,Neonatal and Pediatric Respiratory Care, 6th Edition, Brian K. Walsh Test Bank
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Table of Contents
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Chapter 1. Fetal Lung Development
Xt Xt Xt Xt
Chapter 2. Fetal Gas Exchange and Circulation
Xt Xt Xt Xt Xt Xt
Chapter 3. Antenatal Assessment and High-Risk Delivery
Xt Xt Xt Xt Xt Xt
Chapter 4. Examination and Assessment of the Neonatal and Pediatric Patient
Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt
Chapter 5. Pulmonary Function Testing and Bedside Pulmonary Mechanics
Xt Xt Xt Xt Xt Xt Xt Xt
Chapter 6. Radiographic Assessment
Xt Xt Xt
Chapter 7. Pediatric Flexible Bronchoscopy
Xt Xt Xt Xt
Chapter 8. Invasive Blood Gas Analysis and Cardiovascular Monitoring
Xt Xt Xt Xt Xt Xt Xt Xt
Chapter 9. Noninvasive Monitoring in Neonatal and Pediatric Care
Xt Xt Xt Xt Xt Xt Xt Xt
Chapter 10. Oxygen Administration
Xt Xt Xt
Chapter 11. Aerosols and Administration of Inhaled Medications
Xt Xt Xt Xt Xt Xt Xt
Chapter 12. Airway Clearance Techniques and Hyperinflation Therapy
Xt Xt Xt Xt Xt Xt Xt
Chapter 13. Airway Management
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Chapter 14. Surfactant Replacement Therapy
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Chapter 15. Noninvasive Mechanical Ventilation and Continuous Positive Pressure of the Neonate
Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt
Chapter 16. Noninvasive Mechanical Ventilation of the Infant and Child
Xt Xt Xt Xt Xt Xt Xt Xt Xt
Chapter 17. Invasive Mechanical Ventilation of the Neonate and Pediatric Patient
Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt
Chapter 18. Administration of Gas Mixtures
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Chapter 19. Extracorporeal Membrane Oxygenation
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Chapter 20. Pharmacology
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Chapter 21. Thoracic Organ Transplantation
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Chapter 22. Neonatal Pulmonary Disorders
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Chapter 23. Surgical Disorders in Childhood that Affect Respiratory Care
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Chapter 24. Congenital Cardiac Defects
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Chapter 25. Pediatric Sleep-Disordered Breathing
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Chapter 26. Pediatric Airway Disorders and Parenchymal Lung Diseases
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Chapter 27. Asthma
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Chapter 28. Cystic Fibrosis
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Chapter 29. Acute Respiratory Distress Syndrome
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Chapter 30. Shock
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Chapter 31. Pediatric Trauma
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Chapter 32. Disorders of the Pleura
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Chapter 33. Neurological and Neuromuscular Disorders
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Chapter 34. Pediatric Emergencies
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Chapter 35. Home Care of the Postpartum Family
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Chapter 36. Quality and Safety
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,Chapter 1: Fetal Lung Development
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Walsh: Neonatal & Pediatric Respiratory Care 6th Edition Test Bank (2020)
Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt
MULTIPLE CHOICE Xt
1. Which of the following phases of human lung development is characterized by the formati
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on of a capillary network around airway passages?
Xt Xt Xt Xt Xt Xt Xt
a. Pseudoglandular
b. Saccular
c. Alveolar
d. Canalicular
ANS: D Xt
The canalicular phase follows the pseudoglandular phase, lasting from approximately 17 w
Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt
eeks to 26 weeks of gestation. This phase is so named because of the appearance of vascular
Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt
channels, or capillaries, which begin to grow by forming a capillary network around the a
Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt
ir passages. During the pseudoglandular stage, which begins at day 52 and extends to wee
Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt
k 16 of gestation, the airway system subdivides extensively and the conducting airway sys
Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt
tem develops, ending with the terminal bronchioles. The saccular stage of development, w
Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt
hich takes place from weeks 29 to 36 of gestation, is characterized by the development of s
Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt
acs that later become alveoli. During the saccular phase, a tremendous increase in the pote
Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt
ntial gas- Xt
exchanging surface area occurs. The distinction between the saccular stage and the alveol
Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt
ar stage is arbitrary. The alveolar stage stretches from 39 weeks of gestation to term. This
Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt
stage is represented by the establishment of alveoli.
