NURS5315 EXAM 3 QUESTIONS
WITH 100% VERIFIED ANSWERS
Atypical MI symptoms - Answer-Confusion, N/V, fatigue, palpitations. May not
experience any chest pain
acute coronary syndrome - Answer-sudden symptoms of insufficient blood supply to the
heart indicating unstable angina or acute myocardial infarction
Unstable angina - Answer-_________________ is chest pain at rest or chest pain of
increasing frequency. Ischemia is caused by a labile thrombus which does not occlude
blood flow for any longer than _____ minutes.
Unstable angina - Answer-normal troponin, T-wave inversions, ST-depression
NSTEMI - Answer-Results from a thrombus occluding coronary blood flow for greater
than _____ minutes and results in myocardial_______________ but does not involve
the full thickness of the ventricular wall. May be seen on EKG with ST depression or T
wave inversion. AKA subendocardial infarct.
STEMI - Answer-Trhombus permanently lodged in the vessel causing necrosis of the
entire thickness or the ventricular wall. AKA transmural infarct
STEMI - Answer-Marked by ST elevation on an EKG
10 seconds - Answer-After _____ ____ of occluded blood flow r/t an MI the myocytes
become cyanotic
Lactic acid and hydrogen ions - Answer-Anaerobic metabolism during an MI leads to the
accumulation of (2 things)
Coagulative - Answer-Necrosis that occurs in an MI
20 - Answer-necrosis occurs in ____ minutes following an MI
RCA - Answer-If Mi occurs in the _____, the SA node may be affected
myocardial stunning - Answer-temporary loss of contractile function that persists for
hours to days after perfusion has been restored
,Hibernating myocardium - Answer-Tissue that is persistently ischemic undergoes
metabolic adaptation to prolong myocyte survival
myocardial remodeling - Answer-characterized by an increase in ventricular muscle
mass, involves changes in the size, shape, structure and function of the heart. Helps
maintain cardiac output and reduces myocardial wall stress.
Pulmonary crackles - Answer-Breath sounds heard in an MI
Troponins - Answer-Cardiac specific protein used to monitor MI
Released following cell death (necrosis)
Serum levels are elevated within 2-4 hours of onset of pain and persists for 7-14 days
Transmural - Answer-affecting the entire thickness of a wall
Depression - Answer-ST ______________ is not transmural and indicates a
subendocardial infarction with a small area affected.
Q waves - Answer-indicate necrosis, dead cells which cannot depolarize, may be old
and indicate an old MI
Acute - Answer-The presence of Q waves with ST elevation indicates _____ infarct.
T-wave inversion, decreased - Answer-Myocardial ischemia is marked by _________
___________. This simply means that blood flow is ___________
Anterior Wall MI - Answer--involves LAD artery
-Leads V1, V2, V3, V4 with ST elevation
-supplies blood to left anterior ventricle
-may result in left ventricular pump failure, cardiogenic shock, or death
-increased risk of death
Anteroseptal MI - Answer-ST elevation and/or significant Q in V1 or V2
*left Anterior descending artery is effected
Anterolateral MI - Answer-LAD or LCX, ST elevation, Q wave or ST depression in V4,
V5, or V6
Lateral Wall MI - Answer--involves circumflex artery
-Leads: I, aVL
-often accompanies anterior or inferior wall MI's
-ST elevations or depression
-Q waves
Inferior Wall MI - Answer--involves RCA
-Leads: II, III, aVF
, -may develop bradycardia, sinus arrest
-supplies blood to right ventricle; may develop right HF
-ST elevations or depression
-Q waves
Vfib - Answer-Most common cause of death in MI
Cardiogenic shock - Answer-Results from a large infarct and has a high mortality
Ventricular free wall rupture, LAD - Answer-_____________ ________ ________
___________Results in cardiac tamponade from hemorrhage into the pericardial space.
Associated most commonly with a thrombosis of the _____ coronary artery
Papillary muscle rupture - Answer--A potential complication of an MI during days 3-14
post MI.
-Causes severe mitral valve regurgitation
-Most commonly associated with inferior MIs due to thrombosis of RCA.
Papillary muscle rupture - Answer--Presents with acute onset of mitral valve
regurgitation and left HF
Ventricular septum rupture - Answer-MI complication that results in a VSD. Usually
associated with a thrombosis in the LAD coronary artery. Produces a left to right shunt
causing right HF.
Left, Right - Answer-Papillary muscle rupture causes ________ HF but Ventricular
septum rupture causes ________ HF.
Mural thrombus - Answer-a thrombus formed within the wall of the heart (myocardium)
or vessel (media) following an MI associated with LAD coronary thrombosis. Danger of
peripheral embolus
Ventricular aneurysm - Answer-results when the infarcted myocardial wall becomes
thinned and bulges out during contraction. Usually occurs 4-8 weeks after STEMI but
starts to develop within 48 hours
Friction Rub - Answer-In post infarction fibrinous pericarditis, the person will develop a
______ ____ 1-3 days post MI.
AV stenosis - Answer-Most common cause of sudden cardiac death in children
Wolff-Parkinson-White Syndrome - Answer-is a syndrome of pre-excitation of the
ventricles of the heart due to an accessory pathway known as the bundle of Kent. This
accessory pathway is an abnormal electrical communication from the atria to the
ventricles. A type of atrioventricular reentrant tachycardia. Has a "Delta Wave" on the
EKG
WITH 100% VERIFIED ANSWERS
Atypical MI symptoms - Answer-Confusion, N/V, fatigue, palpitations. May not
experience any chest pain
acute coronary syndrome - Answer-sudden symptoms of insufficient blood supply to the
heart indicating unstable angina or acute myocardial infarction
Unstable angina - Answer-_________________ is chest pain at rest or chest pain of
increasing frequency. Ischemia is caused by a labile thrombus which does not occlude
blood flow for any longer than _____ minutes.
