Test bank for Rubin s pathology clinicopathologic foundations of medicine 7th edition by David S. Strayer, Emanuel Rubin Course

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RUBIN'S PATHOLOGY: CLINICOPATHOLOGIC FOUNDATIONS OF MEDICIN
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E 7TH EDITION
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DAVID S. STRAYER, EMANUEL RUBIN
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,TestmBankmRubin'smPathology:mClinicopathologicmFoundationsmofmMedicinem7thmEdition

TablemofmContents:
Chapterm1:mCellmAdaptation,mInjurymandmDeath
Chapterm2:mInflammation
Chapterm3:mRepair,mRegenerationmandmFibrosis
Chapterm4:mImmunopathology
Chapterm5:mNeoplasia
Chapterm6:mDevelopmentalmandmGeneticmDiseases
Chapterm7:mHemodynamicmDisorders
Chapterm8:mEnvironmentalmandmNutritionalmPathology
Chapterm9:mInfectiousmandmParasiticmDiseases
SectionmII:mPathogenesismofmSystemicmConditionsmExpandablemsection
Chapterm10:mAging
Chapterm11:mSystemicmAutoimmunemDiseases
Chapterm12:mSepsis
Chapterm13:mObesitymandmDiabetesmMellitus
Chapterm14:mThemPathologymofmPregnancy
Chapterm15:mThemAmyloidoses
SectionmIII:mDiseasesmofmIndividualmOrganmSystemsExpandablemsection
Chapterm16:mBloodmVessels
Chapterm17:mThemHeart
Chapterm18:mThemRespiratorymSystem
Chapterm19:mThemGastrointestinalmTract
Chapterm20:mThemLivermandmBiliarymSystem
Chapterm21:mThemPancreas
Chapterm22:mThemKidney
Chapterm23:mThemLowermUrinarymTractmandmMalemReproductivemSystem
Chapterm24:mThemFemalemReproductivemSystemmandmPeritoneum
Chapterm25:mThemBreast
Chapterm26:mHematopathology
Chapterm27:mThemEndocrinemSystem
Chapterm28:mThemSkin
Chapterm29:mThemHeadmandmNeck
Chapterm30:mBones,mJointsmandmSoftmTissue
Chapterm31:mSkeletalmMusclemandmPeripheralmNervousmSystem
Chapterm32:mThemCentralmNervousmSystem
Chapterm33:mThemEye
Chapterm34:mForensicmPathology

,Rubin'smPathology:mClinicopathologicmFoundationsmofm
MedicineChapterm1:mCellmAdaptation,mInjurymandmDeat

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Ischemiamandmothermtoxicminjuriesmincreasemthemaccumulationmofmintracellularmcalciummasmamresult
1. of:
A) releasemofmstoredmcalciummfrommthemmitochondria.
B) improvedmintracellularmvolumemregulation.
C) decreasedminfluxmacrossmthemcellmmembrane.
D) attractionmofmcalciummtomfattyminfiltrates.
Thempatientmismfoundmtomhavemlivermdisease,mresultingminmthemremovalmofmamlobemofmhismliver.
2. Adaptationmtomthemreducedmsizemofmthemlivermleadsmtom_ ofmthemremainingmlivermcells.
A) metaplasia
B) organmatrophy
C) compensatorymhyperplasia
D) physiologicmhypertrophy
Ampersonmeatingmpeanutsmstartsmchokingmandmcollapses.mHismairwaymobstructionmismpartiallymcle
ared,mbutmhemremainsmhypoxicmuntilmhemreachesmthemhospital.mThemprolongedmcellmhypoxia
3. causedmamcerebralminfarctionmandmresulting _minmthembrain.
A) caspasemactivation
B) coagulationmnecrosis
C) rapidmphagocytosis
D) proteinmp53mdeficiency
Bacteriamandmvirusesmcausemcellmdamagemby ,mwhichmismuniquemfrommthemintracellular
4. damagemcausedmbymotherminjuriousmagents.
A) disruptingmthemsodium/potassiummATPasempump
B) interruptingmoxidativemmetabolismmprocesses
C) replicatingmandmproducingmcontinuedminjury
D) decreasingmproteinmsynthesismandmfunction
Thempatientmhasmamprolongedminterruptionminmarterialmbloodmflowmtomhismleftmkidney,mcausing
5. hypoxicmcellminjurymandmthemreleasemofmfreemradicals.mFreemradicalsmdamagemcellsmby:
A) destroyingmphospholipidsminmthemcellmmembrane.
B) alteringmthemimmunemresponsemofmthemcell.
C) disruptingmcalciummstorageminmthemcell.
D) inactivationmofmenzymesmandmmitochondria.

, 6. Injuredmcellsmhavemimpairedmflowmofmsubstancesmthroughmthemcellmmembranemasmamresultmof:
A) increasedmfatmload.
B) alteredmpermeability.
C) alteredmglucosemutilization.
D) increasedmsurfacemreceptors.
7. Reversiblemadaptivemintracellularmresponsesmareminitiatedmby:
A) stimulusmoverload.
B) geneticmmutations.
C) chemicalmmessengers.
D) mitochondrialmDNA.
8. Injuredmcellsmbecomemverymswollenmasmamresultmof:
A) increasedmcellmproteinmsynthesis.
B) alteredmcellmvolumemregulation.
C) passivementrymofmpotassiummintomthemcell.
D) blebmformationminmthemplasmammembrane.
Amdiabeticmpatientmhasmimpairedmsensation,mcirculation,mandmoxygenationmofmhismfeet.mHemstepsm
onmampiecemofmglass,mthemwoundmdoesmnotmheal,mandmthemareamtissuembecomesmnecrotic.mThemnec
rotic
9. cellmdeathmism characterizedmby:
A) rapidmapoptosis.
B) cellularmrupture.
C) shrinkagemandmcollapse.
D) chronicminflammation.
Am99-year-oldmwomanmhasmexperiencedmthemdeclinemofmcellmfunctionmassociatedmwithmage.mA
10. groupmofmtheoriesm ofmcellularmagingmfocusmonmprogrammed:
A) changesmwithmgeneticminfluences.
B) eliminationmofmcellmreceptormsites.
C) insufficientmtelomerasemenzyme.
D) DNAmmutationmormfaultymrepair.
Anm89-year-
oldmfemalempatientmhasmexperiencedmsignificantmdecreasesminmhermmobilitymandmstaminamduring
mam3-

weekmhospitalmstaymformthemtreatmentmofmamfemoralmheadmfracture.mWhichmofmthemfollowingmphe
nomenammostmlikelymaccountsmformthempatientsmdecreaseminmmusclemfunction
11. thatmunderliesmhermreducedmmobility?
A) Impairedmmusclemcellmmetabolismmresultingmfrommmetaplasia
B) Dysplasiamasmamconsequencemofminflammationmduringmbonemremodeling
C) Disusematrophymofmmusclem cellsmduringmamprolongedmperiodmofmimmobility