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Exam (elaborations)

2025 Latest NSG 3850 Pathophysiology Exams 1, 2 & 3 Each Exam contains 60 Correctly Answered Questions from Unit 1-7 / NSG 3850 Exam Prep 2025

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2025 Latest NSG 3850 Pathophysiology Exams 1, 2 & 3 Each Exam contains 60 Correctly Answered Questions from Unit 1-7 / NSG 3850 Exam Prep 2025

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2025 Latest NSG 3850 Pathophysiology Exams 1, 2 &
3 Each Exam contains 60 Correctly Answered
Questions from Unit 1-7 / NSG 3850 Exam Prep 2025
Contents
NSG 3850 Exam 1 .......................................................................................................................... 1
NSG 3850 Exam 2 ........................................................................................................................ 20
NSG 3850 Exam 3 ........................................................................................................................ 35



NSG 3850 Exam 1
(Unit 1 & 2)
A person who has hyperparathyroidism is likely to develop
a. hypokalemia
b. hyperkalemia
c. hypocalcemia
d. hypercalcemia - ANSWER-D


The inward-pulling force of particles in the vascular fluid is called _____ pressure
a. capillary hydrostatic
b. interstitial osmotic
c. capillary osmotic
d. interstitial hydrostatic - ANSWER-C


How do clinical conditions that increase vascular permeability cause edema?
a. through altering the negative charge on the capillary basement membrane, which
enables excessive fluid to accumulate in the interstitial compartment
b. by causing movement of fluid from the vascular compartment into the
intracellular compartment, which leads to cell swelling

pg. 1

,c. through leakage of vascular fluid into the interstitial fluid, which increases
interdigital fluid hydrostatic pressure
d. by allowing plasma proteins to leak into the interstitial fluid, which draws in
excess fluid by increasing the interstitial fluid osmotic pressure - ANSWER-D


The process responsible for distribution of fluid between the interstitial and
intracellular compartments is
a. filtration
b. osmosis
c. active transport
d. diffusion - ANSWER-B


Which electrolyte imbalances cause increase neuromuscular excitability?
a. hypokalemia and hyperphosphatemia
b. hyperkalemia and hypophosphatemia
c. hypocalcemia and hypomagnesemia
d. hypercalcemia and hypermagnesemia - ANSWER-C


Excessive antidiuretic hormone (ADH) secretion can cause ____ concentration
a. increased serum sodium
b. decreased serum sodium
c. increased serum potassium
d. decreased serum potassium - ANSWER-B


Causes of hypomagnesemia include
a. hyperphophatemia



pg. 2

,b. chronic alcoholism
c. oliguric renal failure
d. clinical dehydration - ANSWER-B


Hypernatremia may be caused by
a. decreased aldosterone secretion
b. decreased antidiuretic hormone secretion
c. compulsive water drinking
d. excessive dietary potassium - ANSWER-B


Clinical manifestations of hyponatremia include
a. weak pulse, low BP, increased HR
b. thirst, dry mucous membranes, diarrhea
c. confusion, lethargy, coma, seizures
d. cardiac dysrhythmias, parenthesis, muscle weakness - ANSWER-C


Clinical manifestations of extracellular fluid volume deficit include
a. weak pulse, low BP, increased HR
b. thirst, dry mucous membranes, diarrhea
c. confusion, lethargy, coma, seizures
d. cardiac dysrhythmias, parenthesis, muscle weakness - ANSWER-A


the imbalance that occurs with oliguric renal failure is
a. metabolic alkalosis
b. hyperkalemia



pg. 3

, c. hypokalemia
d. hypophosphatemia - ANSWER-B


A known cause of hypokalemia is
a. oliguric renal failure
b. pancreatitis
c. insulin overdose
d. hyperparathyroidism - ANSWER-C


Effects of hypernatremia on the CNS typically include
a. confusion
b. excitation
c. insomnia
d. hallucinations - ANSWER-A


Clinical manifestations of moderate to severe hypokalemia include
a. muscle spasms and rapid respirations
b. muscle weakness and cardia dysrhythmias
c. confusion nd irritability
d. vomiting and diarrhea - ANSWER-B


qDecreased neuromuscular excitability is often the result of
a. hypercalcemia and hypermagnesemia
b. hypomagnesemia and hyperkalemia
c. hypocalcemia and hypokalemia



pg. 4

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