Critical Care Nursing - HEMODYNAMIC MONITORING
oCO = SV X HR
oNormal CO = 4-8 L
oCI: CO adjusted to body surface area – more accurate
oCO/Total BSA = CI
oMean arterial pressure (MAP) average pressure in large vessels of body
MAP = 2 * DBP + SBP / 3
Normal MAP 70-105, 60-65 is BARE MIN
oPreload: the ventricles filling up with blood during diastole (relaxation) – refers to fluid status
Basically STRETCH – volume of blood received by the heart
Increased preload = too much fluid, super wet
Decreased preload = too little fluid, super dry
It is increased in hypervolemia, regurgitation of cardiac valves, heart failure
oAfterload: the amount of pressure that the heart has to push against to eject blood during systole (contraction) – refers to
fluid status
Basically SQUEEZE – pressure/resistance the heart has to overcome to eject blood
Increased afterload – too much fluid, super wet
Decreased afterload = too little fluid, super dry
oPulmonary Artery Wedge Pressure (PAWP) LEFT SIDED PRELOAD
Measurement of pulmonary capillary pressure which reflects left ventricular end diastolic pressure. Measures
pressure in the lungs – left ventricular preload pressure
Normal 25/10 mmHg (quarter over dime)
Rules out cardiac issues (I.E. HF causing fluid overload or capillary membrane issue like ARDS)
<18 mmHg = ARDS
>18 mmHg = Cardiac issue
oCentral Venous Pressure (CVP) RIGHT SIDED PRELOAD
Right ventricular preload or right ventricular end-diastolic pressure
Normal CVP 2-8 mmHg
>8 = too much fluid (CHF patients, fluid overload)
<3 = too dry (hypovolemia, hypotensive patients)
Reflects amount of blood returning to the heart and the ability of the heart to pump blood into the arterial
system
oLeft (PAWP) and Right preloads (CVP) – mini summary
High CVP & PAWP PATIENT IS TOO WET (fluid overload)
How do you decrease it???
oDIURETICS
oVasodilators
oFluid Restriction
Low CVP & PAWP Patient is too DRY (poor perfusion, decreased CO)
How do you increase it???
oVasopressors (Norepi, Epi)
oVasoconstrictors
oFluid Administration (Crystalloids: NS, LR)
PopS to my LEFT C my Pussy to the RIGHT
PopS (Left side)
oPAWP – left preload (6-12)
oSVR – left after load
C Pussy (right side)
oCVP – right preload (2-8)
oPVR – right after load
oSystemic Vascular resistance (SVR) LEFT SIDED AFTERLOAD
The opposition to blood flow encountered by the left ventricle
Normal SCVR = 800-1200 dynes/sec/cm
800 vessels are hella dilated and loose
1200 the vessels are constricted and tight
, oPulmonary Vascular Resistance (PVR) RIGHT SIDED AFTERLOAD
The opposition to blood flow encountered by the right ventricle
Normal PVR <250 dynes/sec/cm
oLeft (SVR) & Right (PVR) mini summary
High SVR and PVE the cardiac system has to work HARDER (fluid overload)
How do you decrease afterload??
oDiuretics
oVasodilators so the heart doesn’t have to push so hard to circulate blood to the body
Low SVR & PVR there is not enough blood to circulate (decreased CO and decreased preload)
oHow do you increase afterload???
Fluids
oSwan Ganz Catheter (Pulmonary Artery Catheter)
Measures hemodynamics in the body, catheter is advanced into the heart and the balloon sits in the distal tip of
the pulmonary artery
Balloon (1.5 mmHg) should NOT stay inflated – can cause the rupture of the PA and death
What does it tell us?
