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, TABLE OF CONTENT
zz zz




Chapterz z 1:z z Pharmacokinetics
Chapterzz2:zzDrug–
ReceptorzzInteractionszzandzzPharmacodynamicsz z Chapterzz3:z z Thez z AutonomiczzNervouszzSystem
Chapterzz4:zzCholinergiczzAgonistszzChapterzz5:zzCholinergiczzAntagonistszz
Chapterzz6:zzAdrenergiczzAgonistszzChapterzz7:zzAdrenergiczzAntagonists
Chapterzz8:zzDrugszzforzzNeurodegenerativezzDiseaseszzChapterzz9:zzAnxiolyticzzandzzHypnoticzz
Drugs
Chapterzz10:zzAntidepressantszzChapterzz11:zzAntipsychoticzzDrugszzChapte
rzz12:zzDrugszzforzzEpilepsyzzChapterzz13:zzAnesthetics
Chapterz z 14:z z Opioids
Chapterzz15:zzDrugszzofzzAbusezzChapterzz16:zzCNSzzStimulantszzChapterzz17
:zzAntihypertensiveszzChapterzz18:zzDiuretics
Chapterz z 19:z z Heartz z Failure
Chapterzz20:zzAntiarrhythmicszzChapterzz21:zzAntianginalzzDrugs
Chapterzz22:zzAnticoagulantszzandzzAntiplateletzzAgentsz z Chapterzz23:zzDrugszzforzzHyperlipid
emia
Chapterzz24:zzPituitaryzzandzzThyroidzzChapterzz25:zzDrugszzforzzDiabetes
Chapterzz26:zzEstrogenszzandzzAndrogenszzChapterzz27:zzAdrenalzzHormones
Chapterz z 28:z z Drugsz z forz z Obesity
Chapterzz29:zzDrugszzforzzDisorderszzofzzthezzRespiratoryzzSystemz z Chapterzz30:zzAntihistamines
Chapterzz31:zzGastrointestinalzzandzzAntiemeticzzDrugszzChapterzz32:zzDrugszzforzzUrologicz z Di
sorders
Chapterz z 33:z z Drugsz z forz z Anemia
Chapterzz34:zzDrugszzforzzDermatologiczzDisorderszzChapterzz35:zzDrugszzforzzBonezzDisorders
Chapterzz36:zzAnti-
inflammatory,zzAntipyretic,zzandz z AnalgesiczzAgentsz z Chapterzz37:z z Principleszzofz z Antimicrobialz z Therapy
Chapterz z 38:z z Cellzz Wallz z Inhibitors
Chapterz z 39:z z Proteinz z Synthesisz z Inhibitors
Chapterzz40:zzQuinolones,zzFoliczzAcidz z Antagonists,z z andz z Urinaryz z TractzzAntisepticszzChapterzz41:zzAntimycobacterialzzDrug
s
Chapterzz42:zzAntifungalzzDrugszzChapterzz43:zzAntiprotozoalzzDrugszzChapt
erzz44:zzAnthelminticzzDrugszzChapterzz45:zzAntiviralzzDrugs
Chapterz z 46:z z Anticancerz z Drugs
Chapterzz47:zzImmunosuppressantszzChapterzz48:zzClinicalzzToxicology

,
, 3. The nurse is teaching a patient who will be discharged home with a prescription for an enteric-
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coated tablet. Which statement by the patient indicates understanding of the teaching?
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a. Imay crush the tablet and put it in applesauce to improve absorption.
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b. I should consume acidic foodsto enhance absorption of this medication.
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c. I should expect a delay in onset of the drugs effects after taking the tablet.
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d. I should take this medication with high-
.: .: .: .: .: .:



fat foodsto improve its action. ANS: C
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Enteric-
coated tablets resist disintegration in the acidic environment of the stomach and disintegrate when t
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hey reach the small intestine. There is usually some delay in onset of actions after taking these medi
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cations. Enteric- .:



coated tablets should not be crushed or chewed, which would alter the time and location of absorpti
.: .: .: .: .: .: .: .: .: .: .: .: .: .: .: .:



on. Acidic foods will not enhance the absorption of the medication. The patient should not to eat hig
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h-fat food before ingesting an enteric-coated tablet, because high-
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fat foods decrease the absorption rate.
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DIF: COGNITIVE LEVEL: Applying (Application) REF: dm 3 TOP
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: NURSING PROCESS: Nursing Intervention
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MSC: NCLEX: Physiological Integrity: Pharmacological and ParenteralTherapies
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4. A patient who is newly diagnosed with type 1 diabetes mellitus asks why insulin must be given
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by subcutaneous injection instead of bymouth. The nurse willexplain that this is because
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a. absorption is diminished bythe first-pass effects in the liver. .: .: .: .: .: .: .: .: .:



b. absorption is faster when insulin is given subcutaneously. .: .: .: .: .: .: .:



c. digestive enzymes in the gastrointestinaltract prevent absorption. .: .: .: .: .: .: .:



d. the oral formis less predictable with more adverse effects. A
.: .: .: .: .: .: .: .: .: .:



NS: C .:



Insulin, growth hormones, and other protein- .: .: .: .: .:



based drugs are destroyed in the small intestine by digestive enzymes and must be given parenterall
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y. Because insulin is destroyed by digestive enzymes, it would not make it to the liver for metabolis
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m with a first-
.: .: .:



pass effect. Subcutaneous tissue has fewer blood vessels, so absorption is slower in such tissue. Ins
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ulin is given subcutaneously because it is desirable to have it absorb slowly.
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DIF: COGNITIVE LEVEL: Understanding (Comprehension) REF: dm 3 TO
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P: NURSING PROCESS: Nursing Intervention: Patient Teaching
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Pagez z 2z z ofz z 507



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