NR507 Advanced Pathophysiology MidTerm EXAM

NR507 Advanced Pathophysiology MidTerm
NEWEST 2025 ACTUAL EXAM COMPLETE QUESTIONS
AND CORRECT DETAILED ANSWERS (VERIFIED
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1. Hypersensitivity:Type 1: Type 1:Allergic reaction, Mediated by IgE, Inflamma- lk lk lk lk lk lk lk lk lk lk




tion due to mast cell degranulation
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Local symptoms:lk




-itching
-rash
Systemic symptoms: lk




-wheezing
Most dangerous = anaphylactic reaction
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systemic response of hypotension, severe bronchoconstriction Main tr
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eatment: epinephrine reverses the effects
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2. Hypersensitivity:Type 2: Type 2:Cytotoxicreaction;tissue specific (ex:thyroid tissu lk lk lk lk lk lk lk lk lk lk lk




e)

Macrophages are the primary effectors cells involved Can
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cause tissue damage or alter function
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Grave's disease (hyperthyroidism) -
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example of altering thyroid function, but does not destroy thyroid tissue
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, Incompatibleblood type-example ofcell/tissue damage thatoccurs; severe transfu-
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sion reaction occurs and the transfused erythrocytes are destroyed by agglutination or co
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mplement-mediated lysis. lk




3. Type 1 Hypersensitivity VS.Type 2 Hypersensitivity: Type 1 Hypersensitivity Org
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an Specific lk




Antibody binds to the antigen on the cell surface
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Type 2 Hypersensitivity lk lk lk




Not Organ Specific lk lk




Antibody binds to the soluble antigen outside the cell surface that was released into the blo
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d or body fluids, and the complex is then deposited in the tissues
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4. Hypersensitivity:Type 3 - lk lk lk




Examples: Rheumatoid arthritis: Antigen/antibodies are deposited in the joints
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Systemic Lupus Erythematosus (SLE)- very closely related to autoimmunity- anti-
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gen/antibodies deposit in organs that cause tissue damage
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5. Hypersensitivity:Type 4: Delayed response lk lk lk lk