NURS142: Final Exam
1. What causes si- ‣ Vagal stimulation (vomiting; straining for BM
nus bradycardia? ‣ MI
‣ Hypoxia
‣ Digitalis toxicity
‣ Athletes
2. What treatment ‣ None unless the pt is symptomatic
is available for si- ‣ Atropine
nus bradycardia? ‣ Electronic pacemaker
‣ Epinephrine and dopamine
‣ Oxygen
3. What causes si- ‣ Atropine or bronchodilators
nus tachycardia? ‣ Emotional upset
‣ Pulmonary embolus
‣ MI
‣ Congestive heart failure
‣ Fever
‣ Inhibition of vagus nerve
‣ Hypoxia
‣ Thyrotoxicosis (thyroid storm)
, NURS142: Final Exam
4. What is the treat- ‣ Treat the cause
ment for sinus ‣ Persistent sinus tachycardia ’beta blockers to slow HR
tachycardia? ‣ Oxygen
5. What are wan- ‣ Occur when the pacemaking impulses originate from at least three different foci
dering atrial in the atria
pacemaker (WAP) ‣ Each produces its own unique P wave, resulting in a. rhythm with at least three
and multifocal different shapes of P waves
atrial tachycardia
(MAT) rhythms? WAP and MAT are exactly the same rhythms, just with differing heart rates.
6. What is the rate ‣ Mean rate < 100 (50-60s)
of WAP?
7. What is the rate ‣ Mean rate > 100
of MAT?
8. What is the cause ‣ Medication side effects
of WAP? ‣ Hypoxia
‣ Vagal stimulaiton
‣ MI
, NURS142: Final Exam
9. What is the cause ‣ COPD
of MAT? ‣ Heart disease
10. What treatment ‣ Beta blockers or calcium channel blockers if signs of decreased cardiac output
is available for exist
MAT?
11. Characteristics of ‣ Rate: any rate
premature atrial ‣ Regularity: regular but interrupted by the PACs
complexes (PACs) ‣ Premature P waves may be hidden in the T wave of the preceding beat ’deforms
the T wave
’If the PACs P wave is inverted, the PR interval should be the normal 0.12-0.20 secs
‣ Intervals: QRS <0.12; will be absent after a non conducted PAC
12.
1. What causes si- ‣ Vagal stimulation (vomiting; straining for BM
nus bradycardia? ‣ MI
‣ Hypoxia
‣ Digitalis toxicity
‣ Athletes
2. What treatment ‣ None unless the pt is symptomatic
is available for si- ‣ Atropine
nus bradycardia? ‣ Electronic pacemaker
‣ Epinephrine and dopamine
‣ Oxygen
3. What causes si- ‣ Atropine or bronchodilators
nus tachycardia? ‣ Emotional upset
‣ Pulmonary embolus
‣ MI
‣ Congestive heart failure
‣ Fever
‣ Inhibition of vagus nerve
‣ Hypoxia
‣ Thyrotoxicosis (thyroid storm)
, NURS142: Final Exam
4. What is the treat- ‣ Treat the cause
ment for sinus ‣ Persistent sinus tachycardia ’beta blockers to slow HR
tachycardia? ‣ Oxygen
5. What are wan- ‣ Occur when the pacemaking impulses originate from at least three different foci
dering atrial in the atria
pacemaker (WAP) ‣ Each produces its own unique P wave, resulting in a. rhythm with at least three
and multifocal different shapes of P waves
atrial tachycardia
(MAT) rhythms? WAP and MAT are exactly the same rhythms, just with differing heart rates.
6. What is the rate ‣ Mean rate < 100 (50-60s)
of WAP?
7. What is the rate ‣ Mean rate > 100
of MAT?
8. What is the cause ‣ Medication side effects
of WAP? ‣ Hypoxia
‣ Vagal stimulaiton
‣ MI
, NURS142: Final Exam
9. What is the cause ‣ COPD
of MAT? ‣ Heart disease
10. What treatment ‣ Beta blockers or calcium channel blockers if signs of decreased cardiac output
is available for exist
MAT?
11. Characteristics of ‣ Rate: any rate
premature atrial ‣ Regularity: regular but interrupted by the PACs
complexes (PACs) ‣ Premature P waves may be hidden in the T wave of the preceding beat ’deforms
the T wave
’If the PACs P wave is inverted, the PR interval should be the normal 0.12-0.20 secs
‣ Intervals: QRS <0.12; will be absent after a non conducted PAC
12.
