
• HSV-1 usual location. CORRECT ANSWER: Usually above the waist:
lips, face, and mouth.
• HSV-1 nerve location. CORRECT ANSWER: Typically resides in the
trigeminal nerve/ganglion of the face.
• HSV-2 usual location. CORRECT ANSWER: Usually below the waist and
responsible for genital infections.
• HSV-2 nerve location. CORRECT ANSWER: Often resides in the sacral
nerve ganglia; lesions erupt in the genital region.
• HSV blood test. CORRECT ANSWER: Detects HSV exposure; shows
past exposure and does not always mean active infection.
(2026/2027) | Review Pack | Real Exam
Practice | A+
• Epidermis characteristics. CORRECT ANSWER: Avascular outer skin
layer that produces keratin and contains melanocytes.
• Dermis characteristics. CORRECT ANSWER: Vascular skin layer
containing sebaceous/oil glands plus collagen and elastin.
• Subcutaneous tissue characteristics. CORRECT ANSWER: Deep layer
made of fat and connective tissue; provides energy storage.
• Aging skin: cell turnover. CORRECT ANSWER: Cell turnover decreases
with age, increasing infection risk and slowing healing.
• Aging skin: thickness vs sun exposure. CORRECT ANSWER: Advanced
age thins skin; chronic sun/UV exposure thickens skin through DNA
damage.
• Aging skin: vascularity. CORRECT ANSWER: Vascularity decreases with
age, so skin receives less oxygen from the blood.
• Aging skin: strength and elasticity. CORRECT ANSWER: Skin strength
and elasticity decrease with age.
• Aging skin: vascular fragility. CORRECT ANSWER: Vascular fragility
increases with age, increasing bruising and injury risk.
• Aging skin: melanin. CORRECT ANSWER: Melanin production decreases
with age, contributing to white/gray hair.
• Aging skin: sebum. CORRECT ANSWER: Sebum/oil production
decreases with age, causing dry skin.
• Aging skin: sweating. CORRECT ANSWER: Sweat production decreases
with age, impairing thermoregulation.
, • Primary skin lesions. CORRECT ANSWER: Lesions as they originally
appear on intact skin; remember primary = problem begins/pops up first.
• Secondary skin lesions. CORRECT ANSWER: Lesions modified from the
original appearance by scratching, spreading, scarring, breaking, infection,
or healing.
• Verrucae/warts. CORRECT ANSWER: Benign viral growths caused by
papillomavirus; may resolve spontaneously.
• HSV general concept. CORRECT ANSWER: Herpes simplex virus is
recurrent, spreads by direct contact or mucous membranes/sex, and lives
latent in nerves.
• HSV reactivation triggers. CORRECT ANSWER: Stress, decreased
immunity, menstruation, and sun/UV exposure can reactivate HSV.
• HSV prodrome. CORRECT ANSWER: Burning or tingling sensation 24-48
hours before lesions appear; occurs because latent virus reactivates in
nerves and irritates them.
• HSV lesion progression. CORRECT ANSWER: Burning/tingling and
redness progress to vesicles, then pustules, ulcers, and crusting; healing
may take about 10-14 days.
• HSV-1 usual location. CORRECT ANSWER: Usually above the waist:
lips, face, and mouth.
• HSV-1 nerve location. CORRECT ANSWER: Typically resides in the
trigeminal nerve/ganglion of the face.
• HSV-2 usual location. CORRECT ANSWER: Usually below the waist and
responsible for genital infections.
• HSV-2 nerve location. CORRECT ANSWER: Often resides in the sacral
nerve ganglia; lesions erupt in the genital region.
• HSV blood test. CORRECT ANSWER: Detects HSV exposure; shows
past exposure and does not always mean active infection.
