Nsg 3850 Patho Exam 1 Questions And Correct Verified Answers

NSG 3850 PATHO EXAM 1 QUESTIONS AND
CORRECT VERIFIED ANSWERS
DecreasedA2neuromuscularA2excitabilityA2isA2oftenA2theA2resultA2of
a.A2hypercalcemiaA2andA2hypermagnesemia
b.A2hypomagnesemiaA2andA2hyperkalemia
c.A2hypocalcemiaA2andA2hypokalemia
d.A2hypernatremiaA2andA2hypomagnesemiaA2-A2Ans--A

WhatA2isA2likelyA2toA2leadA2toA2hyponatremia?
a.A2insufficientA2ADHA2secretion
b.A2excessA2aldosteroneA2secretion
c.A2administrationA2ofA2IVA2normalA2saline
d.A2frequentA2NGA2tubeA2irrigationA2withA2waterA2-A2Ans--D

AnA2increaseA2intA2heA2restingA2membraneA2potentialA2(hyperpolarizied)A2isA2associatedA2
with
a.A2hypokalemia
b.A2hyperkalemia
c.A2hypocalcemia
d.A2hypercalcemiaA2-A2Ans--A

AbnormalitiesA2inA2intracellularA2regulationA2ofA2enzymeA2activityA2andA2cellularA2producti
onA2ofA2ATPA2areA2associatedA2with
a.A2hyponatremia
b.A2hypocalcemia
c.A2hypophosphatemia
d.A2hypokalemiaA2-A2Ans--C

TheA2fractionA2ofA2totalA2bodyA2waterA2(TBW)A2volumeA2containedA2inA2theA2intracellularA2
spaceA2inA2adultsA2is
a.A2threeA2fourths
b.A2twoA2thirds
c.A2oneA2half
d.A2oneA2thirdA2-A2Ans--B

ClinicalA2manifestationsA2ofA2severeA2symptomaticA2hypophosphatemiaA2areA2causedA2b
y
a.A2excessA2proteins
b.A2renalA2damage
c.A2deficiencyA2ofA2ATP
d.A2hypocalcemiaA2-A2Ans--C

AA2personA2whoA2overusesA2magnesium-
aluminumA2antacidsA2forA2aA2longA2periodA2ofA2timeA2isA2likelyA2toA2develop

, a.A2hypokalemia
b.A2hyperkalemia
c.A2hypophosphatemia
d.A2hyperphosphatemiaA2-A2Ans--C

TheA2electrolyteA2thatA2hasA2aA2higherA2concentrationA2inA2theA2extracellularA2fluidA2thatA2i
nA2theA2intracellularA2fluidA2isA2___A2ions
a.A2sodium
b.A2phosphate
c.A2magnesium
d.A2potassiumA2-A2Ans--A

AA2personA2whoA2hasA2hyperparathyroidismA2isA2likelyA2toA2develop
a.A2hypokalemia
b.A2hyperkalemia
c.A2hypocalcemia
d.A2hypercalcemiaA2-A2Ans--D

TheA2inward-
pullingA2forceA2ofA2particlesA2inA2theA2vascularA2fluidA2isA2calledA2_____A2pressure
a.A2capillaryA2hydrostatic
b.A2interstitialA2osmotic
c.A2capillaryA2osmotic
d.A2interstitialA2hydrostaticA2-A2Ans--C

HowA2doA2clinicalA2conditionsA2thatA2increaseA2vascularA2permeabilityA2causeA2edema?
a.A2throughA2alteringA2theA2negativeA2chargeA2onA2theA2capillaryA2basementA2membrane,A
2whichA2enablesA2excessiveA2fluidA2toA2accumulateA2inA2theA2interstitialA2compartment
b.A2byA2causingA2movementA2ofA2fluidA2fromA2theA2vascularA2compartmentA2intoA2theA2intr
acellularA2compartment,A2whichA2leadsA2toA2cellA2swelling
c.A2throughA2leakageA2ofA2vascularA2fluidA2intoA2theA2interstitialA2fluid,A2whichA2increasesA2
interdigitalA2fluidA2hydrostaticA2pressure
d.A2byA2allowingA2plasmaA2proteinsA2toA2leakA2intoA2theA2interstitialA2fluid,A2whichA2drawsA2
inA2excessA2fluidA2byA2increasingA2theA2interstitialA2fluidA2osmoticA2pressureA2-A2Ans--D

TheA2processA2responsibleA2forA2distributionA2ofA2fluidA2betweenA2theA2interstitialA2andA2int
racellularA2compartmentsA2isA2
a.A2filtration
b.A2osmosis
c.A2activeA2transport
d.A2diffusionA2-A2Ans--B

WhichA2electrolyteA2imbalancesA2causeA2increaseA2neuromuscularA2excitability?
a.A2hypokalemiaA2andA2hyperphosphatemia
b.A2hyperkalemiaA2andA2hypophosphatemia
c.A2hypocalcemiaA2andA2hypomagnesemia