Unstable angina - Answer-normal troponin, T-wave inversions, ST-depression
NSTEMI - Answer-Results from a thrombus occluding coronary blood flow for greater
than _____ minutes and results in myocardial_______________ but does not involve
the full thickness of the ventricular wall. May be seen on EKG with ST depression or T
wave inversion. AKA subendocardial infarct.
STEMI - Answer-Trhombus permanently lodged in the vessel causing necrosis of the
entire thickness or the ventricular wall. AKA transmural infarct
STEMI - Answer-Marked by ST elevation on an EKG
10 seconds - Answer-After _____ ____ of occluded blood flow r/t an MI the myocytes
become cyanotic
Lactic acid and hydrogen ions - Answer-Anaerobic metabolism during an MI leads to the
accumulation of (2 things)
Coagulative - Answer-Necrosis that occurs in an MI
20 - Answer-necrosis occurs in ____ minutes following an MI
RCA - Answer-If Mi occurs in the _____, the SA node may be affected
myocardial stunning - Answer-temporary loss of contractile function that persists for
hours to days after perfusion has been restored
,Hibernating myocardium - Answer-Tissue that is persistently ischemic undergoes
metabolic adaptation to prolong myocyte survival
myocardial remodeling - Answer-characterized by an increase in ventricular muscle
mass, involves changes in the size, shape, structure and function of the heart. Helps
maintain cardiac output and reduces myocardial wall stress.
Pulmonary crackles - Answer-Breath sounds heard in an MI
Troponins - Answer-Cardiac specific protein used to monitor MI
Released following cell death (necrosis)
Serum levels are elevated within 2-4 hours of onset of pain and persists for 7-14 days
Transmural - Answer-affecting the entire thickness of a wall
Depression - Answer-ST ______________ is not transmural and indicates a
subendocardial infarction with a small area affected.
Q waves - Answer-indicate necrosis, dead cells which cannot depolarize, may be old
and indicate an old MI
Acute - Answer-The presence of Q waves with ST elevation indicates _____ infarct.
T-wave inversion, decreased - Answer-Myocardial ischemia is marked by _________
___________. This simply means that blood flow is ___________
Anterior Wall MI - Answer--involves LAD artery
-Leads V1, V2, V3, V4 with ST elevation
-supplies blood to left anterior ventricle
-may result in left ventricular pump failure, cardiogenic shock, or death
-increased risk of death
Anteroseptal MI - Answer-ST elevation and/or significant Q in V1 or V2
*left Anterior descending artery is effected
Anterolateral MI - Answer-LAD or LCX, ST elevation, Q wave or ST depression in V4,
V5, or V6
Lateral Wall MI - Answer--involves circumflex artery
-Leads: I, aVL
-often accompanies anterior or inferior wall MI's
-ST elevations or depression
-Q waves
Inferior Wall MI - Answer--involves RCA
-Leads: II, III, aVF
, -may develop bradycardia, sinus arrest
-supplies blood to right ventricle; may develop right HF
-ST elevations or depression
-Q waves
Vfib - Answer-Most common cause of death in MI
Cardiogenic shock - Answer-Results from a large infarct and has a high mortality
Ventricular free wall rupture, LAD - Answer-_____________ ________ ________
___________Results in cardiac tamponade from hemorrhage into the pericardial space.
Associated most commonly with a thrombosis of the _____ coronary artery
Papillary muscle rupture - Answer--A potential complication of an MI during days 3-14
post MI.
-Causes severe mitral valve regurgitation
-Most commonly associated with inferior MIs due to thrombosis of RCA.
Papillary muscle rupture - Answer--Presents with acute onset of mitral valve
regurgitation and left HF
Ventricular septum rupture - Answer-MI complication that results in a VSD. Usually
associated with a thrombosis in the LAD coronary artery. Produces a left to right shunt
causing right HF.
Left, Right - Answer-Papillary muscle rupture causes ________ HF but Ventricular
septum rupture causes ________ HF.
Mural thrombus - Answer-a thrombus formed within the wall of the heart (myocardium)
or vessel (media) following an MI associated with LAD coronary thrombosis. Danger of
peripheral embolus
Ventricular aneurysm - Answer-results when the infarcted myocardial wall becomes
thinned and bulges out during contraction. Usually occurs 4-8 weeks after STEMI but
starts to develop within 48 hours
Friction Rub - Answer-In post infarction fibrinous pericarditis, the person will develop a
______ ____ 1-3 days post MI.
AV stenosis - Answer-Most common cause of sudden cardiac death in children
Wolff-Parkinson-White Syndrome - Answer-is a syndrome of pre-excitation of the
ventricles of the heart due to an accessory pathway known as the bundle of Kent. This
accessory pathway is an abnormal electrical communication from the atria to the
ventricles. A type of atrioventricular reentrant tachycardia. Has a "Delta Wave" on the
EKG