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REF: pp. 3-5 Xt X t
2. Regarding postnatal lung growth, by approximately what age do most of the alveoli that w
Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt
ill be present in the lungs for life develop?
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a. 6 months Xt
b. 1 year Xt
c. 1.5 years Xt
d. 2 years Xt
ANS: C Xt
Most of the postnatal formation of alveoli in the infant occurs over the first 1.5 years of li
Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt
fe. At 2 years of age, the number of alveoli varies substantially among individuals. After 2 y
Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt
ears of age, males have more alveoli than do females. After alveolar multiplication ends, t
Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt
he alveoli continue to increase in size until thoracic growth is completed.
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REF: p. 6 Xt Xt
3. The respiratory therapist is evaluating a newborn with mild respiratory distress due to trache
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al stenosis. During which period of lung development did this problem develop?
Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt
, a. Embryonal
b. Saccular
c. Canalicular
d. Alveolar
ANS: A Xt
The initial structures of the pulmonary tree develop during the embryonal stage. Errors in
Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt
development during this time may result in laryngeal, tracheal, or esophageal atresia or ste
Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt
nosis. Pulmonary hypoplasia, an incomplete development of the lungs characterized by an ab
Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt
normally low number and/or size of bronchopulmonary segments and/or alveoli, can devel
Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt
op during the pseudoglandular phase. If the fetus is born during the canalicular phase (i.e.,
Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt
prematurely), severe respiratory distress can be expected because the inadequately develo
Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt
ped airways, along with insufficient and immature surfactant production by alveolar type I
Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt
I cells, gives rise to the constellation of problems known as infant respiratory distress syn
Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt
drome.
REF: XtXt p. 6 Xt
4. Which of the following mechanisms is (are) responsible for the possible association betwee
Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt
n oligohydramnios and lung hypoplasia?
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I. Abnormal carbohydrate metabolism Xt Xt
II. Mechanical restriction of the chest wall Xt Xt Xt Xt Xt
III. Interference with fetal breathing Xt Xt Xt
IV. Failure to produce fetal lung liquid Xt Xt Xt Xt Xt
a. I and III only Xt Xt Xt
b. II and III only Xt Xt Xt
c. I, II, and IV only Xt Xt Xt Xt
d. II, III, and IV only Xt Xt Xt Xt
ANS: D Xt
Oligohydramnios, a reduced quantity of amniotic fluid present for an extended period of timXt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt
e, with or without renal anomalies, is associated with lung hypoplasia. The mechanisms by
Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt X
which amniotic fluid volume influences lung growth remain unclear. Possible explanations fo
t Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt
r reduced quantity of amniotic fluid include mechanical restriction of the chest wall, interfe
Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt
rence with fetal breathing, or failure to produce fetal lung liquid. These clinical and experi
Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt
mental observations possibly point to a common denominator, lung stretch, as being a maj
Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt
or growth stimulant.