Co/CI
CVP
SvO2 (mixed venous sample)
Core temperature
PAWP
SVR – great indicator for fluid status
Patient should be supine, CONTINUOUS EKG MONITORING, monitoring coags, ABG, oxygenation
TRANSDUCER positioning needs to be leveled at the phlebostatic axis (4th intercostal space) & zero-point
referenced (leveled at 0)
The patient can move with the SG catheter but every time they move, we need to revel the transducer
CXR verifies the placement
Complications – ARRHYTHMIAS when the catheter is being thread through, it can touch the ventricle and cause
irritation vtach, v fib
Rupture, infection, air embolism
Primary Survey - ABCDE; Emergency Severity Index
oPrimary Survey: ABCDE
Airway management
Cardiac emergency
Rapid response team
Triage & Prioritizing
Poisoning
oAirway: maintain airway with cervical spine control
Responsiveness: awake & alert vs. lethargic, stuporous, obtunded, unarousable, unresponsive
Obstructed: foreign body, physical material, inflammation, edema
Assessment findings: stridor, mufÒed/changing voice, drooling, weak or absent gag reflex, facial trauma/surgery,
foreign object present
Head-tilt/Chin-tilt
Jaw thrust: cervical protection
Airway adjuncts:
Nasopharyngeal:
oCI: facial or skull fractures
Oropharyngeal:
oCI: gag reflex present
Endotracheal intubation:
oProcedure:
Gather supplies
Hyperoxygenate w/bag valve mask (100% O2)
Provider places the tube → hyperoxygenate btw attempts
Prelim confirmation → chest rise, breath sounds, ETCO2
, Secure tube
CXR
oConfirmation:
Assess for chest rise
ETCO2 detector (PURPLE TO YELLOW)
Auscultation
Rising SPO2
CXR
oBreathing: assess breathing and ventilation. Apply high flow O2 on non-rebreather.
Assessment:
Rate, Expansion
Effort
Auscultation
SpO2 & ABG
Interventions:
Positioning
O2 administration
Manual ventilation
CPAP/BiPAP/Mechanical ventilation
Pulmonary hygiene
Respiratory treatments
oCirculation: assess circulation with hemorrhage control
Assess:
Heart rate
Blood pressure
Peripheral pulses
Capillary refill
Need to ensure the patient is being perfused!
Problems:
Not pumping (HEART)
oDysrhythmias
oCHF, cardiogenic shock
oCardiac arrest
Not distributing (VESSELS)
oOcclusion
Low volume
oBleeding: internal vs. external
Interventions:
Not pumping (HEART)
oCompressions
oMedications
oElectricity
Not distributing (VESSELS)
oMedications/Treatment per orders
Low volume
oIVF or blood products
oApply direct pressure if bleeding
Cardiac Emergency/Arrest
Rapid assessment and intervention → within 10 seconds
Activation of code blue
PRIORITY INTERVENTIONS:
oIMMEDIATE COMPRESSIONS and EARLY DEFIBRILLATION when indicated
Cause of Cardiac Arrest:
Hs:
oHypovolemia
oHypoxia
oCO = SV X HR
oNormal CO = 4-8 L
oCI: CO adjusted to body surface area – more accurate
oCO/Total BSA = CI
oMean arterial pressure (MAP) average pressure in large vessels of body
MAP = 2 * DBP + SBP / 3
Normal MAP 70-105, 60-65 is BARE MIN
oPreload: the ventricles filling up with blood during diastole (relaxation) – refers to fluid status
Basically STRETCH – volume of blood received by the heart
Increased preload = too much fluid, super wet
Decreased preload = too little fluid, super dry
It is increased in hypervolemia, regurgitation of cardiac valves, heart failure
oAfterload: the amount of pressure that the heart has to push against to eject blood during systole (contraction) – refers to
fluid status
Basically SQUEEZE – pressure/resistance the heart has to overcome to eject blood
Increased afterload – too much fluid, super wet
Decreased afterload = too little fluid, super dry
oPulmonary Artery Wedge Pressure (PAWP) LEFT SIDED PRELOAD
Measurement of pulmonary capillary pressure which reflects left ventricular end diastolic pressure. Measures
pressure in the lungs – left ventricular preload pressure
Normal 25/10 mmHg (quarter over dime)
Rules out cardiac issues (I.E. HF causing fluid overload or capillary membrane issue like ARDS)
<18 mmHg = ARDS
>18 mmHg = Cardiac issue
oCentral Venous Pressure (CVP) RIGHT SIDED PRELOAD
Right ventricular preload or right ventricular end-diastolic pressure
Normal CVP 2-8 mmHg
>8 = too much fluid (CHF patients, fluid overload)
<3 = too dry (hypovolemia, hypotensive patients)
Reflects amount of blood returning to the heart and the ability of the heart to pump blood into the arterial
system
oLeft (PAWP) and Right preloads (CVP) – mini summary
High CVP & PAWP PATIENT IS TOO WET (fluid overload)
How do you decrease it???
oDIURETICS
oVasodilators
oFluid Restriction
Low CVP & PAWP Patient is too DRY (poor perfusion, decreased CO)
How do you increase it???
