Galen College of Nursing-Louisville NSG 3850
NSG 3850 exam 4
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Patho Exam 4 review questions Exam 4 NSG 3850 LATEST VERSION... Exam 4 NSG 3850 LATEST VERSION... NSG 38
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Cholelithiasis Supersaturation of bile w/ cholesterol causing nucleation of crystals and
hypomotility allowing stone growth
♦ Nucleation occurs where the cholesterol crystals aggregate together.
Continued growth of the crystals then depends on the balance between
cholesterol growth
♦ What would you see in your patient with this?
• Intermittent biliary colic, persistent epigastric or RU abdominal pain
• N/V, sweating, flatus, pain radiates to the back
• May be precipitated by a meal, may manifest at night
♦ What would you teach your patient about what this is?
• Need to worry about bile and cholesterol
• No fatty foods, avoid alcohol,
• Somatostatin---esophgeal varices -do not give
• Not going to have somatostatin
Cholecystitis Acute inflammation of the gallbladder wall—obstruction of cystic duct=statis of
bile in gallbladder
♦ What are the risk factors?
• Diabetes and obesity
♦ What would you see in your patient with this?
• Severe right upper abdominal pain that radiates to the back, abdominal
tenderness, fever
♦ What testing should you anticipate?
• Leukocytosis, mild elevations in bilirubin,
• Ultrasound, CT, ERCP, no biopsy
• Increased risk of cancer
• Huge risk for sepsis
, Hepatitis A enteric hepatitis -RNA virus
♦ Fecal-oral route—lack of sanitation and safe water
♦ 2-7-week incubation period
♦ Malaise, anorexia, nausea, low grade fever, RUQ pain, with or without jaundice
♦ Anti-HAV IgM =acute infection
♦ Anti-HAV IgG= previous infection or vaccination
• immunity
♦ Supportive management—avoid alcohol and NSAIDs
• Vaccination
Hepatitis B partially double stranded DNA virus
♦ Parenteral contact—contaminated blood, needles, sexual contact
♦ 2-6 month incubation period or 15-45 days
♦ Urticaria (hives), rashes, arthralgia, arthritis, angioedema, serum sickness,
glomerulonephritis
♦ HBsAg—implies active infection
♦ Ongoing liver inflammation and active viral replication
♦ Supportive—liver transplant only definitive treatment
♦ Vaccination
Hepatitis C single stranded RNA virus
♦ IV drug use or blood transfusions
♦ Asymptomatic, mild
♦ Assessment findings: Edema, altered mental status, jaundice
♦ Have 70% increased risk for diabetes
♦ Supportive care
♦ Anti-HCV positive or IgG—antibody to HCV antigens
Hepatitis D ♦ Requires Hep B virus for replication
♦ Parenteral routes and intimate contact
♦ Accelerates the progress of liver disease
Hepatitis E RNA virus
♦ Spread via fecal-oral route---contaminated water
♦ 2-9 week incubation period
Alcoholic hepatitis Active inflammation of the centrilobular region of the liver—liver cells show
pathologic changes of hepatocyte necrosis with neutrophilic infiltration and
intracellular inclusions known as Mallory bodies
♦ Mallory Bodies on the liver
♦ How would you explain this?
NSG 3850 exam 4
Leave the first rating
Save
Students also studied
Flashcard sets Study guides
Patho Exam 4 review questions Exam 4 NSG 3850 LATEST VERSION... Exam 4 NSG 3850 LATEST VERSION... NSG 38
50 terms Teacher 54 terms Teacher 54 terms 89 terms
keshia28 Preview fabycalb Preview Mercy_Mutinda4 Preview see
Terms in this set (34) Hide definitions
Cholelithiasis Supersaturation of bile w/ cholesterol causing nucleation of crystals and
hypomotility allowing stone growth
♦ Nucleation occurs where the cholesterol crystals aggregate together.
Continued growth of the crystals then depends on the balance between
cholesterol growth
♦ What would you see in your patient with this?
• Intermittent biliary colic, persistent epigastric or RU abdominal pain
• N/V, sweating, flatus, pain radiates to the back
• May be precipitated by a meal, may manifest at night
♦ What would you teach your patient about what this is?
• Need to worry about bile and cholesterol
• No fatty foods, avoid alcohol,
• Somatostatin---esophgeal varices -do not give
• Not going to have somatostatin
Cholecystitis Acute inflammation of the gallbladder wall—obstruction of cystic duct=statis of
bile in gallbladder
♦ What are the risk factors?
• Diabetes and obesity
♦ What would you see in your patient with this?
• Severe right upper abdominal pain that radiates to the back, abdominal
tenderness, fever
♦ What testing should you anticipate?
• Leukocytosis, mild elevations in bilirubin,
• Ultrasound, CT, ERCP, no biopsy
• Increased risk of cancer
• Huge risk for sepsis
, Hepatitis A enteric hepatitis -RNA virus
♦ Fecal-oral route—lack of sanitation and safe water
♦ 2-7-week incubation period
♦ Malaise, anorexia, nausea, low grade fever, RUQ pain, with or without jaundice
♦ Anti-HAV IgM =acute infection
♦ Anti-HAV IgG= previous infection or vaccination
• immunity
♦ Supportive management—avoid alcohol and NSAIDs
• Vaccination
Hepatitis B partially double stranded DNA virus
♦ Parenteral contact—contaminated blood, needles, sexual contact
♦ 2-6 month incubation period or 15-45 days
♦ Urticaria (hives), rashes, arthralgia, arthritis, angioedema, serum sickness,
glomerulonephritis
♦ HBsAg—implies active infection
♦ Ongoing liver inflammation and active viral replication
♦ Supportive—liver transplant only definitive treatment
♦ Vaccination
Hepatitis C single stranded RNA virus
♦ IV drug use or blood transfusions
♦ Asymptomatic, mild
♦ Assessment findings: Edema, altered mental status, jaundice
♦ Have 70% increased risk for diabetes
♦ Supportive care
♦ Anti-HCV positive or IgG—antibody to HCV antigens
Hepatitis D ♦ Requires Hep B virus for replication
♦ Parenteral routes and intimate contact
♦ Accelerates the progress of liver disease
Hepatitis E RNA virus
♦ Spread via fecal-oral route---contaminated water
♦ 2-9 week incubation period
Alcoholic hepatitis Active inflammation of the centrilobular region of the liver—liver cells show
pathologic changes of hepatocyte necrosis with neutrophilic infiltration and
intracellular inclusions known as Mallory bodies
♦ Mallory Bodies on the liver
♦ How would you explain this?