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REF: pp. 6-7 Xt Xt
5. What is the purpose of the substance secreted by the type II pneumocyte?
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a. To increase the gas exchange surface area
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b. To reduce surface tensionXt Xt Xt
c. To maintain lung elasticity
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d. To preserve the volume of the amniotic fluid
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Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt
Table of Contents
Xt Xt
Chapter 1. Fetal Lung Development
Xt Xt Xt Xt
Chapter 2. Fetal Gas Exchange and Circulation
Xt Xt Xt Xt Xt Xt
Chapter 3. Antenatal Assessment and High-Risk Delivery
Xt Xt Xt Xt Xt Xt
Chapter 4. Examination and Assessment of the Neonatal and Pediatric Patient
Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt
Chapter 5. Pulmonary Function Testing and Bedside Pulmonary Mechanics
Xt Xt Xt Xt Xt Xt Xt Xt
Chapter 6. Radiographic Assessment
Xt Xt Xt
Chapter 7. Pediatric Flexible Bronchoscopy
Xt Xt Xt Xt
Chapter 8. Invasive Blood Gas Analysis and Cardiovascular Monitoring
Xt Xt Xt Xt Xt Xt Xt Xt
Chapter 9. Noninvasive Monitoring in Neonatal and Pediatric Care
Xt Xt Xt Xt Xt Xt Xt Xt
Chapter 10. Oxygen Administration
Xt Xt Xt
Chapter 11. Aerosols and Administration of Inhaled Medications
Xt Xt Xt Xt Xt Xt Xt
Chapter 12. Airway Clearance Techniques and Hyperinflation Therapy
Xt Xt Xt Xt Xt Xt Xt
Chapter 13. Airway Management
Xt Xt Xt
Chapter 14. Surfactant Replacement Therapy
Xt Xt Xt Xt
Chapter 15. Noninvasive Mechanical Ventilation and Continuous Positive Pressure of the Neonate
Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt
Chapter 16. Noninvasive Mechanical Ventilation of the Infant and Child
Xt Xt Xt Xt Xt Xt Xt Xt Xt
Chapter 17. Invasive Mechanical Ventilation of the Neonate and Pediatric Patient
Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt
Chapter 18. Administration of Gas Mixtures
Xt Xt Xt Xt Xt
Chapter 19. Extracorporeal Membrane Oxygenation
Xt Xt Xt Xt
Chapter 20. Pharmacology
Xt Xt
Chapter 21. Thoracic Organ Transplantation
Xt Xt Xt Xt
Chapter 22. Neonatal Pulmonary Disorders
Xt Xt Xt Xt
Chapter 23. Surgical Disorders in Childhood that Affect Respiratory Care
Xt Xt Xt Xt Xt Xt Xt Xt Xt
Chapter 24. Congenital Cardiac Defects
Xt Xt Xt Xt
Chapter 25. Pediatric Sleep-Disordered Breathing
Xt Xt Xt Xt
Chapter 26. Pediatric Airway Disorders and Parenchymal Lung Diseases
Xt Xt Xt Xt Xt Xt Xt Xt
Chapter 27. Asthma
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Chapter 28. Cystic Fibrosis
Xt Xt Xt
Chapter 29. Acute Respiratory Distress Syndrome
Xt Xt Xt Xt Xt
Chapter 30. Shock
Xt Xt
Chapter 31. Pediatric Trauma
Xt Xt Xt
Chapter 32. Disorders of the Pleura
Xt Xt Xt Xt Xt
Chapter 33. Neurological and Neuromuscular Disorders
Xt Xt Xt Xt Xt
Chapter 34. Pediatric Emergencies
Xt Xt Xt
Chapter 35. Home Care of the Postpartum Family
Xt Xt Xt Xt Xt Xt Xt
Chapter 36. Quality and Safety
Xt Xt Xt Xt
,Chapter 1: Fetal Lung Development
Xt Xt Xt Xt
Walsh: Neonatal & Pediatric Respiratory Care 6th Edition Test Bank (2020)
Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt
MULTIPLE CHOICE Xt
1. Which of the following phases of human lung development is characterized by the formati
Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt
on of a capillary network around airway passages?
Xt Xt Xt Xt Xt Xt Xt
a. Pseudoglandular
b. Saccular
c. Alveolar
d. Canalicular
ANS: D Xt
The canalicular phase follows the pseudoglandular phase, lasting from approximately 17 w
Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt
eeks to 26 weeks of gestation. This phase is so named because of the appearance of vascular
Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt
channels, or capillaries, which begin to grow by forming a capillary network around the a
Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt
ir passages. During the pseudoglandular stage, which begins at day 52 and extends to wee
Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt
k 16 of gestation, the airway system subdivides extensively and the conducting airway sys
Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt
tem develops, ending with the terminal bronchioles. The saccular stage of development, w
Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt
hich takes place from weeks 29 to 36 of gestation, is characterized by the development of s
Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt
acs that later become alveoli. During the saccular phase, a tremendous increase in the pote
Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt
ntial gas- Xt
exchanging surface area occurs. The distinction between the saccular stage and the alveol
Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt
ar stage is arbitrary. The alveolar stage stretches from 39 weeks of gestation to term. This
Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt
stage is represented by the establishment of alveoli.