oVasopressors (Norepi, Epi)
oVasoconstrictors
oFluid Administration (Crystalloids: NS, LR)
PopS to my LEFT C my Pussy to the RIGHT
PopS (Left side)
oPAWP – left preload (6-12)
oSVR – left after load
C Pussy (right side)
oCVP – right preload (2-8)
oPVR – right after load
oSystemic Vascular resistance (SVR) LEFT SIDED AFTERLOAD
The opposition to blood flow encountered by the left ventricle
Normal SCVR = 800-1200 dynes/sec/cm
800 vessels are hella dilated and loose
1200 the vessels are constricted and tight
, oPulmonary Vascular Resistance (PVR) RIGHT SIDED AFTERLOAD
The opposition to blood flow encountered by the right ventricle
Normal PVR <250 dynes/sec/cm
oLeft (SVR) & Right (PVR) mini summary
High SVR and PVE the cardiac system has to work HARDER (fluid overload)
How do you decrease afterload??
oDiuretics
oVasodilators so the heart doesn’t have to push so hard to circulate blood to the body
Low SVR & PVR there is not enough blood to circulate (decreased CO and decreased preload)
oHow do you increase afterload???
Fluids
oSwan Ganz Catheter (Pulmonary Artery Catheter)
Measures hemodynamics in the body, catheter is advanced into the heart and the balloon sits in the distal tip of
the pulmonary artery
Balloon (1.5 mmHg) should NOT stay inflated – can cause the rupture of the PA and death
What does it tell us?
Co/CI
CVP
SvO2 (mixed venous sample)
Core temperature
PAWP
SVR – great indicator for fluid status
Patient should be supine, CONTINUOUS EKG MONITORING, monitoring coags, ABG, oxygenation
TRANSDUCER positioning needs to be leveled at the phlebostatic axis (4th intercostal space) & zero-point
referenced (leveled at 0)
The patient can move with the SG catheter but every time they move, we need to revel the transducer
CXR verifies the placement
Complications – ARRHYTHMIAS when the catheter is being thread through, it can touch the ventricle and cause
irritation vtach, v fib
Rupture, infection, air embolism
Primary Survey - ABCDE; Emergency Severity Index
oPrimary Survey: ABCDE
Airway management
Cardiac emergency
Rapid response team
Triage & Prioritizing
Poisoning
oAirway: maintain airway with cervical spine control
Responsiveness: awake & alert vs. lethargic, stuporous, obtunded, unarousable, unresponsive
Obstructed: foreign body, physical material, inflammation, edema
Assessment findings: stridor, mufÒed/changing voice, drooling, weak or absent gag reflex, facial trauma/surgery,
foreign object present
Head-tilt/Chin-tilt
Jaw thrust: cervical protection
Airway adjuncts:
Nasopharyngeal:
oCI: facial or skull fractures
Oropharyngeal:
oCI: gag reflex present
Endotracheal intubation:
oProcedure:
Gather supplies
Hyperoxygenate w/bag valve mask (100% O2)
Provider places the tube → hyperoxygenate btw attempts
Prelim confirmation → chest rise, breath sounds, ETCO2
, Secure tube
CXR
oConfirmation:
Assess for chest rise
ETCO2 detector (PURPLE TO YELLOW)
Auscultation
Rising SPO2
CXR
oBreathing: assess breathing and ventilation. Apply high flow O2 on non-rebreather.
Assessment:
Rate, Expansion
Effort
Auscultation
SpO2 & ABG
Interventions:
Positioning
O2 administration
Manual ventilation
CPAP/BiPAP/Mechanical ventilation
Pulmonary hygiene
Respiratory treatments
oCirculation: assess circulation with hemorrhage control
Assess:
Heart rate
Blood pressure
Peripheral pulses
Capillary refill
Need to ensure the patient is being perfused!
Problems:
Not pumping (HEART)
oDysrhythmias
oCHF, cardiogenic shock
oCardiac arrest
Not distributing (VESSELS)
oOcclusion
Low volume
oBleeding: internal vs. external
Interventions:
Not pumping (HEART)
oCompressions
oMedications
oElectricity
Not distributing (VESSELS)
oMedications/Treatment per orders
Low volume
oIVF or blood products
oApply direct pressure if bleeding
Cardiac Emergency/Arrest
Rapid assessment and intervention → within 10 seconds
Activation of code blue
PRIORITY INTERVENTIONS:
oIMMEDIATE COMPRESSIONS and EARLY DEFIBRILLATION when indicated
Cause of Cardiac Arrest:
Hs:
oHypovolemia
oHypoxia