ALTERATIONS OF HEMATOLOGIC FUNCTION IN CHILDREN CH 30:
QUESTIONS WITH 100% CORRECT ANSWERS + RATIONALES 2025-2026
(A+ GRADED)
,
QUESTIONS WITH 100% CORRECT ANSWERS + RATIONALES 2025-2026
(A+ GRADED)
,
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ALTERATIONS OF HEMATOLOGIC FUNCTION IN CHILDREN CH 30: QUESTIONS WITH 100% CORRECT ANSWERS + RATIONALES (A+ GRADED)
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ALTERATIONS OF HEMATOLOGIC FUNCTION IN CHILDREN CH 30: QUESTIONS WITH 100% CORRECT ANSWERS + RATIONALES (A+ GRADED) What is the cause of polycythemia in the fetus? a. Fetal hemoglobin has a greater affinity for oxygen as a result of diphosphoglycerate (DPG). b. The fetus has a different hemoglobin structure of two x- and two y-chains rather than two x-and two b-chains. c. Increased erythropoiesis occurs in response to the hypoxic intrauterine environment. d. The lungs of the fetus are undeveloped and unable to diffuse oxygen adequately to the pulmonary capillaries. - Correct Answer-ANS: C The hypoxic intrauterine environment stimulates erythropoietin production in the fetus. This selection is the only option that accurately explains why the fetus develops polycythemia. Why does fetal hemoglobin have a greater affinity for oxygen than adult hemoglobin? a. The fetus does not have its own oxygen supply and must rely on oxygen from the maternal vascular system. b. The fetus has two -chains on the hemoglobin, rather than two -chains as in the adult. c. Fetal hemoglobin interacts less readily with diphosphoglycerate (DPG), which inhibits hemoglobin-oxygen binding. d. Fetal hemoglobin production occurs in the vessels and liver rather than in the bone marrow as in the adult. - Correct Answer-ANS Continues...
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- Subido en
- 12 de septiembre de 2025
- Número de páginas
- 15
- Escrito en
- 2025/2026
- Tipo
- Examen
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- Preguntas y respuestas
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