Xt Xt Xt Xt Xt Xt Xt
REF: pp. 3-5 Xt X t
2. Regarding postnatal lung growth, by approximately what age do most of the alveoli that w
Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt
ill be present in the lungs for life develop?
Xt Xt Xt Xt Xt Xt Xt Xt
a. 6 months Xt
b. 1 year Xt
c. 1.5 years Xt
d. 2 years Xt
ANS: C Xt
Most of the postnatal formation of alveoli in the infant occurs over the first 1.5 years of li
Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt
fe. At 2 years of age, the number of alveoli varies substantially among individuals. After 2 y
Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt
ears of age, males have more alveoli than do females. After alveolar multiplication ends, t
Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt
he alveoli continue to increase in size until thoracic growth is completed.
Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt
REF: p. 6 Xt Xt
3. The respiratory therapist is evaluating a newborn with mild respiratory distress due to trache
Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt
al stenosis. During which period of lung development did this problem develop?
Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt
, a. Embryonal
b. Saccular
c. Canalicular
d. Alveolar
ANS: A Xt
The initial structures of the pulmonary tree develop during the embryonal stage. Errors in
Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt
development during this time may result in laryngeal, tracheal, or esophageal atresia or ste
Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt
nosis. Pulmonary hypoplasia, an incomplete development of the lungs characterized by an ab
Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt
normally low number and/or size of bronchopulmonary segments and/or alveoli, can devel
Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt
op during the pseudoglandular phase. If the fetus is born during the canalicular phase (i.e.,
Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt
prematurely), severe respiratory distress can be expected because the inadequately develo
Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt
ped airways, along with insufficient and immature surfactant production by alveolar type I
Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt
I cells, gives rise to the constellation of problems known as infant respiratory distress syn
Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt
drome.
REF: XtXt p. 6 Xt
4. Which of the following mechanisms is (are) responsible for the possible association betwee
Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt
n oligohydramnios and lung hypoplasia?
Xt Xt Xt Xt
I. Abnormal carbohydrate metabolism Xt Xt
II. Mechanical restriction of the chest wall Xt Xt Xt Xt Xt
III. Interference with fetal breathing Xt Xt Xt
IV. Failure to produce fetal lung liquid Xt Xt Xt Xt Xt
a. I and III only Xt Xt Xt
b. II and III only Xt Xt Xt
c. I, II, and IV only Xt Xt Xt Xt
d. II, III, and IV only Xt Xt Xt Xt
ANS: D Xt
Oligohydramnios, a reduced quantity of amniotic fluid present for an extended period of timXt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt
e, with or without renal anomalies, is associated with lung hypoplasia. The mechanisms by
Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt X
which amniotic fluid volume influences lung growth remain unclear. Possible explanations fo
t Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt
r reduced quantity of amniotic fluid include mechanical restriction of the chest wall, interfe
Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt
rence with fetal breathing, or failure to produce fetal lung liquid. These clinical and experi
Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt
mental observations possibly point to a common denominator, lung stretch, as being a maj
Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt
or growth stimulant.
Xt Xt
REF: pp. 6-7 Xt Xt
5. What is the purpose of the substance secreted by the type II pneumocyte?
Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt Xt
a. To increase the gas exchange surface area
Xt Xt Xt Xt Xt Xt
b. To reduce surface tensionXt Xt Xt
c. To maintain lung elasticity
Xt Xt Xt
d. To preserve the volume of the amniotic fluid
Xt Xt Xt Xt Xt Xt Xt