NSG 3850 EXAM 4 ACTUAL EXAM 2026 || MOST RECENT EXAM
2026|2027 ACTUAL COMPLETE REAL EXAM QUESTIONS AND
CORRECT ANSWERS (VERIFIED ANSWERS) ALREADY
GRADED A+ | GUARANTEED SUCCESS!! BRAND NEW
VERSION | JUST RELEASED!!| GALEN COLLEGE OF NURSING
liver cells show pathologic changes of hepatocyte necrosis
with neutrophilic infiltration and intracellular inclusions
(Mallory bodies). from large alcoholic binges. - ANSWER-
alcoholic hepatitis
s/s: hepatomegaly, fever, acute liver failure, encephalopathy.
may be complicated by acute alcohol withdrawal and delirium
tremors. – ANSWER-alcoholic hepatitis
risk factors: obesity (BMI>30), high cholesterol, diabetic -
ANSWER-NASH
metal storage diseases such as hereditary hemochromatosis
(iron overload)
and Wilsons disease (copper overload) cause liver damage can
worsen or mimic what? - ANSWER-NASH
inflammation stays in the portal triads with no signs of
significant fibrosis or
cirrhosis - ANSWER-chronic
persistent hepatitis
,destructive inflammatory disease that extends beyond the
portal triad to the
hepatic lobe - ANSWER-chronic active
hepatitis
progressive liver inflammation with presence of distinctive
autoantibodies.
increase seen with covid 19 - ANSWER-
autoimmune hepatitis
an important hormone in the response to acute and chronic
stress - ANSWER-
cortisol
produced by special cells in the thyroid - ANSWER-calcitonin
intrinsic dysfunction of the thyroid gland, surgical removal
of gland, having
Hashimoto's thyroiditis - ANSWER-primary
hypothyroidism
progressive destruction of the thyroid that causes the
release of T3 and T4
hormones - ANSWER-
Hashimotos
can be caused by severe head trauma, pituitary failure (doesn't
release TSH),
,brain infections cranial neoplasms or irradiation,
neurosurgery - ANSWER-
secondary
hypothyroidism
clinical manifestations: weakness/fatigue, lethargy, cold
intolerance, bradycardia, constipation, slowed cognitive ability
and speech, difficulty with concertation and memory,
forgetfulness, goiter, menstrual irregularities - ANSWER-
hypothyroidism
diagnosed through elevated TSH, decreased T3 and T4
- ANSWER-
hypothyroidism
treat with levothyroxine - ANSWER-hypothyroidism
complication is myxedema coma (causes changes in skin,
change in mental
status, and hypothermia. life threatening) - ANSWER-
hypothyroidism
can be caused by graves disease (high levels/too much
TSH) - ANSWER-
Hyperthyroidism
clinical manifestations: insomnia, restlessness, diaphoresis,
irritability,
, tachycardia, increased appetite and intestinal motility, weight
loss, exophthalmia (prominent eyes), amenorrhea (no periods) -
ANSWER-hyperthyroidism
complication is thyroid storm. thyroid gland releases large
amount of TSH in
short period. life threatening. - ANSWER-
hyperthyroidism
occurs because of low serum calcium levels - ANSWER-
hypoparathyroidism
clinical manifestations include paresthesia, muscle cramps,
tetany, fatigue,
neuromuscular irritability, seizures, anxiety prolongation
of Q-T intervals,
laryngospasms, depression - ANSWER-
hypoparathyroidism
clinical manifestations are related to hypercalcemia. pt more
prone to kidney stones, polyuria, dehydration, anorexia, N/V,
and constipation. causes cardiac issues like bradycardia, heart
blocks, and cardiac arrest – ANSWER-hyperparathyroidism
primary: idiopathic, autoimmune, trauma. Secondary:
corticosteroid use,
damage to hypothalamus or pituitary gland. also called adrenal
insufficiency - ANSWER-Addison's disease
2026|2027 ACTUAL COMPLETE REAL EXAM QUESTIONS AND
CORRECT ANSWERS (VERIFIED ANSWERS) ALREADY
GRADED A+ | GUARANTEED SUCCESS!! BRAND NEW
VERSION | JUST RELEASED!!| GALEN COLLEGE OF NURSING
liver cells show pathologic changes of hepatocyte necrosis
with neutrophilic infiltration and intracellular inclusions
(Mallory bodies). from large alcoholic binges. - ANSWER-
alcoholic hepatitis
s/s: hepatomegaly, fever, acute liver failure, encephalopathy.
may be complicated by acute alcohol withdrawal and delirium
tremors. – ANSWER-alcoholic hepatitis
risk factors: obesity (BMI>30), high cholesterol, diabetic -
ANSWER-NASH
metal storage diseases such as hereditary hemochromatosis
(iron overload)
and Wilsons disease (copper overload) cause liver damage can
worsen or mimic what? - ANSWER-NASH
inflammation stays in the portal triads with no signs of
significant fibrosis or
cirrhosis - ANSWER-chronic
persistent hepatitis
,destructive inflammatory disease that extends beyond the
portal triad to the
hepatic lobe - ANSWER-chronic active
hepatitis
progressive liver inflammation with presence of distinctive
autoantibodies.
increase seen with covid 19 - ANSWER-
autoimmune hepatitis
an important hormone in the response to acute and chronic
stress - ANSWER-
cortisol
produced by special cells in the thyroid - ANSWER-calcitonin
intrinsic dysfunction of the thyroid gland, surgical removal
of gland, having
Hashimoto's thyroiditis - ANSWER-primary
hypothyroidism
progressive destruction of the thyroid that causes the
release of T3 and T4
hormones - ANSWER-
Hashimotos
can be caused by severe head trauma, pituitary failure (doesn't
release TSH),
,brain infections cranial neoplasms or irradiation,
neurosurgery - ANSWER-
secondary
hypothyroidism
clinical manifestations: weakness/fatigue, lethargy, cold
intolerance, bradycardia, constipation, slowed cognitive ability
and speech, difficulty with concertation and memory,
forgetfulness, goiter, menstrual irregularities - ANSWER-
hypothyroidism
diagnosed through elevated TSH, decreased T3 and T4
- ANSWER-
hypothyroidism
treat with levothyroxine - ANSWER-hypothyroidism
complication is myxedema coma (causes changes in skin,
change in mental
status, and hypothermia. life threatening) - ANSWER-
hypothyroidism
can be caused by graves disease (high levels/too much
TSH) - ANSWER-
Hyperthyroidism
clinical manifestations: insomnia, restlessness, diaphoresis,
irritability,
, tachycardia, increased appetite and intestinal motility, weight
loss, exophthalmia (prominent eyes), amenorrhea (no periods) -
ANSWER-hyperthyroidism
complication is thyroid storm. thyroid gland releases large
amount of TSH in
short period. life threatening. - ANSWER-
hyperthyroidism
occurs because of low serum calcium levels - ANSWER-
hypoparathyroidism
clinical manifestations include paresthesia, muscle cramps,
tetany, fatigue,
neuromuscular irritability, seizures, anxiety prolongation
of Q-T intervals,
laryngospasms, depression - ANSWER-
hypoparathyroidism
clinical manifestations are related to hypercalcemia. pt more
prone to kidney stones, polyuria, dehydration, anorexia, N/V,
and constipation. causes cardiac issues like bradycardia, heart
blocks, and cardiac arrest – ANSWER-hyperparathyroidism
primary: idiopathic, autoimmune, trauma. Secondary:
corticosteroid use,
damage to hypothalamus or pituitary gland. also called adrenal
insufficiency - ANSWER-Addison's disease
