N244 – Nursing Pharmacology – Respiratory Medications Questions and Answers
Big things that can cause ARDS are: *gastric aspiration
*septic shock/sepsis*****
*multiple blood transfusions
*near drowning
*DIC
*pancreatitis
Defining if the patient has ARDs is by comparing? PaOx to FiOs of 200mmHg or less****
ALI >300mgHg
Differeniate ARDS from HF by? *PAWP <18 for ARDS, <18 for HF
*Bronchoalveloar lavage fluid will be rich in protein and inflammatory cells with ARDS
*BNP <100pg/ml indicates ARDS, A high BNP indicates heart failure
*Chest -x-ray Pulmonary infiltrates throughout the lungs
ARDs facts: *comes on approx. 48-72 hours after precipitating event (sepsis is a big one)
*respiratory rate increases
*hypoxemia
*dyspnea
The increased breathing results in respiratory alkalosis in abgs.
*as it progresses
*cyanosis
*accessory muscles engage
*cough with sputum develops
, *heart rate increases
*crackles
*Blood gases turn toward acidosis and its refractory to oxygen. Due to shunt from alveolar
collapse and become dysfunctionable.
The nursing goal for ARDS is? *maintaining hemodynamic and oxygenation until the
syndrome resolves.
*ventilation with PEEP (that is inspiratory and expiratory pressure
*patient postion strategies
*drug therapy.
According to ARDS consensus criteria, a diagnosis of ARDS requires a P/F ration of less than?
<200
What condition is the most common indirect predisposing disorder of ARDS?
*sepsis***********************
The pulmonary edema associated with ARDS is cause by which condition? injured alveolar-
capillary membrane
Ther term permissive hypercapnia refers to which therapeutic strategy? Reducing peak
airway pressure by allowing some increase in PaCO2.
What will we see on ARDS? *lung infiltrates from leaking capillaries/fluid filled lungs
*oxygen PaO2/FiO2<200
*no elevated pulmonary pressure
Big things that can cause ARDS are: *gastric aspiration
*septic shock/sepsis*****
*multiple blood transfusions
*near drowning
*DIC
*pancreatitis
Defining if the patient has ARDs is by comparing? PaOx to FiOs of 200mmHg or less****
ALI >300mgHg
Differeniate ARDS from HF by? *PAWP <18 for ARDS, <18 for HF
*Bronchoalveloar lavage fluid will be rich in protein and inflammatory cells with ARDS
*BNP <100pg/ml indicates ARDS, A high BNP indicates heart failure
*Chest -x-ray Pulmonary infiltrates throughout the lungs
ARDs facts: *comes on approx. 48-72 hours after precipitating event (sepsis is a big one)
*respiratory rate increases
*hypoxemia
*dyspnea
The increased breathing results in respiratory alkalosis in abgs.
*as it progresses
*cyanosis
*accessory muscles engage
*cough with sputum develops
, *heart rate increases
*crackles
*Blood gases turn toward acidosis and its refractory to oxygen. Due to shunt from alveolar
collapse and become dysfunctionable.
The nursing goal for ARDS is? *maintaining hemodynamic and oxygenation until the
syndrome resolves.
*ventilation with PEEP (that is inspiratory and expiratory pressure
*patient postion strategies
*drug therapy.
According to ARDS consensus criteria, a diagnosis of ARDS requires a P/F ration of less than?
<200
What condition is the most common indirect predisposing disorder of ARDS?
*sepsis***********************
The pulmonary edema associated with ARDS is cause by which condition? injured alveolar-
capillary membrane
Ther term permissive hypercapnia refers to which therapeutic strategy? Reducing peak
airway pressure by allowing some increase in PaCO2.
What will we see on ARDS? *lung infiltrates from leaking capillaries/fluid filled lungs
*oxygen PaO2/FiO2<200
*no elevated pulmonary pressure
