CLT LANA NEW EXAM WITH ACCURATE SOLUTIONS (A+)
Stage 0 CVI - ANSWER No symptoms, norm protein
Stage I cvi - ANSWER Mild edema, normal protein, norm lymph vessels,
phlebolymphodynamic insufficiency
Stage 2 cvi - ANSWER Mod symptoms ( pigmentation, viscosity, pain), high protein,
vessels morphologically changed, TC reduced, phlebolymphostatic insufficiency
Stage 3 cvi - ANSWER Severe symptoms( hypoxia, necrosis, pain), very high protein
load, lymph vessels morphologically change, TC reduced, severe phlebolymphostatic
insufficiency
Stage 0 cvi treatment ANSWER Compression therapy, elevation, exercise
Stage 1 cvi treatment ANSWER Compression therapy, elevation, exercise
Stage 2 cvi treatment ANSWER CDT
Stage 3 cvi treatment ANSWER CDT and wound care
Ambulatory venous hypertension ANSWER Failure of venous valves such that blood flow
bidirectional resulting in increased pressure to the capillaries
What occurs to leukocytes with blood capillaries in low flow states-ANSWER They
become trapped and release proteolytic enzymes and oxygen free radicals which
damage capillary basement membranes
, What occurs to plasma proteins with blood capillaries in low flow states-ANSWER
Plasma proteins leak into leak into the surrounding tissue creating a fibrin cuff, edema
and hypoxia
What is responsible for the reddish brown skin associated with edema/ lymphedema -
ANSWER High intracApillary pressure stretches endothelial cells away from each other
and eurythrocytes leave the blood capillaries staining skin with hemosiderin deposits
Ambulatory venous hypertension causes an _______________ in net filtration - ANSWER
Increase
What is the cause of mechanical insufficiency in cvi - ANSWER Fibrin leakage into the
tissue as well as an inflammatory reaction overall increase in protein and water load
What is not a consideration with staging lymphedema - ANSWER Limb volume
Mild lymphedema severity volume increase - ANSWER <20%
Moderate lymphedema severity volume increase - ANSWER 20-40%
Severe lymphedema severity increase - ANSWER >40%
Where do DVT form - ANSWER Sub fascial veins
Most common ulceration and where do they form - ANS Venous stasis and they form in
the distal 1/3 of the leg usually around the malleoli
vascular venous wounds - ANS Shallow, irregular, on anterior/ medial leg with peri
wound with hemosiderin staining,scaly, weepy, warm skin
Stage 0 CVI - ANSWER No symptoms, norm protein
Stage I cvi - ANSWER Mild edema, normal protein, norm lymph vessels,
phlebolymphodynamic insufficiency
Stage 2 cvi - ANSWER Mod symptoms ( pigmentation, viscosity, pain), high protein,
vessels morphologically changed, TC reduced, phlebolymphostatic insufficiency
Stage 3 cvi - ANSWER Severe symptoms( hypoxia, necrosis, pain), very high protein
load, lymph vessels morphologically change, TC reduced, severe phlebolymphostatic
insufficiency
Stage 0 cvi treatment ANSWER Compression therapy, elevation, exercise
Stage 1 cvi treatment ANSWER Compression therapy, elevation, exercise
Stage 2 cvi treatment ANSWER CDT
Stage 3 cvi treatment ANSWER CDT and wound care
Ambulatory venous hypertension ANSWER Failure of venous valves such that blood flow
bidirectional resulting in increased pressure to the capillaries
What occurs to leukocytes with blood capillaries in low flow states-ANSWER They
become trapped and release proteolytic enzymes and oxygen free radicals which
damage capillary basement membranes
, What occurs to plasma proteins with blood capillaries in low flow states-ANSWER
Plasma proteins leak into leak into the surrounding tissue creating a fibrin cuff, edema
and hypoxia
What is responsible for the reddish brown skin associated with edema/ lymphedema -
ANSWER High intracApillary pressure stretches endothelial cells away from each other
and eurythrocytes leave the blood capillaries staining skin with hemosiderin deposits
Ambulatory venous hypertension causes an _______________ in net filtration - ANSWER
Increase
What is the cause of mechanical insufficiency in cvi - ANSWER Fibrin leakage into the
tissue as well as an inflammatory reaction overall increase in protein and water load
What is not a consideration with staging lymphedema - ANSWER Limb volume
Mild lymphedema severity volume increase - ANSWER <20%
Moderate lymphedema severity volume increase - ANSWER 20-40%
Severe lymphedema severity increase - ANSWER >40%
Where do DVT form - ANSWER Sub fascial veins
Most common ulceration and where do they form - ANS Venous stasis and they form in
the distal 1/3 of the leg usually around the malleoli
vascular venous wounds - ANS Shallow, irregular, on anterior/ medial leg with peri
wound with hemosiderin staining,scaly, weepy, warm skin
